Wen Liu

11.0k total citations
226 papers, 7.2k citations indexed

About

Wen Liu is a scholar working on Molecular Biology, Cancer Research and Immunology. According to data from OpenAlex, Wen Liu has authored 226 papers receiving a total of 7.2k indexed citations (citations by other indexed papers that have themselves been cited), including 175 papers in Molecular Biology, 39 papers in Cancer Research and 29 papers in Immunology. Recurrent topics in Wen Liu's work include Cancer-related gene regulation (31 papers), RNA modifications and cancer (28 papers) and Epigenetics and DNA Methylation (25 papers). Wen Liu is often cited by papers focused on Cancer-related gene regulation (31 papers), RNA modifications and cancer (28 papers) and Epigenetics and DNA Methylation (25 papers). Wen Liu collaborates with scholars based in China, United States and United Kingdom. Wen Liu's co-authors include Michael G. Rosenfeld, Wenjie Guo, Guo-Sheng Hu, Aneel K. Aggarwal, Jian‐cheng Ding, Qiang Xu, Yum-Shing Wong, Yanyu Huang, Cundong Fan and Tianfeng Chen and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Wen Liu

218 papers receiving 7.2k citations

Peers

Wen Liu
Comparison fields: 5 of 147
  • Molecular Biology 5.0k
  • Cancer Research 1.1k
  • Oncology 952
  • Immunology 892
  • Pulmonary and Respiratory Medicine 470
Replace Yan Xu with:
Yan Xu United States
Martin O. Bergö Sweden
Jakob Troppmair Austria
Bob van de Water Netherlands
Marco Falasca Australia
Xueying Sun China
François M. Vallette France
Peter Störz United States
Juan Miguel Redondo Spain
Gianluca Tell Italy
Yan Xu United States View profile →
Citations per field, relative to Wen Liu
Wen Liu · 1×
Citations per year, relative to Wen Liu
Wen Liu · 1×

Countries citing papers authored by Wen Liu

Since Specialization
Citations

This map shows the geographic impact of Wen Liu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wen Liu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wen Liu more than expected).

Fields of papers citing papers by Wen Liu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wen Liu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wen Liu. The network helps show where Wen Liu may publish in the future.

Co-authorship network of co-authors of Wen Liu

This figure shows the co-authorship network connecting the top 25 collaborators of Wen Liu. A scholar is included among the top collaborators of Wen Liu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wen Liu. Wen Liu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 2
2 1
3 1
4 0
5 8
6 2
7 13
8 2
9 8
10 11
11 1
12 32
13 4
14 13
15 5
16 55
17 149
18 13
19 85
20
Examination of immune tolerance to heterologous-transplanted tumor of human stomach cancer in Syria Hamsters
1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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