Wenlin An

33 papers receiving 1.8k citations

Peers

Wenlin An
Comparison fields: 5 of 112
  • Physiology 586
  • Aging 29
  • Molecular Biology 1.1k
  • Cellular and Molecular Neuroscience 277
  • Biological Psychiatry 29
Replace Vanessa A. Morais with:
Vanessa A. Morais Portugal
Marthe H. R. Ludtmann United Kingdom
Sandra Pierre Germany
Mi-Sung Kim South Korea
Ritchie Williamson United Kingdom
Marialaura Amadio Italy
Wenming Li China
Silva Hečimović Croatia
Robert C. Cumming Canada
Wenlin An relative to Vanessa A. Morais Portugal Vanessa A. Morais's profile →
Citations per field
00.5×1.5×
Vanessa A. Morais · 1×
Citations per year

Countries citing papers authored by Wenlin An

Since Specialization
Citations

This map shows the geographic impact of Wenlin An's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wenlin An with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wenlin An more than expected).

Fields of papers citing papers by Wenlin An

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wenlin An. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wenlin An. The network helps show where Wenlin An may publish in the future.

Co-authors

The 25 scholars most cited alongside Wenlin An, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Wenlin An Line = papers co-authored together Wenlin An links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 34 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2003262
2 2010231
3 2002208
4 2002140
5 2022134
6 2003119
7 2009110
8 200595
9 200582
10 201470
11 202259
12 200458
13 202044
14 202138
15 200533
16 202326
17 202424
18 202420
19 202218
20 202215

About Wenlin An

Wenlin An is a scholar working on Molecular Biology, Physiology, Pharmacology, Cancer Research and Biomaterials, having authored 34 papers that have together received 1.9k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (7 papers), Extracellular vesicles in disease (6 papers), RNA Interference and Gene Delivery (4 papers), MicroRNA in disease regulation (4 papers), Cholinesterase and Neurodegenerative Diseases (3 papers), Trace Elements in Health (3 papers), Cellular transport and secretion (2 papers) and Electrospun Nanofibers in Biomedical Applications (2 papers). The work is most often cited by research in Physiology (586 citations), Aging (29 citations), Molecular Biology (1.1k citations), Cellular and Molecular Neuroscience (277 citations) and Biological Psychiatry (29 citations). Wenlin An has collaborated with scholars based in China, Sweden and United States. Frequent co-authors include Bengt Winblad, Jin-Jing Pei, Richard F. Cowburn, Jason W. Chin, Khalid Iqbal, Heiko Braak, Inge Grundke‐Iqbal, Xian Zhao, Youxiu Zhong and Xudong Wang. Their work appears in journals such as Frontiers in Bioengineering and Biotechnology, Pharmaceutics, American Journal Of Pathology, Proceedings of the National Academy of Sciences and Neurobiology of Aging.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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