Thomas M. Cocks

6.2k total citations · 1 hit paper
79 papers, 5.3k citations indexed

About

Thomas M. Cocks is a scholar working on Physiology, Cardiology and Cardiovascular Medicine and Molecular Biology. According to data from OpenAlex, Thomas M. Cocks has authored 79 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Physiology, 30 papers in Cardiology and Cardiovascular Medicine and 21 papers in Molecular Biology. Recurrent topics in Thomas M. Cocks's work include Nitric Oxide and Endothelin Effects (38 papers), Blood Coagulation and Thrombosis Mechanisms (21 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (17 papers). Thomas M. Cocks is often cited by papers focused on Nitric Oxide and Endothelin Effects (38 papers), Blood Coagulation and Thrombosis Mechanisms (21 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (17 papers). Thomas M. Cocks collaborates with scholars based in Australia, United Kingdom and Sri Lanka. Thomas M. Cocks's co-authors include James A. Angus, Geoffrey Burnstock, James D. Moffatt, B. Kemp, Justin R. Hamilton, C J Garland, Frances Plane, R. Crowe, Albert G. Frauman and Gordon Campbell and has published in prestigious journals such as Nature, Gastroenterology and Circulation Research.

In The Last Decade

Thomas M. Cocks

79 papers receiving 5.0k citations

Hit Papers

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What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Endothelium-dependent relaxation of coronary arteries by noradrenaline and serotonin

1983 727 citations Thomas M. Cocks, James A. Angus profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
Thomas M. Cocks	2.6k	1.5k	1.4k	1.1k	943	79	5.3k
Hideo Kanaide	2.2k 0.8×	1.6k 1.1×	3.1k 2.2×	929 0.9×	496 0.5×	218	6.0k
Pedro D’Orléans-Juste	3.5k 1.4×	2.1k 1.4×	3.2k 2.3×	2.1k 2.0×	133 0.1×	220	7.5k
Dewan Zeng	683 0.3×	1.1k 0.7×	1.5k 1.1×	763 0.7×	1.5k 1.6×	55	4.9k
Albert Smolenski	1.8k 0.7×	1.4k 0.9×	2.6k 1.9×	311 0.3×	556 0.6×	67	5.0k
Nina Wettschureck	1.4k 0.5×	1.1k 0.7×	3.7k 2.6×	708 0.7×	319 0.3×	87	6.7k
Hiroshi Nonoguchi	1.1k 0.4×	1.0k 0.7×	2.5k 1.8×	193 0.2×	374 0.4×	144	5.2k
Anna‐Leena Sirén	980 0.4×	350 0.2×	1.9k 1.3×	1.2k 1.1×	1.6k 1.7×	146	6.3k
Ryuichi Hattori	1.0k 0.4×	964 0.7×	838 0.6×	492 0.5×	518 0.5×	83	3.8k
Gaétan Thibault	1.4k 0.6×	5.4k 3.6×	3.4k 2.4×	791 0.8×	129 0.1×	203	8.8k
Gervaise Loirand	1.4k 0.5×	1.6k 1.1×	3.6k 2.6×	861 0.8×	102 0.1×	133	6.5k

Countries citing papers authored by Thomas M. Cocks

Since Specialization

Citations

This map shows the geographic impact of Thomas M. Cocks's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas M. Cocks with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas M. Cocks more than expected).

Fields of papers citing papers by Thomas M. Cocks

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas M. Cocks. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas M. Cocks. The network helps show where Thomas M. Cocks may publish in the future.

Co-authorship network of co-authors of Thomas M. Cocks

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas M. Cocks. A scholar is included among the top collaborators of Thomas M. Cocks based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas M. Cocks. Thomas M. Cocks is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Lohman, Rink‐Jan, Terence J. O’Brien, & Thomas M. Cocks. (2008). Protease-activated receptor-2 regulates trypsin expression in the brain and protects against seizures and epileptogenesis. Neurobiology of Disease. 30(1). 84–93. 26 indexed citations

Devlin, Mark, et al.. (2007). Hepta and octapeptide agonists of protease‐activated receptor 2. Journal of Peptide Science. 13(12). 856–861. 8 indexed citations

Selemidis, Stavros & Thomas M. Cocks. (2007). Smooth muscle mediates circumferential conduction of hyperpolarization and relaxation to focal endothelial cell activation in large coronary arteries. Naunyn-Schmiedeberg s Archives of Pharmacology. 375(2). 85–94. 4 indexed citations

Fairlie, David P., et al.. (2006). Protease-Activated Receptor-2 Peptides Activate Neurokinin-1 Receptors in the Mouse Isolated Trachea. Journal of Pharmacology and Experimental Therapeutics. 317(2). 598–605. 21 indexed citations

Cocks, Thomas M., et al.. (2006). Lipopolysaccharide Induces Epithelium- and Prostaglandin E2-Dependent Relaxation of Mouse Isolated Trachea through Activation of Cyclooxygenase (COX)-1 and COX-2. Journal of Pharmacology and Experimental Therapeutics. 317(2). 806–812. 27 indexed citations

