Xinming Cai
Impact in
- Immunology and Allergy top 1%
- Cell Adhesion Molecules Research
- Cell Biology top 5%
- Cellular Mechanics and Interactions
- Hippo pathway signaling and YAP/TAZ
- Cellular transport and secretion
Papers in
-
- Polyamine Metabolism and Applications 1
- Ubiquitin and proteasome pathways 1
- Protein Degradation and Inhibitors 1
-
- Cellular Mechanics and Interactions 2
- Co-authors
- Michael D. Schaller (4 shared papers)Daniel Lietha (2 shared papers)Derek F. Ceccarelli (2 shared papers)Michael J. Eck (2 shared papers)Yiqun Li (1 shared paper)Julie A. Vrana (1 shared paper)Min Li (1 shared paper)Zenon Rajfur (1 shared paper)
- Journals
- Molecular and Cellular Biology (2 papers)mAbs (1 paper)The Journal of Immunology (1 paper)Diabetes (1 paper)IUBMB Life (1 paper)
- Partner nations
- United StatesSwitzerlandCanada
In The Last Decade
Xinming Cai
8 papers receiving 721 citations
Peers
Comparison fields: 5 of 65
- Immunology and Allergy 334
- Cell Biology 343
- Molecular Biology 404
- Cancer Research 70
- Immunology 74
Countries citing papers authored by Xinming Cai
This map shows the geographic impact of Xinming Cai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xinming Cai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xinming Cai more than expected).
Fields of papers citing papers by Xinming Cai
This network shows the impact of papers produced by Xinming Cai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xinming Cai. The network helps show where Xinming Cai may publish in the future.
Co-authors
The 25 scholars most cited alongside Xinming Cai, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2007 | 375 | |
| 2 | 2007 | 145 | |
| 3 | 2006 | 73 | |
| 4 | 2019 | 41 | |
| 5 | 2007 | 35 | |
| 6 | 2014 | 33 | |
| 7 | 2020 | 18 | |
| 8 | 2024 | 6 |
About Xinming Cai
Xinming Cai is a scholar working on Molecular Biology, Cell Biology, Immunology and Allergy, Radiology, Nuclear Medicine and Imaging and Genetics, having authored 8 papers that have together received 726 indexed citations. Recurring topics across this work include Cell Adhesion Molecules Research (3 papers), Monoclonal and Polyclonal Antibodies Research (2 papers), Immune Response and Inflammation (2 papers), Cellular Mechanics and Interactions (2 papers), Polyamine Metabolism and Applications (1 paper), Ubiquitin and proteasome pathways (1 paper), Diabetes and associated disorders (1 paper) and Protein Degradation and Inhibitors (1 paper). The work is most often cited by research in Immunology and Allergy (334 citations), Cell Biology (343 citations), Molecular Biology (404 citations), Cancer Research (70 citations) and Immunology (74 citations). Xinming Cai has collaborated with scholars based in United States, Switzerland and Canada. Frequent co-authors include Michael D. Schaller, Daniel Lietha, Derek F. Ceccarelli, Michael J. Eck, Yiqun Li, Julie A. Vrana, Min Li, Zenon Rajfur, Kenneth A. Jacobson and Andrei V. Karginov. Their work appears in journals such as Molecular and Cellular Biology, mAbs, The Journal of Immunology, Diabetes and IUBMB Life.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.