W. Gillon

695 citations
15 papers · 555 · h-index 12

Impact in

Papers in

W. Gillon

15 papers receiving 537 citations

Peers

W. Gillon
Comparison fields: 5 of 68
  • Endocrine and Autonomic Systems 83
  • Physiology 259
  • Aging 12
  • Epidemiology 204
  • Genetics 41
Replace Ji Seul Han with:
Ji Seul Han South Korea
Nira B. Garty Israel
Stephanie Kadow Germany
Rókus Kriszt Singapore
Sara M. Nowinski United States
Bethany C. Hazen United States
Giuseppina Covello Italy
Leonore Wigger Switzerland
Nuraly K. Avliyakulov United States
Natalie Weber Germany
W. Gillon relative to Ji Seul Han South Korea Ji Seul Han's profile →
Citations per field
00.5×2.6×
Ji Seul Han · 1×
Citations per year

Countries citing papers authored by W. Gillon

Since Specialization
Citations

This map shows the geographic impact of W. Gillon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. Gillon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. Gillon more than expected).

Fields of papers citing papers by W. Gillon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. Gillon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. Gillon. The network helps show where W. Gillon may publish in the future.

Co-authors

The 20 scholars most cited alongside W. Gillon, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with W. Gillon Line = papers co-authored together W. Gillon links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1 1990150
2 199474
3 200469
4 200650
5
Effects of fat depot site on differentiation-dependent gene expression in rat preadipocytes.
199643
6 200541
7 200231
8 199325
9 199815
10 199214
11
Increasing vimentin expression associated with differentiation of human and rat preadipocytes.
199612
12 199311
13 19938
14 19797
15 19795

About W. Gillon

W. Gillon is a scholar working on Physiology, Molecular Biology, Epidemiology, Cell Biology and Nutrition and Dietetics, having authored 15 papers that have together received 555 indexed citations. Recurring topics across this work include Adipose Tissue and Metabolism (6 papers), Adipokines, Inflammation, and Metabolic Diseases (5 papers), Fatty Acid Research and Health (3 papers), Enzyme Structure and Function (3 papers), Plant Pathogens and Fungal Diseases (2 papers), Enzyme-mediated dye degradation (2 papers), Microbial Metabolism and Applications (2 papers) and Telomeres, Telomerase, and Senescence (2 papers). The work is most often cited by research in Endocrine and Autonomic Systems (83 citations), Physiology (259 citations), Aging (12 citations), Epidemiology (204 citations) and Genetics (41 citations). W. Gillon has collaborated with scholars based in Canada, United States and Vietnam. Frequent co-authors include C. H. Hollenberg, James L. Kirkland, E.F. Pai, Robert Garces, Ning Wu, Jian Payandeh, M. Fujihashi, Stefan Kindler, Rosemary C. Hynes and Emma L. A. Macfarlane. Their work appears in journals such as Protein Science, Mycologia, The EMBO Journal, Biochemistry and Cell Biology and Endocrinology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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