A. M. Zalin

988 total citations · 1 hit paper
9 papers, 660 citations indexed

About

A. M. Zalin is a scholar working on Molecular Biology, Oncology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, A. M. Zalin has authored 9 papers receiving a total of 660 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Molecular Biology, 3 papers in Oncology and 3 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in A. M. Zalin's work include Bone health and treatments (3 papers), Dermatological and Skeletal Disorders (2 papers) and Parathyroid Disorders and Treatments (2 papers). A. M. Zalin is often cited by papers focused on Bone health and treatments (3 papers), Dermatological and Skeletal Disorders (2 papers) and Parathyroid Disorders and Treatments (2 papers). A. M. Zalin collaborates with scholars based in United Kingdom and Belgium. A. M. Zalin's co-authors include Terry Feest, David M. Vigushin, Mark B. Pepys, Anne K. Soutar, Ong Nam Phuong Nguyen, Nicholas F. Totty, Philip N. Hawkins, GA Tennent, C. C. F. Blake and J. Justin Hsuan and has published in prestigious journals such as Nature, European Journal of Endocrinology and QJM.

In The Last Decade

A. M. Zalin

9 papers receiving 645 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Human lysozyme gene mutations cause hereditary systemic amyloidosis

1993 550 citations Mark B. Pepys, Philip N. Hawkins et al. Nature profile →

Peers

A. M. Zalin

MB

PHYSI

MC

CB

ND

P.K. De Bree Netherlands

B. A. Gordon Canada

Alvaro Ortiz United States

Rita Sigmund United States

Li Cao United States

C. H. Evans United States

Hideki Mitsuhashi Japan

María Victoria Aguirre Argentina

Arthur Kahlenberg Canada

Victoria Sluzky United States

P.K. De Bree Netherlands

A. M. Zalin

764 ×1.5

MB

91 ×0.3

PHYSI

15 ×0.1

MC

39 ×0.6

CB

44 ×0.7

ND

Citations per year, relative to A. M. Zalin A. M. Zalin (= 1×) peers P.K. De Bree

Countries citing papers authored by A. M. Zalin

Since Specialization

Citations

This map shows the geographic impact of A. M. Zalin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. M. Zalin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. M. Zalin more than expected).

Fields of papers citing papers by A. M. Zalin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. M. Zalin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. M. Zalin. The network helps show where A. M. Zalin may publish in the future.

Co-authorship network of co-authors of A. M. Zalin

This figure shows the co-authorship network connecting the top 25 collaborators of A. M. Zalin. A scholar is included among the top collaborators of A. M. Zalin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A. M. Zalin. A. M. Zalin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

9 of 9 papers shown

1.

Pepys, Mark B., Philip N. Hawkins, David R. Booth, et al.. (1993). Human lysozyme gene mutations cause hereditary systemic amyloidosis. Nature. 362(6420). 553–557. 550 indexed citations breakdown →

2.

Zalin, A. M., et al.. (1993). Care of diabetic patients in hospital clinics and general practice clinics: a study in Dudley.. PubMed. 43(367). 65–9. 19 indexed citations

3.

Zalin, A. M., Sally Jones, N J Fitch, & D. Ramsden. (1991). Familial nephropathic non-neuropathic amyloidosis: clinical features, immunohistochemistry and chemistry.. PubMed. 81(295). 945–56. 16 indexed citations

4.

Sidhu, Baldeep S., et al.. (1987). Disappearing hypercalcaemia. Postgraduate Medical Journal. 63(743). 741–744. 8 indexed citations

5.

Wood, Diana, A. M. Zalin, W. A. Ratcliffe, & Michael C. Sheppard. (1987). Elevation of Free Thyroxine Measurements in Patients without Thyrotoxicosis. QJM. 65(246). 863–70. 18 indexed citations

6.

Zalin, A. M., et al.. (1984). Hyponatraemia during treatment with chlorpropamide and Moduretic (amiloride plus hydrochlorothiazide).. BMJ. 289(6446). 659.1–659. 14 indexed citations

7.

Zalin, A. M. & AF Lant. (1984). Anorexia Nervosa Presenting as Reversible Hypoglycaemic Coma. Journal of the Royal Society of Medicine. 77(3). 193–195. 14 indexed citations

8.

Zalin, A. M. & R Hoffenberg. (1977). UPTAKE OF MACROMOLECULES BY THE THYROID. European Journal of Endocrinology. 84(1). 93–104. 1 indexed citations

9.

Zalin, A. M., A. J. Darby, Stuart L. Vaughan, & E. B. Raftery. (1974). Primary Neuropathic Amyloidosis in Three Brothers. BMJ. 1(5897). 65–66. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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