In Hwa Bae

980 total citations
29 papers, 823 citations indexed

About

In Hwa Bae is a scholar working on Molecular Biology, Cancer Research and Physiology. According to data from OpenAlex, In Hwa Bae has authored 29 papers receiving a total of 823 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 17 papers in Cancer Research and 4 papers in Physiology. Recurrent topics in In Hwa Bae's work include MicroRNA in disease regulation (14 papers), Circular RNAs in diseases (10 papers) and Cancer-related molecular mechanisms research (7 papers). In Hwa Bae is often cited by papers focused on MicroRNA in disease regulation (14 papers), Circular RNAs in diseases (10 papers) and Cancer-related molecular mechanisms research (7 papers). In Hwa Bae collaborates with scholars based in South Korea. In Hwa Bae's co-authors include Hong‐Duck Um, Seung Bum Lee, Myung‐Jin Park, Yun Soo Bae, Sung Hwan Yoon, Jae Yeon Choi, Eun‐Sook Kim, Seung‐Sook Lee, Kyung-Mi Choi and Young Eun Choi and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Cancer Research.

In The Last Decade

In Hwa Bae

27 papers receiving 812 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
In Hwa Bae South Korea 17 595 329 141 124 88 29 823
Yanling Jing China 10 669 1.1× 468 1.4× 140 1.0× 96 0.8× 85 1.0× 17 929
Fang Guo China 8 762 1.3× 580 1.8× 146 1.0× 107 0.9× 55 0.6× 23 1.0k
Yong-Nyun Kim South Korea 14 377 0.6× 206 0.6× 193 1.4× 156 1.3× 79 0.9× 23 732
Gabriella Cirmena Italy 18 383 0.6× 229 0.7× 188 1.3× 98 0.8× 133 1.5× 41 813
Young‐Kyoung Lee South Korea 15 424 0.7× 217 0.7× 83 0.6× 122 1.0× 169 1.9× 25 687
Huzoor Akbar United States 10 451 0.8× 304 0.9× 99 0.7× 98 0.8× 58 0.7× 22 799
Mylène Tajan France 8 624 1.0× 221 0.7× 161 1.1× 266 2.1× 74 0.8× 12 816
Jee‐Youn Kim South Korea 13 472 0.8× 236 0.7× 130 0.9× 76 0.6× 49 0.6× 28 696
Daniela Flügel Germany 9 452 0.8× 377 1.1× 134 1.0× 62 0.5× 111 1.3× 9 739

Countries citing papers authored by In Hwa Bae

Since Specialization
Citations

This map shows the geographic impact of In Hwa Bae's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by In Hwa Bae with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites In Hwa Bae more than expected).

Fields of papers citing papers by In Hwa Bae

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by In Hwa Bae. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by In Hwa Bae. The network helps show where In Hwa Bae may publish in the future.

