Young‐Gyu Ko
- Molecular Biology top 10%
- Cell Biology top 5%
- Physiology top 10%
- Immunology top 10%
- Materials Chemistry
- Co-authors
- Jae‐Seon LeeJong‐Il KimChunying LiJeong-Sun SeoHyo‐Jung ChooSoonyoung JungChang Seok LeeHwan Myung Kim
- Topics
- Caveolin-1 and cellular processes (12 papers)Metabolism, Diabetes, and Cancer (5 papers)Lipid Membrane Structure and Behavior (5 papers)
- Cited by
- Cell BiologyMolecular BiologyAging
- Journals
- Journal of Biological ChemistryAngewandte Chemie International EditionNature Communications
- Partner nations
- South KoreaUnited StatesSwitzerland
In The Last Decade
Young‐Gyu Ko
49 papers receiving 1.8k citations
Hit Papers
Peers
Comparison fields: 5 of 116
- Molecular Biology 1.2k
- Cell Biology 410
- Physiology 321
- Immunology 221
- Materials Chemistry 157
Countries citing papers authored by Young‐Gyu Ko
This map shows the geographic impact of Young‐Gyu Ko's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Young‐Gyu Ko with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Young‐Gyu Ko more than expected).
Fields of papers citing papers by Young‐Gyu Ko
This network shows the impact of papers produced by Young‐Gyu Ko. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Young‐Gyu Ko. The network helps show where Young‐Gyu Ko may publish in the future.
Co-authorship network of co-authors of Young‐Gyu Ko
This figure shows the co-authorship network connecting the top 25 collaborators of Young‐Gyu Ko. A scholar is included among the top collaborators of Young‐Gyu Ko based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Young‐Gyu Ko. Young‐Gyu Ko is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Therapy-induced senescent cancer cells contribute to cancer progression by promoting ribophorin 1-dependent PD-L1 upregulationbreakdown → | 23 |
| 2 | 15 | |
| 3 | 11 | |
| 4 | 29 | |
| 5 | 9 | |
| 6 | 21 | |
| 7 | 3 | |
| 8 | 11 | |
| 9 | 54 | |
| 10 | 38 | |
| 11 | 36 | |
| 12 | 9 | |
| 13 | 77 | |
| 14 | 9 | |
| 15 | 33 | |
| 16 | 144 | |
| 17 | 16 | |
| 18 | 106 | |
| 19 | 2 | |
| 20 | 31 |
About Young‐Gyu Ko
Young‐Gyu Ko is a scholar working on Cell Biology, Biophysics and Immunology and Allergy, having authored 49 papers that have together received 1.8k indexed citations. Recurring topics across this work include Caveolin-1 and cellular processes (12 papers), Metabolism, Diabetes, and Cancer (5 papers) and Lipid Membrane Structure and Behavior (5 papers). The work is most often cited by research in Cell Biology (410 citations), Molecular Biology (1.2k citations) and Aging (27 citations). Young‐Gyu Ko has collaborated with scholars based in South Korea, United States and Switzerland. Frequent co-authors include Jae‐Seon Lee, Jong‐Il Kim, Chunying Li, Jeong-Sun Seo, Hyo‐Jung Choo, Soonyoung Jung, Chang Seok Lee, Hwan Myung Kim, Jin Hee Hong and Guy A. Thompson. Their work appears in journals such as Journal of Biological Chemistry, Angewandte Chemie International Edition and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.