Thomas Pulinilkunnil

8.6k total citations · 1 hit paper
80 papers, 4.7k citations indexed

About

Thomas Pulinilkunnil is a scholar working on Molecular Biology, Physiology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Thomas Pulinilkunnil has authored 80 papers receiving a total of 4.7k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Molecular Biology, 38 papers in Physiology and 24 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Thomas Pulinilkunnil's work include Adipose Tissue and Metabolism (28 papers), Metabolism, Diabetes, and Cancer (17 papers) and Cardiovascular Function and Risk Factors (15 papers). Thomas Pulinilkunnil is often cited by papers focused on Adipose Tissue and Metabolism (28 papers), Metabolism, Diabetes, and Cancer (17 papers) and Cardiovascular Function and Risk Factors (15 papers). Thomas Pulinilkunnil collaborates with scholars based in Canada, United States and Austria. Thomas Pulinilkunnil's co-authors include Petra C. Kienesberger, Purvi Trivedi, J. Bartlett, Jason R.B. Dyck, Brian Rodrigues, Ashraf Abrahani, Dake Qi, An Ding, Sanjoy Ghosh and Jeevan Nagendran and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Thomas Pulinilkunnil

79 papers receiving 4.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Single phosphorylation sites in Acc1 and Acc2 regulate lipid homeostasis and the insulin-sensitizing effects of metformin

2013 671 citations Sandra Galić, Thomas Pulinilkunnil et al. profile →

Peers

Countries citing papers authored by Thomas Pulinilkunnil

Since Specialization

Citations

This map shows the geographic impact of Thomas Pulinilkunnil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Pulinilkunnil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Pulinilkunnil more than expected).

Fields of papers citing papers by Thomas Pulinilkunnil

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Pulinilkunnil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Pulinilkunnil. The network helps show where Thomas Pulinilkunnil may publish in the future.

