GA Tennent

1.3k total citations · 1 hit paper
7 papers, 982 citations indexed

About

GA Tennent is a scholar working on Molecular Biology, Cell Biology and Physiology. According to data from OpenAlex, GA Tennent has authored 7 papers receiving a total of 982 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 2 papers in Cell Biology and 2 papers in Physiology. Recurrent topics in GA Tennent's work include Amyloidosis: Diagnosis, Treatment, Outcomes (4 papers), Alzheimer's disease research and treatments (2 papers) and Endoplasmic Reticulum Stress and Disease (2 papers). GA Tennent is often cited by papers focused on Amyloidosis: Diagnosis, Treatment, Outcomes (4 papers), Alzheimer's disease research and treatments (2 papers) and Endoplasmic Reticulum Stress and Disease (2 papers). GA Tennent collaborates with scholars based in United Kingdom, United States and Spain. GA Tennent's co-authors include M B Pepys, Laurence Lovat, Philip N. Hawkins, David R. Booth, Mark B. Pepys, Nicholas F. Totty, J. Justin Hsuan, Anne K. Soutar, Terry Feest and David M. Vigushin and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

In The Last Decade

GA Tennent

7 papers receiving 965 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Human lysozyme gene mutations cause hereditary systemic amyloidosis

1993 550 citations Mark B. Pepys, Philip N. Hawkins et al. Nature profile →

Peers

GA Tennent

MB

PHYSI

MC

ONCOL

ND

Ong Nam Phuong Nguyen Germany

Luis M. Blancas‐Mejía United States

G. G. Glenner United States

Jens O. Lagerstedt Sweden

Angela Williams United States

Shigekuni Hosogi Japan

Juan Pablo F.C. Rossi Argentina

Hubert Carchon Belgium

Patricia L. Brown United States

Itzhak Yuli Israel

Ong Nam Phuong Nguyen Germany

GA Tennent

793 ×1.0

MB

352 ×0.7

PHYSI

122 ×1.0

MC

65 ×0.6

ONCOL

97 ×1.0

ND

Citations per year, relative to GA Tennent GA Tennent (= 1×) peers Ong Nam Phuong Nguyen

Countries citing papers authored by GA Tennent

Since Specialization

Citations

This map shows the geographic impact of GA Tennent's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by GA Tennent with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites GA Tennent more than expected).

Fields of papers citing papers by GA Tennent

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by GA Tennent. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by GA Tennent. The network helps show where GA Tennent may publish in the future.

Co-authorship network of co-authors of GA Tennent

This figure shows the co-authorship network connecting the top 25 collaborators of GA Tennent. A scholar is included among the top collaborators of GA Tennent based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with GA Tennent. GA Tennent is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Tennent, GA, Mark Head, Philip N. Hawkins, et al.. (2007). Disease‐associated prion protein is not detectable in human systemic amyloid deposits. The Journal of Pathology. 213(4). 376–383. 3 indexed citations

2.

Booth, David R., et al.. (2000). Transthyretin Ile84Thr is associated with familial amyloid polyneuropathy. Human Mutation. 16(5). 447–447. 3 indexed citations

3.

Gillmore, JD, S E Booth, GA Tennent, et al.. (1998). Transthyretin Ile73Val is associated with familial amyloidotic polyneuropathy in a Bangladeshi family. Human Mutation. 12(2). 135–135. 8 indexed citations

4.

Booth, David R., Shouyong Tan, S E Booth, et al.. (1996). Hereditary hepatic and systemic amyloidosis caused by a new deletion/insertion mutation in the apolipoprotein AI gene.. Journal of Clinical Investigation. 97(12). 2714–2721. 75 indexed citations

5.

Tennent, GA, Laurence Lovat, & M B Pepys. (1995). Serum amyloid P component prevents proteolysis of the amyloid fibrils of Alzheimer disease and systemic amyloidosis.. Proceedings of the National Academy of Sciences. 92(10). 4299–4303. 332 indexed citations

6.

Pepys, Mark B., Philip N. Hawkins, David R. Booth, et al.. (1993). Human lysozyme gene mutations cause hereditary systemic amyloidosis. Nature. 362(6420). 553–557. 550 indexed citations breakdown →

7.

Rordorf, Christiane, et al.. (1982). THE ACUTE-PHASE RESPONSE IN (NZB X NZW)F1 AND MRL/1 MICE - CONTRASTING PATTERNS RESEMBLING THOSE IN HUMAN SYSTEMIC LUPUS-ERYTHEMATOSUS AND RHEUMATOID-ARTHRITIS, RESPECTIVELY. The Journal of Experimental Medicine. 1286. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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