Yavuz Oktay

3.0k total citations
33 papers, 2.1k citations indexed

About

Yavuz Oktay is a scholar working on Molecular Biology, Cancer Research and Infectious Diseases. According to data from OpenAlex, Yavuz Oktay has authored 33 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 9 papers in Cancer Research and 5 papers in Infectious Diseases. Recurrent topics in Yavuz Oktay's work include Mitochondrial Function and Pathology (6 papers), SARS-CoV-2 and COVID-19 Research (5 papers) and Cancer, Hypoxia, and Metabolism (4 papers). Yavuz Oktay is often cited by papers focused on Mitochondrial Function and Pathology (6 papers), SARS-CoV-2 and COVID-19 Research (5 papers) and Cancer, Hypoxia, and Metabolism (4 papers). Yavuz Oktay collaborates with scholars based in Türkiye, United States and Canada. Yavuz Oktay's co-authors include Carla M. Koehler, Joseph A. Garcia, Marzia Scortegagna, Kan Ding, Steven M. Claypool, Pinmanee Boontheung, Joseph A. Loo, Michael J. Bennett, James A. Richardson and John M. Shelton and has published in prestigious journals such as Cell, Journal of Biological Chemistry and Nature Genetics.

In The Last Decade

Yavuz Oktay

29 papers receiving 2.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yavuz Oktay Türkiye 13 1.4k 760 303 276 147 33 2.1k
Laura Hulea Canada 14 1.6k 1.1× 403 0.5× 149 0.5× 102 0.4× 70 0.5× 21 2.0k
Pekka Katajisto Finland 20 1.5k 1.0× 389 0.5× 390 1.3× 307 1.1× 54 0.4× 42 2.4k
Narendra Wajapeyee United States 31 2.7k 1.9× 941 1.2× 340 1.1× 283 1.0× 75 0.5× 95 3.7k
Benilde Jiménez Spain 28 2.2k 1.5× 841 1.1× 127 0.4× 215 0.8× 137 0.9× 49 3.1k
Sandrine Marchetti France 24 1.2k 0.9× 375 0.5× 135 0.4× 125 0.5× 142 1.0× 42 2.1k
Prem K. Premsrirut United States 14 1.2k 0.8× 301 0.4× 454 1.5× 199 0.7× 113 0.8× 22 1.9k
Naoharu Takano Japan 23 1.3k 0.9× 978 1.3× 129 0.4× 164 0.6× 72 0.5× 45 2.5k
Hua Lu United States 24 1.8k 1.3× 395 0.5× 155 0.5× 156 0.6× 73 0.5× 43 2.3k
Jikui Shen United States 33 1.7k 1.2× 293 0.4× 89 0.3× 152 0.6× 60 0.4× 59 2.7k
Corrado Garbi Italy 33 1.7k 1.2× 270 0.4× 195 0.6× 277 1.0× 51 0.3× 80 2.6k

Countries citing papers authored by Yavuz Oktay

Since Specialization
Citations

This map shows the geographic impact of Yavuz Oktay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yavuz Oktay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yavuz Oktay more than expected).

Fields of papers citing papers by Yavuz Oktay

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yavuz Oktay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yavuz Oktay. The network helps show where Yavuz Oktay may publish in the future.

