Tim De Pooter
- Neurology top 2%
- Molecular Biology
- Physiology top 10%
- Cellular and Molecular Neuroscience top 10%
- Neurology top 5%
- Co-authors
- Christine Van BroeckhovenKristel SleegersSebastiaan EngelborghsJulie van der ZeePeter De RijkMarleen Van den BroeckPeter Paul De DeynMojca Stražišar
- Topics
- Parkinson's Disease Mechanisms and Treatments (7 papers)Alzheimer's disease research and treatments (5 papers)Neurological diseases and metabolism (4 papers)
- Cited by
- NeurologyPhysiology
- Journals
- BioinformaticsBrainNeurology
- Partner nations
- BelgiumNetherlandsUnited States
In The Last Decade
Tim De Pooter
24 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 83
- Neurology 556
- Molecular Biology 464
- Physiology 361
- Cellular and Molecular Neuroscience 197
- Neurology 197
Countries citing papers authored by Tim De Pooter
This map shows the geographic impact of Tim De Pooter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tim De Pooter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tim De Pooter more than expected).
Fields of papers citing papers by Tim De Pooter
This network shows the impact of papers produced by Tim De Pooter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tim De Pooter. The network helps show where Tim De Pooter may publish in the future.
Co-authorship network of co-authors of Tim De Pooter
This figure shows the co-authorship network connecting the top 25 collaborators of Tim De Pooter. A scholar is included among the top collaborators of Tim De Pooter based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tim De Pooter. Tim De Pooter is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 5 | |
| 3 | 14 | |
| 4 | 10 | |
| 5 | 7 | |
| 6 | 25 | |
| 7 | 100 | |
| 8 | 68 | |
| 9 | 9 | |
| 10 | 38 | |
| 11 | 72 | |
| 12 | 128 | |
| 13 | Neuronal inclusion protein TDP-43 has no primary genetic role in FTD and ALS | 3 |
| 14 | 121 | |
| 15 | 60 | |
| 16 | 145 | |
| 17 | 1 | |
| 18 | 35 | |
| 19 | 63 | |
| 20 | 162 |
About Tim De Pooter
Tim De Pooter is a scholar working on Molecular Medicine, Neurology and Endocrinology, having authored 24 papers that have together received 1.1k indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (7 papers), Alzheimer's disease research and treatments (5 papers) and Neurological diseases and metabolism (4 papers). The work is most often cited by research in Neurology (556 citations), Neurology (197 citations) and Physiology (361 citations). Tim De Pooter has collaborated with scholars based in Belgium, Netherlands and United States. Frequent co-authors include Christine Van Broeckhoven, Kristel Sleegers, Sebastiaan Engelborghs, Julie van der Zee, Peter De Rijk, Marleen Van den Broeck, Peter Paul De Deyn, Mojca Stražišar, Bart Dermaut and Marc Cruts. Their work appears in journals such as Bioinformatics, Brain and Neurology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.