Nicolai Kohlschmidt
- Molecular Biology
- Genetics top 10%
- Rheumatology top 10%
- Pediatrics, Perinatology and Child Health top 10%
- Surgery
- Co-authors
- Leena PeltonenMarjo KestiläRiitta SalonenJonna TallilaMira KyttäläOuti KopraPaulina Paavola–SakkiManuela C. Koch
- Topics
- Genetic Syndromes and Imprinting (5 papers)Renal and related cancers (4 papers)DNA Repair Mechanisms (3 papers)
- Cited by
- Sensory SystemsGeneticsRheumatology
- Partner nations
- GermanyUnited KingdomCanada
In The Last Decade
Nicolai Kohlschmidt
30 papers receiving 607 citations
Peers
Comparison fields: 5 of 64
- Molecular Biology 387
- Genetics 293
- Rheumatology 122
- Pediatrics, Perinatology and Child Health 121
- Surgery 98
Countries citing papers authored by Nicolai Kohlschmidt
This map shows the geographic impact of Nicolai Kohlschmidt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nicolai Kohlschmidt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nicolai Kohlschmidt more than expected).
Fields of papers citing papers by Nicolai Kohlschmidt
This network shows the impact of papers produced by Nicolai Kohlschmidt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nicolai Kohlschmidt. The network helps show where Nicolai Kohlschmidt may publish in the future.
Co-authorship network of co-authors of Nicolai Kohlschmidt
This figure shows the co-authorship network connecting the top 25 collaborators of Nicolai Kohlschmidt. A scholar is included among the top collaborators of Nicolai Kohlschmidt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nicolai Kohlschmidt. Nicolai Kohlschmidt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 5 | |
| 3 | 5 | |
| 4 | 15 | |
| 5 | 8 | |
| 6 | 1 | |
| 7 | Homozygote und „compound“-heterozygote RYR1-Mutationen: Neue Erkenntnisse zu Prävalenz und Penetranz der malignen Hyperthermie | 0 |
| 8 | 35 | |
| 9 | 22 | |
| 10 | 17 | |
| 11 | 17 | |
| 12 | 15 | |
| 13 | 19 | |
| 14 | 45 | |
| 15 | 180 | |
| 16 | 4 | |
| 17 | 12 | |
| 18 | 5 | |
| 19 | 16 | |
| 20 | 10 |
About Nicolai Kohlschmidt
Nicolai Kohlschmidt is a scholar working on Sensory Systems, Developmental Biology and Otorhinolaryngology, having authored 32 papers that have together received 624 indexed citations. Recurring topics across this work include Genetic Syndromes and Imprinting (5 papers), Renal and related cancers (4 papers) and DNA Repair Mechanisms (3 papers). The work is most often cited by research in Sensory Systems (60 citations), Genetics (293 citations) and Rheumatology (122 citations). Nicolai Kohlschmidt has collaborated with scholars based in Germany, United Kingdom and Canada. Frequent co-authors include Leena Peltonen, Marjo Kestilä, Riitta Salonen, Jonna Tallila, Mira Kyttälä, Outi Kopra, Paulina Paavola–Sakki, Manuela C. Koch, Andreas Ziegler and Oliver Bartsch. Their work appears in journals such as Nature Genetics, PLoS ONE and Human Molecular Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.