Yu-Cheng Ho

461 total citations
18 papers, 371 citations indexed

About

Yu-Cheng Ho is a scholar working on Pharmacology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Yu-Cheng Ho has authored 18 papers receiving a total of 371 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Pharmacology, 6 papers in Cellular and Molecular Neuroscience and 5 papers in Physiology. Recurrent topics in Yu-Cheng Ho's work include Pain Mechanisms and Treatments (4 papers), Neuroscience and Neuropharmacology Research (3 papers) and Hereditary Neurological Disorders (3 papers). Yu-Cheng Ho is often cited by papers focused on Pain Mechanisms and Treatments (4 papers), Neuroscience and Neuropharmacology Research (3 papers) and Hereditary Neurological Disorders (3 papers). Yu-Cheng Ho collaborates with scholars based in Taiwan, Malaysia and United States. Yu-Cheng Ho's co-authors include Tzer-Bin Lin, Hsien‐Yu Peng, Gin‐Den Chen, Ming-Chun Hsieh, Cheng‐Yuan Lai, Hsin-Jung Lee, Lih‐Chu Chiou, Dylan Chou, Hsueh-Hsiao Wang and An‐Sheng Lee and has published in prestigious journals such as Pain, International Journal of Molecular Sciences and Endocrinology.

In The Last Decade

Yu-Cheng Ho

14 papers receiving 367 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yu-Cheng Ho Taiwan 10 155 125 101 98 93 18 371
Alberto Casti Italy 11 233 1.5× 156 1.2× 46 0.5× 35 0.4× 39 0.4× 17 429
Ludovic D. Langlois United States 10 147 0.9× 44 0.4× 40 0.4× 45 0.5× 27 0.3× 20 321
Raphael Wuo‐Silva Brazil 13 216 1.4× 71 0.6× 136 1.3× 52 0.5× 48 0.5× 42 423
Szilvia Vas Hungary 12 134 0.9× 62 0.5× 173 1.7× 49 0.5× 142 1.5× 29 490
Silvia Fanni Italy 9 133 0.9× 97 0.8× 23 0.2× 21 0.2× 34 0.4× 14 312
Attilio Iemolo United States 12 288 1.9× 60 0.5× 61 0.6× 115 1.2× 108 1.2× 18 520
Adam Kimbrough United States 11 216 1.4× 70 0.6× 106 1.0× 72 0.7× 23 0.2× 26 401
E. Papalexi Greece 9 132 0.9× 29 0.2× 32 0.3× 74 0.8× 38 0.4× 13 384
Juan Antonio García-Carmona Spain 11 133 0.9× 38 0.3× 38 0.4× 41 0.4× 35 0.4× 26 289
Zuzana Babinská Czechia 12 202 1.3× 109 0.9× 38 0.4× 60 0.6× 38 0.4× 17 401

Countries citing papers authored by Yu-Cheng Ho

Since Specialization
Citations

This map shows the geographic impact of Yu-Cheng Ho's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yu-Cheng Ho with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yu-Cheng Ho more than expected).

Fields of papers citing papers by Yu-Cheng Ho

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yu-Cheng Ho. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yu-Cheng Ho. The network helps show where Yu-Cheng Ho may publish in the future.