Co-authorship network of co-authors of In Hwa Bae

This figure shows the co-authorship network connecting the top 25 collaborators of In Hwa Bae. A scholar is included among the top collaborators of In Hwa Bae based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with In Hwa Bae. In Hwa Bae is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Choi, Jae Yeon, et al.. (2024). Atomoxetine suppresses radioresistance in glioblastoma via circATIC/miR-520d-5p/Notch2-Hey1 axis. Cell Communication and Signaling. 22(1). 532–532.
2.
Choi, Jae Yeon, Dong Hyeon Lee, Junhye Kwon, et al.. (2024). miR-1226-5p is involved in radioresistance of colorectal cancer by activating M2 macrophages through suppressing IRF1. Journal of Translational Medicine. 22(1). 980–980. 4 indexed citations
3.
Choi, Jae Yeon, Dong Hyeon Lee, Eun‐Ju Lee, et al.. (2024). Tumor-derived miR-6794-5p enhances cancer growth by promoting M2 macrophage polarization. Cell Communication and Signaling. 22(1). 190–190. 19 indexed citations
4.
Choi, Jae Yeon, et al.. (2023). miR-765 as a promising biomarker for low-dose radiation-induced pulmonary fibrosis. Non-coding RNA Research. 9(1). 33–43. 2 indexed citations
5.
Choi, Jae Yeon, et al.. (2023). Targeting miR-5088-5p attenuates radioresistance by suppressing Slug. Non-coding RNA Research. 8(2). 164–173. 5 indexed citations
6.
Choi, Young Eun, Jae Yeon Choi, Joo Mi Yi, et al.. (2021). Novel miR-5088-5p promotes malignancy of breast cancer by inhibiting DBC2. Molecular Therapy — Nucleic Acids. 25. 127–142. 11 indexed citations
7.
Choi, Jae Yeon, et al.. (2021). miR-519d-3p suppresses tumorigenicity and metastasis by inhibiting Bcl-w and HIF-1α in NSCLC. Molecular Therapy — Oncolytics. 22. 368–379. 10 indexed citations
8.
Choi, Jae Yeon, et al.. (2019). miR-340-5p Suppresses Aggressiveness in Glioblastoma Multiforme by Targeting Bcl-w and Sox2. Molecular Therapy — Nucleic Acids. 17. 245–255. 27 indexed citations
9.
Yu, Hyeong Won, In Hwa Bae, Su‐jin Kim, et al.. (2018). Comparison of Intra-Operative Vital Sign Changes during Total Thyroidectomy in Patients with Controlled and Uncontrolled Graves’ Disease. Journal of Clinical Medicine. 7(12). 566–566. 3 indexed citations
10.
Choi, Jae Yeon, Hyun Jin Shin, & In Hwa Bae. (2018). miR-93-5p suppresses cellular senescence by directly targeting Bcl-w and p21. Biochemical and Biophysical Research Communications. 505(4). 1134–1140. 23 indexed citations
11.
Kim, Eun‐Sook, Jae Yeon Choi, Su Jin Hwang, & In Hwa Bae. (2018). Hypermethylation of miR-205-5p by IR Governs Aggressiveness and Metastasis via Regulating Bcl-w and Src. Molecular Therapy — Nucleic Acids. 14. 450–464. 19 indexed citations
12.
Lee, Hyung Chul, Nam-Gu Her, Donghee Kang, et al.. (2017). Radiation-inducible miR-770-5p sensitizes tumors to radiation through direct targeting of PDZ-binding kinase. Cell Death and Disease. 8(3). e2693–e2693. 29 indexed citations
13.
Hwang, Hyun Jung, Seung Hee Jung, Hyung‐Chul Lee, et al.. (2015). Identification of novel therapeutic targets in the secretome of ionizing radiation-induced senescent tumor cells. Oncology Reports. 35(2). 841–850. 11 indexed citations
14.
Kwak, Seo‐Young, et al.. (2013). Ionizing radiation-inducible miR-494 promotes glioma cell invasion through EGFR stabilization by targeting p190B RhoGAP. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1843(3). 508–516. 38 indexed citations
15.
Ho, Jin‐Nyoung, et al.. (2010). Bcl‐X L and STAT3 mediate malignant actions of γ‐irradiation in lung cancer cells. Cancer Science. 101(6). 1417–1423. 73 indexed citations
16.
Lee, Seung Bum, In Hwa Bae, Yun Soo Bae, & Hong‐Duck Um. (2006). Link between Mitochondria and NADPH Oxidase 1 Isozyme for the Sustained Production of Reactive Oxygen Species and Cell Death. Journal of Biological Chemistry. 281(47). 36228–36235. 161 indexed citations
17.
Bae, In Hwa, Sung Wook Kang, Sung Hwan Yoon, & Hong‐Duck Um. (2006). Cellular components involved in the cell death induced by cisplatin in the absence of p53 activation. Oncology Reports. 15(5). 1175–80. 20 indexed citations
19.
Youn, Sang Woong, et al.. (2004). Cellular senescence induced loss of stem cell proportion in the skin in vitro. Journal of Dermatological Science. 35(2). 113–123. 38 indexed citations
20.
Youn, Sang Woong, et al.. (2003). Assessment of Skin Toxicity Using Living Skin Equivalents. Linchuang pifuke zazhi. 41(3). 308–313. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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