Co-authorship network of co-authors of Thomas Pulinilkunnil

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Pulinilkunnil. A scholar is included among the top collaborators of Thomas Pulinilkunnil based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Pulinilkunnil. Thomas Pulinilkunnil is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Yap Is a Nutrient Sensor Sensitive to the Amino Acid L-Isoleucine and Regulates the Expression of Ctgf in Cardiomyocytes 2024 ·Biomolecules ·Victoria L. Nelson, (unknown), Lester J. Pérez, (unknown), Mathew J. Platt, (unknown), Thomas Pulinilkunnil, Petra C. Kienesberger, Jeremy A. Simpson, Keith R. Brunt	1
2	Supplementation of cyanidin-3-O-β-glucoside-rich haskap (Lonicera caerulea L.) berry extract attenuates hepatic lipid dysregulation in diet-induced obese mice 2023 ·Journal of Functional Foods ·Dipsikha Biswas, (unknown), (unknown), (unknown), Petra C. Kienesberger, Morgan G. I. Langille, H.P. Vasantha Rupasinghe, Thomas Pulinilkunnil	7
3	Inhibiting BCKDK in triple negative breast cancer suppresses protein translation, impairs mitochondrial function, and potentiates doxorubicin cytotoxicity 2021 ·Cell Death Discovery ·Dipsikha Biswas, (unknown), (unknown), (unknown), Khoi Dao, (unknown), (unknown), Yassine El Hiani, Petra C. Kienesberger, Thomas Pulinilkunnil	28
4	Deletion of BCATm increases insulin-stimulated glucose oxidation in the heart 2021 ·Metabolism ·Golam M. Uddin, Qutuba G. Karwi, Simran Pherwani, Keshav Gopal, Cory S. Wagg, Dipsikha Biswas, (unknown), (unknown), Guoqing Wu, Liyan Zhang, Kim L. Ho, Thomas Pulinilkunnil, John R. Ussher, Gary D. Lopaschuk	30
5	Branched-chain ketoacid overload inhibits insulin action in the muscle 2020 ·Journal of Biological Chemistry ·Dipsikha Biswas, Khoi Dao, (unknown), Andrew Cowie, (unknown), Yassine El Hiani, Petra C. Kienesberger, Thomas Pulinilkunnil	36
6	Hemodynamic Assessment and In vivo Catabolism of Adenosine 5’-triphosphate in Doxorubicin or Isoproterenol-induced Cardiovascular Toxicity 2020 ·Drug Metabolism Letters ·Pollen Yeung, (unknown), (unknown), (unknown), Remigius U. Agu, Thomas Pulinilkunnil	1
7	Lysosomal Biology and Function: Modern View of Cellular Debris Bin 2020 ·Cells ·Purvi Trivedi, J. Bartlett, Thomas Pulinilkunnil	190
8	Whey Peptides Stimulate Differentiation and Lipid Metabolism in Adipocytes and Ameliorate Lipotoxicity-Induced Insulin Resistance in Muscle Cells 2020 ·Nutrients ·Kenneth D’Souza, (unknown), (unknown), Thomas Pulinilkunnil, Chibuike C. Udenigwe, Petra C. Kienesberger	31
9	A lysosome independent role for TFEB in activating DNA repair and inhibiting apoptosis in breast cancer cells 2019 ·Biochemical Journal ·(unknown), Dipsikha Biswas, (unknown), Yassine El Hiani, Petra C. Kienesberger, Thomas Pulinilkunnil	33
10	Validation of optimal reference genes for quantitative real time PCR in muscle and adipose tissue for obesity and diabetes research 2017 ·Scientific Reports ·Lester J. Pérez, Liliam Ríos, Purvi Trivedi, Kenneth D’Souza, Andrew Cowie, (unknown), Duncan Webster, Keith R. Brunt, Jean‐François Légaré, Ansar Hassan, Petra C. Kienesberger, Thomas Pulinilkunnil	45
11	Autophagic dysregulation in doxorubicin cardiomyopathy 2017 ·Journal of Molecular and Cellular Cardiology ·J. Bartlett, Purvi Trivedi, Thomas Pulinilkunnil	181
12	Dieldrin Augments mTOR Signaling and Regulates Genes Associated with Cardiovascular Disease in the Adult Zebrafish Heart (Danio rerio) 2017 ·Journal of Pharmacology and Experimental Therapeutics ·(unknown), Andrew Cowie, Christopher J. Martyniuk, Petra C. Kienesberger, Thomas Pulinilkunnil	11
13	Glucolipotoxicity diminishes cardiomyocyte TFEB and inhibits lysosomal autophagy during obesity and diabetes 2016 ·Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids ·Purvi Trivedi, J. Bartlett, Lester J. Pérez, Keith R. Brunt, Jean Légaré, Ansar Hassan, Petra C. Kienesberger, Thomas Pulinilkunnil	62
14	Skeletal muscle ACC2 S212 phosphorylation is not required for the control of fatty acid oxidation during exercise 2015 ·Physiological Reports ·Hayley M. O’Neill, James Lally, Sandra Galić, Thomas Pulinilkunnil, Rebecca J. Ford, Jason R.B. Dyck, Bryce J. van Denderen, Bruce E. Kemp, Gregory R. Steinberg	28
15	Short Communication: Ischemia/Reperfusion Tolerance Is Time-of-Day–Dependent 2009 ·Circulation Research ·David J. Durgan, Thomas Pulinilkunnil, Carolina Villegas‐Montoya, (unknown), Nikolaos G. Frangogiannis, Lloyd H. Michael, Chi-Wing Chow, Jason R.B. Dyck, Martin E. Young	197
16	Neuronal Protein Tyrosine Phosphatase 1B Deficiency Results in Inhibition of Hypothalamic AMPK and Isoform-Specific Activation of AMPK in Peripheral Tissues 2009 ·Molecular and Cellular Biology ·Bingzhong Xue, Thomas Pulinilkunnil, Incoronata Murano, Kendra K. Bence, Huamei He, Yasuhiko Minokoshi, Kenji Asakura, Anna Lee, Fawaz G. Haj, Noboru Furukawa, Karyn J. Catalano, Mirela Delibegović, James A. Balschi, Saverio Cinti, Benjamin G. Neel, Barbara B. Kahn	64
17	Direct Regulation of Myocardial Triglyceride Metabolism by the Cardiomyocyte Circadian Clock 2009 ·Journal of Biological Chemistry ·(unknown), Petra C. Kienesberger, Thomas Pulinilkunnil, Mary H. Sailors, David J. Durgan, Carolina Villegas‐Montoya, (unknown), Raquel Muñoz-González, (unknown), Brandon B. Boland, (unknown), Tracy A McElfresh, (unknown), Chi-Wing Chow, William C. Heird, Margaret P. Chandler, Jason R.B. Dyck, Molly S. Bray, Martin E. Young	91
18	Altered cardiac fatty acid composition and utilization following dexamethasone-induced insulin resistance 2006 ·American Journal of Physiology-Endocrinology and Metabolism ·Dake Qi, An Ding, Girish Kewalramani, (unknown), Thomas Pulinilkunnil, Ashraf Abrahani, (unknown), Sanjoy Ghosh, Richard B. Wambolt, Michael F. Allard, Sheila M. Innis, Brian Rodrigues	21
19	Cardiomyocyte apoptosis induced by short-term diabetes requires mitochondrial GSH depletion 2005 ·American Journal of Physiology-Heart and Circulatory Physiology ·Sanjoy Ghosh, Thomas Pulinilkunnil, (unknown), Girish Kewalramani, An Ding, Dake Qi, Ashraf Abrahani, Brian Rodrigues	107
20	Circulating triglyceride lipolysis facilitates lipoprotein lipase translocation from cardiomyocyte to myocardial endothelial lining 2003 ·Cardiovascular Research ·Thomas Pulinilkunnil	32

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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