Aida Calderón‐Rivera

444 total citations
23 papers, 245 citations indexed

About

Aida Calderón‐Rivera is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Aida Calderón‐Rivera has authored 23 papers receiving a total of 245 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 12 papers in Physiology and 11 papers in Cellular and Molecular Neuroscience. Recurrent topics in Aida Calderón‐Rivera's work include Pain Mechanisms and Treatments (12 papers), Ion channel regulation and function (10 papers) and Neuroscience and Neuropharmacology Research (5 papers). Aida Calderón‐Rivera is often cited by papers focused on Pain Mechanisms and Treatments (12 papers), Ion channel regulation and function (10 papers) and Neuroscience and Neuropharmacology Research (5 papers). Aida Calderón‐Rivera collaborates with scholars based in United States, Mexico and China. Aida Calderón‐Rivera's co-authors include Ricardo Felix, Kimberly Gómez, Alejandro Sandoval, Arturo Andrade, Ricardo González‐Ramírez, Rajesh Khanna, Vinicio Granados‐Soto, Rodolfo Delgado‐Lezama, Christian González‐Billault and Juan Carlos Gómora and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Aida Calderón‐Rivera

21 papers receiving 243 citations

Peers

Aida Calderón‐Rivera
Val Millar United Kingdom
Kevin B.S. Chow Hong Kong
Hesham M. Ismail Switzerland
Val Millar United Kingdom
Aida Calderón‐Rivera
Citations per year, relative to Aida Calderón‐Rivera Aida Calderón‐Rivera (= 1×) peers Val Millar

Countries citing papers authored by Aida Calderón‐Rivera

Since Specialization
Citations

This map shows the geographic impact of Aida Calderón‐Rivera's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aida Calderón‐Rivera with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aida Calderón‐Rivera more than expected).

Fields of papers citing papers by Aida Calderón‐Rivera

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aida Calderón‐Rivera. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aida Calderón‐Rivera. The network helps show where Aida Calderón‐Rivera may publish in the future.