Co-authorship network of co-authors of Yu-Cheng Ho

This figure shows the co-authorship network connecting the top 25 collaborators of Yu-Cheng Ho. A scholar is included among the top collaborators of Yu-Cheng Ho based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yu-Cheng Ho. Yu-Cheng Ho is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
2.
Lee, Yu Zhao, et al.. (2025). Neurobiology of chronic caffeine use and withdrawal: Mechanisms, effects and implications. Food and Chemical Toxicology. 207. 115817–115817.
3.
Lee, Yu Zhao, et al.. (2025). Antidiabetic potentials of gypenosides: A review on the preclinical effects in glucose and insulin modulation as well as diabetes-related complications. Naunyn-Schmiedeberg s Archives of Pharmacology. 398(11). 14813–14829.
4.
Ho, Yu-Cheng, et al.. (2025). Prenatal maternal HFD exposure impairs cognition via a hippocampal NMDA mechanism. Translational Psychiatry. 15(1). 294–294.
6.
Wu, Cheng‐Chun, Yuting Su, Yu-Cheng Ho, et al.. (2024). Cholinesterase Inhibitor Reveals Synergistic Potential for Neural Stem Cell-Based Therapy in the 5xFAD Mouse Model of Alzheimer’s Disease. Biologics. Volume 18. 363–375. 3 indexed citations
7.
Lee, Yu Zhao, et al.. (2024). Neuroprotective effects of Gypenosides: A review on preclinical studies in neuropsychiatric disorders. European Journal of Pharmacology. 978. 176766–176766. 6 indexed citations
8.
Peng, Hsien‐Yu, Tzer-Bin Lin, Ming-Chun Hsieh, et al.. (2020). NMDA receptor partial agonist GLYX-13 alleviates chronic stress-induced depression-like behavior through enhancement of AMPA receptor function in the periaqueductal gray. Neuropharmacology. 178. 108269–108269. 17 indexed citations
9.
Lin, Tzer-Bin, Cheng‐Yuan Lai, Ming-Chun Hsieh, et al.. (2020). Inhibiting MLL1-WDR5 interaction ameliorates neuropathic allodynia by attenuating histone H3 lysine 4 trimethylation-dependent spinal mGluR5 transcription. Pain. 161(9). 1995–2009. 17 indexed citations
10.
Hsieh, Ming-Chun, Hsien‐Yu Peng, Yu-Cheng Ho, et al.. (2019). Transcription Repressor Hes1 Contributes to Neuropathic Pain Development by Modifying CDK9/RNAPII-Dependent Spinal mGluR5 Transcription. International Journal of Molecular Sciences. 20(17). 4177–4177. 6 indexed citations
11.
Chou, Dylan, Hsien‐Yu Peng, Tzer-Bin Lin, et al.. (2018). (2R,6R)-hydroxynorketamine rescues chronic stress-induced depression-like behavior through its actions in the midbrain periaqueductal gray. Neuropharmacology. 139. 1–12. 82 indexed citations
12.
Hsieh, Ming-Chun, Yu-Cheng Ho, Cheng‐Yuan Lai, et al.. (2018). Spinal TNF-α impedes Fbxo45-dependent Munc13-1 ubiquitination to mediate neuropathic allodynia in rats. Cell Death and Disease. 9(8). 811–811. 17 indexed citations
13.
Ho, Yu-Cheng, Tzer-Bin Lin, Ming-Chun Hsieh, et al.. (2017). Periaqueductal Gray Glutamatergic Transmission Governs Chronic Stress-Induced Depression. Neuropsychopharmacology. 43(2). 302–312. 42 indexed citations
14.
Lee, Hsin-Jung, et al.. (2016). Stress induces analgesia via orexin 1 receptor-initiated endocannabinoid/CB1 signaling in the mouse periaqueductal gray. Neuropharmacology. 105. 577–586. 52 indexed citations
15.
Chiou, Lih‐Chu, Hsin-Jung Lee, Yu-Cheng Ho, et al.. (2010). Orexins/Hypocretins: Pain Regulation and Cellular Actions. Current Pharmaceutical Design. 16(28). 3089–3100. 74 indexed citations
16.
Chen, Gin‐Den, Hsien‐Yu Peng, Kwong‐Chung Tung, et al.. (2007). Descending facilitation of spinal NMDA-dependent reflex potentiation from pontine tegmentum in rats. American Journal of Physiology-Renal Physiology. 293(4). F1115–F1122. 15 indexed citations
17.
Chen, Gin‐Den, Jiuan-Miaw Liao, Hsien‐Yu Peng, et al.. (2006). Estrogen Modulates the SpinalN-Methyl-d-Aspartic Acid-Mediated Pelvic Nerve-to-Urethra Reflex Plasticity in Rats. Endocrinology. 147(6). 2956–2963. 22 indexed citations
18.
Peng, Hsien‐Yu, Cheng‐Hsu Chen, Jiuan-Miaw Liao, et al.. (2006). Acute unilateral ureteral distension inhibits glutamate-dependent spinal pelvic-urethra reflex potentiation via GABAergic neurotransmission in anesthetized rats. American Journal of Physiology-Renal Physiology. 292(3). F1007–F1015. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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