Wei Shi

9.5k total citations · 1 hit paper
123 papers, 7.2k citations indexed

About

Wei Shi is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Surgery. According to data from OpenAlex, Wei Shi has authored 123 papers receiving a total of 7.2k indexed citations (citations by other indexed papers that have themselves been cited), including 64 papers in Pulmonary and Respiratory Medicine, 50 papers in Molecular Biology and 38 papers in Surgery. Recurrent topics in Wei Shi's work include Neonatal Respiratory Health Research (50 papers), Congenital Diaphragmatic Hernia Studies (30 papers) and Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (12 papers). Wei Shi is often cited by papers focused on Neonatal Respiratory Health Research (50 papers), Congenital Diaphragmatic Hernia Studies (30 papers) and Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (12 papers). Wei Shi collaborates with scholars based in United States, China and Canada. Wei Shi's co-authors include David Warburton, Savério Bellusci, Makoto Matsumoto, Hui Chen, Kenji Nakanishi, Masashi Minami, Nobuaki Yoshida, Kohsuke Takeda, Tadamitsu Kishimoto and Shin‐ichiro Kashiwamura and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Wei Shi

120 papers receiving 7.1k citations

Hit Papers

Essential role of Stat6 in IL-4 signalling 1996 2026 2006 2016 1996 400 800 1.2k

Peers

Wei Shi
Mary E. Sunday United States
Jalees Rehman United States
Xiaozhu Huang United States
Jason R. Rock United States
Kory J. Lavine United States
Wei Shi
Citations per year, relative to Wei Shi Wei Shi (= 1×) peers Yujiro Asada

Countries citing papers authored by Wei Shi

Since Specialization
Citations

This map shows the geographic impact of Wei Shi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Shi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Shi more than expected).

Fields of papers citing papers by Wei Shi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei Shi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Shi. The network helps show where Wei Shi may publish in the future.

Co-authorship network of co-authors of Wei Shi

This figure shows the co-authorship network connecting the top 25 collaborators of Wei Shi. A scholar is included among the top collaborators of Wei Shi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei Shi. Wei Shi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Luo, Yongfeng, Joanne Chiu, Hui Chen, et al.. (2023). Defective mesenchymal Bmpr1a-mediated BMP signaling causes congenital pulmonary cysts. eLife. 12. 1 indexed citations
2.
Wang, Ji, Harilaos Filippakis, Heng Du, et al.. (2021). Interleukin-6 mediates PSAT1 expression and serine metabolism in TSC2-deficient cells. Proceedings of the National Academy of Sciences. 118(39). 17 indexed citations
3.
Vierhout, Megan, et al.. (2021). Monocyte and macrophage derived myofibroblasts: Is it fate? A review of the current evidence. Wound Repair and Regeneration. 29(4). 548–562. 53 indexed citations
4.
Feng, Lingfang, Zhaoqiang Jiang, Junqiang Chen, et al.. (2020). Exploration of identifying novel serum biomarkers for malignant mesothelioma using iTRAQ combined with 2D-LC-MS/MS. Environmental Research. 193. 110467–110467. 4 indexed citations
5.
Chu, Ling, Yongfeng Luo, Hui Chen, et al.. (2020). Mesenchymal folliculin is required for alveolar development: implications for cystic lung disease in Birt-Hogg-Dubé syndrome. Thorax. 75(6). 486–493. 10 indexed citations
6.
Shi, Wei, et al.. (2019). Perineuronal nets protect long-term memory by limiting activity-dependent inhibition from parvalbumin interneurons. Proceedings of the National Academy of Sciences. 116(52). 27063–27073. 83 indexed citations
7.
Guo, Yi, Benhong Xu, Zhenxing Sun, et al.. (2018). Quantitative protein profiling and pathway analysis of spinal arteriovenous malformations. Microvascular Research. 120. 47–54. 3 indexed citations
8.
Bellaye, Pierre‐Simon, Chiko Shimbori, Chandak Upagupta, et al.. (2017). Lysyl Oxidase–Like 1 Protein Deficiency Protects Mice from Adenoviral Transforming Growth Factor-β1–induced Pulmonary Fibrosis. American Journal of Respiratory Cell and Molecular Biology. 58(4). 461–470. 49 indexed citations
9.
Tao, Hongwei, et al.. (2017). Developmental dislocation of the hip in infants: the hip′s ultrasound quantitative analysis. 14(3). 210–219. 1 indexed citations
10.
Wang, Xin, et al.. (2016). An enriched environment enhances synaptic plasticity and cognition post-stroke. Zhonghua wuli yixue zazhi. 38(9). 647–651. 1 indexed citations
11.
Ren, Siying, Yongfeng Luo, Hui Chen, et al.. (2016). Inactivation of Tsc2 in Mesoderm-Derived Cells Causes Polycystic Kidney Lesions and Impairs Lung Alveolarization. American Journal Of Pathology. 186(12). 3261–3272. 21 indexed citations
12.
Shi, Wei, Wei Xu, Mark R. Frey, et al.. (2015). Increased alveolar soluble annexin V promotes lung inflammation and fibrosis. European Respiratory Journal. 46(5). 1417–1429. 14 indexed citations
13.
Man, Kevin, Maria Miasari, Wei Shi, et al.. (2014). The transcription factor IRF4 is essential for TCR affinity-mediated metabolic programming and clonal expansion of T cells (vol 14, pg 1155, 2013). Nature Immunology. 15(9). 2 indexed citations
14.
Sun, Jianping, Yi‐Hsin Liu, Hui Chen, et al.. (2007). Deficient Alk3-mediated BMP signaling causes prenatal omphalocele-like defect. Biochemical and Biophysical Research Communications. 360(1). 238–243. 18 indexed citations
15.
Moral, Pierre-Marie del, Frédéric Sala, Denise Tefft, et al.. (2005). VEGF-A signaling through Flk-1 is a critical facilitator of early embryonic lung epithelial to endothelial crosstalk and branching morphogenesis. Developmental Biology. 290(1). 177–188. 101 indexed citations
16.
Shi, Wei, Hui Chen, Jianping Sun, et al.. (2004). Overexpression of Smurf1 negatively regulates mouse embryonic lung branching morphogenesis by specifically reducing Smad1 and Smad5 proteins. American Journal of Physiology-Lung Cellular and Molecular Physiology. 286(2). L293–L300. 46 indexed citations
17.
Buckley, S., Wei Shi, B. Driscoll, et al.. (2004). BMP4 signaling induces senescence and modulates the oncogenic phenotype of A549 lung adenocarcinoma cells. American Journal of Physiology-Lung Cellular and Molecular Physiology. 286(1). L81–L86. 63 indexed citations
18.
Warburton, David, Denise Tefft, Arnaud Mailleux, et al.. (2001). Do Lung Remodeling, Repair, and Regeneration Recapitulate Respiratory Ontogeny?. American Journal of Respiratory and Critical Care Medicine. 164(Supplement_2). S59–S62. 60 indexed citations
19.
Watanabe, Chie, Atsushi Kumanogoh, Wei Shi, et al.. (2001). Enhanced Immune Responses in Transgenic Mice Expressing a Truncated Form of the Lymphocyte Semaphorin CD100. The Journal of Immunology. 167(8). 4321–4328. 47 indexed citations
20.
Kumanogoh, Atsushi, Chie Watanabe, Xiaosong Wang, et al.. (2000). Identification of CD72 as a Lymphocyte Receptor for the Class IV Semaphorin CD100. Immunity. 13(5). 621–631. 285 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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