Tomasz Stępień
Impact in
- Biological Psychiatry top 5%
- Tryptophan and brain disorders
- Neurology top 5%
- Neuroinflammation and Neurodegeneration Mechanisms
- Intracerebral and Subarachnoid Hemorrhage Research
- Cerebrovascular and genetic disorders
Papers in
- Neurology 21
- Cerebrovascular and genetic disorders 9
- Intracerebral and Subarachnoid Hemorrhage Research 8
- Neuroinflammation and Neurodegeneration Mechanisms 4
- Co-authors
- Teresa Wierzba‐Bobrowicz (40 shared papers)Eliza Lewandowska (21 shared papers)G M Szpak (15 shared papers)Elżbieta Pasennik (11 shared papers)W Lechowicz (2 shared papers)Tadeusz Mendel (8 shared papers)Sylwia Tarka (16 shared papers)B Schmidt-Sidor (6 shared papers)
- Journals
- Scientific Reports (2 papers)Folia Neuropathologica (15 papers)Cells (1 paper)Biomolecules (1 paper)Brain Research (1 paper)
- Partner nations
- PolandSwitzerlandUnited States
In The Last Decade
Tomasz Stępień
52 papers receiving 554 citations
Peers
Comparison fields: 5 of 76
- Biological Psychiatry 89
- Neurology 158
- Behavioral Neuroscience 45
- Neurology 175
- Developmental Neuroscience 33
Countries citing papers authored by Tomasz Stępień
This map shows the geographic impact of Tomasz Stępień's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomasz Stępień with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomasz Stępień more than expected).
Fields of papers citing papers by Tomasz Stępień
This network shows the impact of papers produced by Tomasz Stępień. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomasz Stępień. The network helps show where Tomasz Stępień may publish in the future.
Co-authors
The 25 scholars most cited alongside Tomasz Stępień, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 55 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Quantitative analysis of activated microglia, ramified and damage of processes in the frontal and temporal lobes of chronic schizophrenics. | 2005 | 102 |
| 2 | Astroglia disturbances during development of the central nervous system in fetuses with Down's syndrome. | 2011 | 51 |
| 3 | Small cerebral vessel disease in familial amyloid and non-amyloid angiopathies: FAD-PS-1 (P117L) mutation and CADASIL. Immunohistochemical and ultrastructural studies. | 2007 | 39 |
| 4 | 2021 | 25 | |
| 5 | Pathology of skeletal muscle cells in adult-onset glycogenosis type II (Pompe disease): ultrastructural study. | 2008 | 25 |
| 6 | Amyloid angiopathy in idiopathic Parkinson's disease. Immunohistochemical and ultrastructural study. | 2008 | 21 |
| 7 | Cerebral amyloid angiopathy as a cause of an extensive brain hemorrhage in adult patient with Down's syndrome - a case report. | 2010 | 20 |
| 8 | 2013 | 19 | |
| 9 | Capillary vessel wall in CADASIL angiopathy. | 2010 | 17 |
| 10 | Differential expression of calbindin D28k, calretinin and parvalbumin in the cerebellum of pups of ethanol-treated female rats. | 2011 | 17 |
| 11 | 2013 | 16 | |
| 12 | 2018 | 15 | |
| 13 | Brain and cerebellar hemidysplasia in a case with ipsilateral body dysplasia and suspicion of CHILD syndrome. | 2008 | 13 |
| 14 | Inflammatory cerebral amyloid angiopathy: the overlap of perivascular (PAN-like) with vasculitic (Aβ-related angiitis) form: an autopsy case. | 2011 | 13 |
| 15 | Sneddon's syndrome as a disorder of small arteries with endothelial cells proliferation: ultrastructural and neuroimaging study. | 2005 | 12 |
| 16 | 2012 | 11 | |
| 17 | Immunohistochemical and ultrastructural changes in the brain in probable adult glycogenosis type IV: adult polyglucosan body disease. | 2008 | 11 |
| 18 | 2023 | 10 | |
| 19 | Granular cell astrocytoma. A case report with immunohistochemical and ultrastructural characterization. | 2008 | 10 |
| 20 | Giant cell ependymoma of the spinal cord and fourth ventricle coexisting with syringomyelia. | 2008 | 9 |
About Tomasz Stępień
Tomasz Stępień is a scholar working on Neurology, Molecular Biology, Physiology, Epidemiology and Genetics, having authored 55 papers that have together received 562 indexed citations. Recurring topics across this work include Cerebrovascular and genetic disorders (9 papers), Alzheimer's disease research and treatments (8 papers), Intracerebral and Subarachnoid Hemorrhage Research (8 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers), Neurogenesis and neuroplasticity mechanisms (4 papers), Metabolism and Genetic Disorders (4 papers), Acute Ischemic Stroke Management (4 papers) and Lysosomal Storage Disorders Research (3 papers). The work is most often cited by research in Biological Psychiatry (89 citations), Neurology (158 citations), Behavioral Neuroscience (45 citations), Neurology (175 citations) and Developmental Neuroscience (33 citations). Tomasz Stępień has collaborated with scholars based in Poland, Switzerland and United States. Frequent co-authors include Teresa Wierzba‐Bobrowicz, Eliza Lewandowska, G M Szpak, Elżbieta Pasennik, W Lechowicz, Tadeusz Mendel, Sylwia Tarka, B Schmidt-Sidor, Hanna Mierzewska and Agnieszka Ługowska. Their work appears in journals such as Scientific Reports, Folia Neuropathologica, Cells, Biomolecules and Brain Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.