Co-authorship network of co-authors of Yavuz Oktay

This figure shows the co-authorship network connecting the top 25 collaborators of Yavuz Oktay. A scholar is included among the top collaborators of Yavuz Oktay based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yavuz Oktay. Yavuz Oktay is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Can, Güray, et al.. (2025). Functional Annotation of Bipolar Disorder 2 Risk Location Implicates Novel Susceptibility Genes. Neuropsychobiology. 84(2). 65–73.
2.
Oktay, Yavuz, et al.. (2024). tubg1 Somatic Mutants Show Tubulinopathy-Associated Neurodevelopmental Phenotypes in a Zebrafish Model. Molecular Neurobiology. 62(3). 3024–3039.
3.
Oktay, Yavuz, et al.. (2023). An in silico approach to the identification of diagnostic and prognostic markers in low-grade gliomas. PeerJ. 11. e15096–e15096. 6 indexed citations
4.
Lochmüller, Hanns, et al.. (2023). DPAGT1-CDG: Report of Two New Pediatric Patients and Brief Review of the Literature. Molecular Syndromology. 14(4). 322–330. 3 indexed citations
5.
Kohlschmidt, Nicolai, Andreas Hentschel, Enrika Bartels, et al.. (2022). Novel insights into PORCN mutations, associated phenotypes and pathophysiological aspects. Orphanet Journal of Rare Diseases. 17(1). 29–29. 5 indexed citations
6.
7.
Süner, Aslı, et al.. (2021). Different selection dynamics of S and RdRp between SARS-CoV-2 genomes with and without the dominant mutations. Infection Genetics and Evolution. 91. 104796–104796. 12 indexed citations
8.
Karakülah, Gökhan, et al.. (2021). Current mutatome of SARS-CoV-2 in Turkey reveals mutations of interest. TURKISH JOURNAL OF BIOLOGY. 45(1). 104–113. 9 indexed citations
9.
Karakülah, Gökhan, et al.. (2020). RdRp mutations are associated with SARS-CoV-2 genome evolution. PeerJ. 8. e9587–e9587. 50 indexed citations
10.
Yaramış, Ahmet, Hanns Lochmüller, Ana Töpf, et al.. (2020). COL4A1 -related autosomal recessive encephalopathy in 2 Turkish children. Neurology Genetics. 6(1). e392–e392. 8 indexed citations
11.
Ülgen, Ege, Kaya Bilgüvar, Yavuz Oktay, et al.. (2020). Mutations and Copy Number Alterations in IDH Wild-Type Glioblastomas Are Shaped by Different Oncogenic Mechanisms. Biomedicines. 8(12). 574–574. 4 indexed citations
12.
Töpf, Ana, Yavuz Oktay, Uluç Yiş, et al.. (2019). Severe neurodevelopmental disease caused by a homozygous TLK2 variant. European Journal of Human Genetics. 28(3). 383–387. 4 indexed citations
13.
Akyerli, Cemaliye Boylu, E. Zeynep Erson‐Omay, Yavuz Oktay, et al.. (2017). Use of telomerase promoter mutations to mark specific molecular subsets with reciprocal clinical behavior in IDH mutant and IDH wild-type diffuse gliomas. Journal of neurosurgery. 128(4). 1102–1114. 25 indexed citations
14.
Pavlopoulou, Athanasia, Yavuz Oktay, Konstantinos Vougas, et al.. (2016). Determinants of resistance to chemotherapy and ionizing radiation in breast cancer stem cells. Cancer Letters. 380(2). 485–493. 74 indexed citations
15.
Oktay, Yavuz, et al.. (2011). Leiomyosarcoma of the breast. Journal of Surgical Case Reports. 2011(7). 1–1. 8 indexed citations
16.
Wang, Geng, Hsiao‐Wen Chen, Yavuz Oktay, et al.. (2010). PNPASE Regulates RNA Import into Mitochondria. Cell. 142(3). 456–467. 285 indexed citations
17.
Oktay, Yavuz, Elhadji M. Dioum, Satoshi Matsuzaki, et al.. (2007). Hypoxia-inducible Factor 2α Regulates Expression of the Mitochondrial Aconitase Chaperone Protein Frataxin. Journal of Biological Chemistry. 282(16). 11750–11756. 79 indexed citations
18.
Scortegagna, Marzia, Kan Ding, Quiyang Zhang, et al.. (2004). HIF-2α regulates murine hematopoietic development in an erythropoietin-dependent manner. Blood. 105(8). 3133–3140. 187 indexed citations
19.
Scortegagna, Marzia, Margaret A. Morris, Yavuz Oktay, Malcolm J. Bennett, & Joseph A. Garcia. (2003). The HIF family member EPAS1/HIF-2α is required for normal hematopoiesis in mice. Blood. 102(5). 1634–1640. 164 indexed citations
20.
Scortegagna, Marzia, Kan Ding, Yavuz Oktay, et al.. (2003). Multiple organ pathology, metabolic abnormalities and impaired homeostasis of reactive oxygen species in Epas1−/− mice. Nature Genetics. 35(4). 331–340. 407 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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