Moffatt, James D., Thomas M. Cocks, & Clive Page. (2004). Role of the epithelium and acetylcholine in mediating the contraction to 5‐hydroxytryptamine in the mouse isolated trachea. British Journal of Pharmacology. 141(7). 1159–1166. 52 indexed citations

Hamilton, Justin R., James D. Moffatt, James Tatoulis, & Thomas M. Cocks. (2002). Enzymatic activation of endothelial protease‐activated receptors is dependent on artery diameter in human and porcine isolated coronary arteries. British Journal of Pharmacology. 136(4). 492–501. 18 indexed citations

Selemidis, Stavros & Thomas M. Cocks. (2002). Endothelium-dependent hyperpolarization as a remote anti-atherogenic mechanism. Trends in Pharmacological Sciences. 23(5). 213–220. 21 indexed citations

Hamilton, Justin R., James D. Moffatt, Albert G. Frauman, & Thomas M. Cocks. (2001). Protease-Activated Receptor (PAR) 1 but Not PAR2 or PAR4 Mediates Endothelium-Dependent Relaxation to Thrombin and Trypsin in Human Pulmonary Arteries. Journal of Cardiovascular Pharmacology. 38(1). 108–119. 51 indexed citations

10.

Drummond, Grant R., Stavros Selemidis, & Thomas M. Cocks. (2000). Apamin‐sensitive, non‐nitric oxide (NO) endothelium‐dependent relaxations to bradykinin in the bovine isolated coronary artery: no role for cytochrome P₄₅₀ and K⁺. British Journal of Pharmacology. 129(4). 811–819. 26 indexed citations

11.

Hamilton, Justin R. & Thomas M. Cocks. (2000). Heterogeneous mechanisms of endothelium‐dependent relaxation for thrombin and peptide activators of protease‐activated receptor‐1 in porcine isolated coronary artery. British Journal of Pharmacology. 130(1). 181–188. 44 indexed citations

12.

Kemp, B., Joseph J. Smolich, & Thomas M. Cocks. (1997). Evidence for specific regional patterns of responses to different vasoconstrictors and vasodilators in sheep isolated pulmonary arteries and veins. British Journal of Pharmacology. 121(3). 441–450. 27 indexed citations

13.

Selemidis, Stavros, D.G. Satchell, & Thomas M. Cocks. (1997). Evidence that NO acts as a redundant NANC inhibitory neurotransmitter in the guinea‐pig isolated taenia coli. British Journal of Pharmacology. 121(3). 604–611. 20 indexed citations

14.

Drummond, Grant R. & Thomas M. Cocks. (1996). Evidence for mediation by endothelium‐derived hyperpolarizing factor of relaxation to bradykinin in the bovine isolated coronary artery independently of voltage‐operated Ca²⁺ channels. British Journal of Pharmacology. 117(6). 1035–1040. 35 indexed citations

15.

Drummond, Grant R. & Thomas M. Cocks. (1995). SPECIAL REPORT Endothelium‐dependent relaxation to the B₁ kinin receptor agonist des‐Arg ‐bradykinin in human coronary arteries. British Journal of Pharmacology. 116(8). 3083–3085. 36 indexed citations

16.

Cocks, Thomas M., et al.. (1992). 5‐Hydroxytryptamine (5‐HT) mediates potent relaxation in the sheep isolated pulmonary vein via activation of 5‐HT₄ receptors. British Journal of Pharmacology. 107(2). 591–596. 36 indexed citations

17.

Cocks, Thomas M. & James A. Angus. (1991). Evidence That Contractions of Isolated Arteries by L-NMMA and NOLA Are Not Due to Inhibition of Basal EDRF Release. Journal of Cardiovascular Pharmacology. 17(Supplement). 159???164–159???164. 10 indexed citations

18.

Cocks, Thomas M., Errol Malta, Susannah King, Robyn L. Woods, & James A. Angus. (1991). Oxyhaemoglobin increases the production of endothelin-1 by endothelial cells in culture. European Journal of Pharmacology. 196(2). 177–182. 56 indexed citations

19.

Cocks, Thomas M., A. Broughton, Mobin Dib, Krishnankutty Sudhir, & James A. Angus. (1989). ENDOTHELIN IS BLOOD VESSEL SELECTIVE: STUDIES ON A VARIETY OF HUMAN AND DOG VESSELS IN VITRO AND ON REGIONAL BLOOD FLOW IN THE CONSCIOUS RABBIT. Clinical and Experimental Pharmacology and Physiology. 16(4). 243–246. 35 indexed citations

20.

Cocks, Thomas M., J A Manderson, Peter R.L. Mosse, Gordon Campbell, & James A. Angus. (1987). Development of a Large Fibromuscular Intimal Thickening Does Not Impair Endothelium-Dependent Relaxation in the Rabbit Carotid Artery. Journal of Vascular Research. 24(4). 192–200. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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