Co-authorship network of co-authors of Aida Calderón‐Rivera

This figure shows the co-authorship network connecting the top 25 collaborators of Aida Calderón‐Rivera. A scholar is included among the top collaborators of Aida Calderón‐Rivera based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aida Calderón‐Rivera. Aida Calderón‐Rivera is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Martin, Laurent, Harrison J. Stratton, Kimberly Gómez, et al.. (2025). Anti-CV2/CRMP5 autoantibodies as drivers of sensory neuron excitability and pain in rats. Nature Communications. 16(1). 7311–7311.
3.
Calderón‐Rivera, Aida, et al.. (2024). SUMOylation and DeSUMOylation: Tug of War of Pain Signaling. Molecular Neurobiology. 62(3). 3305–3321. 2 indexed citations
4.
Peach, Chloe J., Raquel Tonello, Kimberly Gómez, et al.. (2024). Neuropilin-1 inhibition suppresses nerve growth factor signaling and nociception in pain models. Journal of Clinical Investigation. 135(4). 6 indexed citations
5.
Calderón‐Rivera, Aida, et al.. (2024). Targeting Nav1.7 and Nav1.8 with a PIKfyve inhibitor to reverse inflammatory and neuropathic pain. PubMed. 17. 100174–100174. 1 indexed citations
6.
Allen, Heather N., Aida Calderón‐Rivera, Kimberly Gómez, et al.. (2023). Intranasal CRMP2-Ubc9 inhibitor regulates NaV1.7 to alleviate trigeminal neuropathic pain. Pain. 165(3). 573–588. 13 indexed citations
7.
Gómez, Kimberly, Harrison J. Stratton, Cheng Tang, et al.. (2023). Identification and targeting of a unique Na V 1.7 domain driving chronic pain. Proceedings of the National Academy of Sciences. 120(32). e2217800120–e2217800120. 23 indexed citations
8.
Gómez, Kimberly, Raquel Tonello, Lisa Boinon, et al.. (2023). Neuropilin-1 Is Essential For VEGFA-Mediated Increase Of Sensory Neuron Activity And Development Of Pain-Like Behaviors. Journal of Pain. 24(4). 40–40.
9.
Calderón‐Rivera, Aida, Kimberly Gómez, E. M. Kithsiri Wijeratne, et al.. (2023). Betulinic acid analogs inhibit N- and T-type voltage-gated calcium channels to attenuate nerve-injury associated neuropathic and formalin models of pain. SHILAP Revista de lepidopterología. 13. 100116–100116. 7 indexed citations
10.
Calderón‐Rivera, Aida, Natanael Zarco, Eduardo Monjaraz, et al.. (2023). Molecular cloning of the gene promoter encoding the human CaVγ2/Stargazin divergent transcript (CACNG2-DT): characterization and regulation by the cAMP-PKA/CREB signaling pathway. Frontiers in Physiology. 14. 1286808–1286808. 1 indexed citations
11.
Xu, Ya-Ming, E. M. Kithsiri Wijeratne, Aida Calderón‐Rivera, et al.. (2023). Argentatin C Analogues with Potential Antinociceptive Activity and Other Triterpenoid Constituents from the Aerial Parts of Parthenium incanum. ACS Omega. 8(22). 20085–20095. 2 indexed citations
12.
Gómez, Kimberly, Raquel Tonello, Heather N. Allen, et al.. (2023). Neuropilin-1 is essential for vascular endothelial growth factor A–mediated increase of sensory neuron activity and development of pain-like behaviors. Pain. 164(12). 2696–2710. 12 indexed citations
13.
Ran, Dongzhi, et al.. (2022). Cell specific regulation of NaV1.7 activity and trafficking in rat nodose ganglia neurons. SHILAP Revista de lepidopterología. 12. 100109–100109. 3 indexed citations
14.
Gómez, Kimberly, Cheng Tang, Bin Tan, et al.. (2022). Stereospecific Effects of Benzimidazolonepiperidine Compounds on T-Type Ca2+Channels and Pain. ACS Chemical Neuroscience. 13(13). 2035–2047. 3 indexed citations
15.
Sandoval, Alejandro, et al.. (2020). Cdk5 phosphorylates CaV1.3 channels and regulates GABAA-mediated miniature inhibitory post-synaptic currents in striato-nigral terminals. Biochemical and Biophysical Research Communications. 524(1). 255–261. 6 indexed citations
16.
Gómez, Kimberly, Aida Calderón‐Rivera, Alejandro Sandoval, et al.. (2019). Cdk5-Dependent Phosphorylation of CaV3.2 T-Type Channels: Possible Role in Nerve Ligation-Induced Neuropathic Allodynia and the Compound Action Potential in Primary Afferent C Fibers. Journal of Neuroscience. 40(2). 283–296. 45 indexed citations
17.
Calderón‐Rivera, Aida, Alejandro Sandoval, Ricardo González‐Ramírez, Christian González‐Billault, & Ricardo Felix. (2015). Regulation of Neuronal Cav3.1 Channels by Cyclin-Dependent Kinase 5 (Cdk5). PLoS ONE. 10(3). e0119134–e0119134. 19 indexed citations
18.
Felix, Ricardo, Aida Calderón‐Rivera, & Arturo Andrade. (2013). Regulation of high‐voltage‐activated Ca2+ channel function, trafficking, and membrane stability by auxiliary subunits. PubMed. 2(5). 207–220. 29 indexed citations
19.
Vega, Ana V., Roberto Ramos‐Mondragón, Aida Calderón‐Rivera, Ángel Zarain‐Herzberg, & Guillermo Ávila. (2011). Calcitonin gene‐related peptide restores disrupted excitation–contraction coupling in myotubes expressing central core disease mutations in RyR1. The Journal of Physiology. 589(19). 4649–4669. 14 indexed citations
20.
Calderón‐Rivera, Aida, Arturo Andrade, Óscar Hernández‐Hernández, et al.. (2011). Identification of a disulfide bridge essential for structure and function of the voltage-gated Ca2+ channel α2δ-1 auxiliary subunit. Cell Calcium. 51(1). 22–30. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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