Vrinda Kalia

2.8k citations
29 papers · 2.0k indexed · 1 hit paper · h-index 10

Vrinda Kalia

27 papers receiving 2.0k citations

Hit Papers

Molecular Signature of CD8+ T Cell Exhaustion during Chro...1.5k200720262013201950010001.5k

Peers

Vrinda Kalia
Comparison fields: 5 of 114
  • Immunology 1.3k
  • Virology 121
  • Oncology 672
  • Aging 30
  • Health, Toxicology and Mutagenesis 208
Replace Xiaoming Zhu with:
Xiaoming Zhu China
Patrick Ziegler Germany
Loı̈c Blanchon France
Jodene K. Moore United States
Patricia de Nadaı̈ France
Ramón María Alvargonzález Rodríguez Spain
Leah C. Kottyan United States
Fabian Beier Germany
Dan Hu China
Patrick Günther Germany
Vrinda Kalia relative to Xiaoming Zhu China Xiaoming Zhu's profile →
Citations per field
00.5×8.7×
Xiaoming Zhu · 1×
Citations per year

Countries citing papers authored by Vrinda Kalia

Since Specialization
Citations

This map shows the geographic impact of Vrinda Kalia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vrinda Kalia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vrinda Kalia more than expected).

Fields of papers citing papers by Vrinda Kalia

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Vrinda Kalia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vrinda Kalia. The network helps show where Vrinda Kalia may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Vrinda Kalia, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Vrinda Kalia Line = papers co-authored together Vrinda Kalia links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20251
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5 20243
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7 20248
8 20234
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11 20233
12 202219
13 20223
14 20228
15 20217
16 2020109
17 202013
18 20205
19 201657
20
Molecular Signature of CD8+ T Cell Exhaustion during Chronic Viral Infectionbreakdown →
20071534

About Vrinda Kalia

Vrinda Kalia is a scholar working on Aging, Health, Toxicology and Mutagenesis and Complementary and Manual Therapy, having authored 29 papers that have together received 2.0k indexed citations. Recurring topics across this work include Air Quality and Health Impacts (6 papers), Extracellular vesicles in disease (5 papers), Health, Environment, Cognitive Aging (5 papers), Metabolomics and Mass Spectrometry Studies (4 papers), Genetics, Aging, and Longevity in Model Organisms (4 papers), MicroRNA in disease regulation (4 papers), Alzheimer's disease research and treatments (3 papers) and Nutritional Studies and Diet (3 papers). The work is most often cited by research in Immunology (1.3k citations), Virology (121 citations) and Oncology (672 citations). Vrinda Kalia has collaborated with scholars based in United States, Canada and Spain. Frequent co-authors include W. Nicholas Haining, Surojit Sarkar, Joseph N. Blattman, Sang‐Jun Ha, Susan M. Kaech, Daniel L. Barber, Shruti Subramaniam, Rafi Ahmed, E. John Wherry and Gary W. Miller. Their work appears in journals such as Proceedings of the National Academy of Sciences, Environmental Science & Technology and Immunity.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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