Thomas Koed Doktor

1.8k total citations
33 papers, 1.1k citations indexed

About

Thomas Koed Doktor is a scholar working on Molecular Biology, Genetics and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Thomas Koed Doktor has authored 33 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 9 papers in Genetics and 3 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Thomas Koed Doktor's work include RNA Research and Splicing (20 papers), RNA modifications and cancer (14 papers) and Neurogenetic and Muscular Disorders Research (9 papers). Thomas Koed Doktor is often cited by papers focused on RNA Research and Splicing (20 papers), RNA modifications and cancer (14 papers) and Neurogenetic and Muscular Disorders Research (9 papers). Thomas Koed Doktor collaborates with scholars based in Denmark, United States and Switzerland. Thomas Koed Doktor's co-authors include Brage Storstein Andresen, Gitte Hoffmann Bruun, Henriette S. Andersen, Kinji Ohno, Akio Masuda, Adrian R. Krainer, Lisbeth Dahl Schroeder, Anders Etzerodt, Mikako Ito and Marcello Delfini and has published in prestigious journals such as Nature, Nucleic Acids Research and The Journal of Experimental Medicine.

In The Last Decade

Thomas Koed Doktor

31 papers receiving 1.1k citations

Peers

Thomas Koed Doktor

IMMUN

GENET

ONCOL

Julio Castaño Spain

Adam S. Lazorchak United States

Borislav Stoyanov Germany

Andy Watt United States

Reinhard Grausenburger Austria

Peter Wend United States

Ichiro Okabe Japan

Caterina Marchetti Italy

Susan F. Law United States

Erikjan Rijkers Netherlands

Julio Castaño Spain

Thomas Koed Doktor

1.2k ×1.4

105 ×0.6

IMMUN

74 ×0.4

GENET

176 ×1.6

ONCOL

93 ×1.0

Citations per year, relative to Thomas Koed Doktor Thomas Koed Doktor (= 1×) peers Julio Castaño

Countries citing papers authored by Thomas Koed Doktor

Since Specialization

Citations

This map shows the geographic impact of Thomas Koed Doktor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Koed Doktor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Koed Doktor more than expected).

Fields of papers citing papers by Thomas Koed Doktor

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Koed Doktor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Koed Doktor. The network helps show where Thomas Koed Doktor may publish in the future.

Co-authorship network of co-authors of Thomas Koed Doktor

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Koed Doktor. A scholar is included among the top collaborators of Thomas Koed Doktor based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Koed Doktor. Thomas Koed Doktor is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Doktor, Thomas Koed, et al.. (2024). All exons are not created equal—exon vulnerability determines the effect of exonic mutations on splicing. Nucleic Acids Research. 52(8). 4588–4603. 8 indexed citations

Guerra, Bárbara, Thomas Koed Doktor, Aarif Siddiqui, et al.. (2024). Protein kinase CK2 sustains de novo fatty acid synthesis by regulating the expression of SCD-1 in human renal cancer cells. Cancer Cell International. 24(1). 432–432.

Staunstrup, Nicklas Heine, Guillaume Flandin, Tina Fuglsang Daugaard, et al.. (2023). Genome-wide epigenetic and mRNA-expression profiling followed by CRISPR/Cas9-mediated gene-disruptions corroborate the MIR141/MIR200C-ZEB1/ZEB2-FGFR1 axis in acquired EMT-associated EGFR TKI-resistance in NSCLC cells. Translational Lung Cancer Research. 12(1). 42–65. 9 indexed citations

Dembić, Maja, Eva Richard, Jesper F. Havelund, et al.. (2023). Regulating PCCA gene expression by modulation of pseudoexon splicing patterns to rescue enzyme activity in propionic acidemia. Molecular Therapy — Nucleic Acids. 35(1). 102101–102101. 7 indexed citations

Leal, Fátima, Thomas Koed Doktor, Beat Thöny, et al.. (2022). Antisense Oligonucleotide Rescue of Deep-Intronic Variants Activating Pseudoexons in the 6-Pyruvoyl-Tetrahydropterin Synthase Gene. Nucleic Acid Therapeutics. 32(5). 378–390. 11 indexed citations

Doktor, Thomas Koed, Anne Louise Askou, Ulvi Ahmadov, et al.. (2022). VEGFA-targeting miR-agshRNAs combine efficacy with specificity and safety for retinal gene therapy. Molecular Therapy — Nucleic Acids. 28. 58–76. 13 indexed citations

Doktor, Thomas Koed, et al.. (2020). DeepCLIP: predicting the effect of mutations on protein–RNA binding with deep learning. Nucleic Acids Research. 48(13). 7099–7118. 63 indexed citations

Etzerodt, Anders, Thomas Koed Doktor, Marcello Delfini, et al.. (2020). Tissue-resident macrophages in omentum promote metastatic spread of ovarian cancer. The Journal of Experimental Medicine. 217(4). 208 indexed citations

Doktor, Thomas Koed, et al.. (2019). Down-regulation of CK2α correlates with decreased expression levels of DNA replication minichromosome maintenance protein complex (MCM) genes. Scientific Reports. 9(1). 14581–14581. 7 indexed citations

10.

Dembić, Maja, Henriette S. Andersen, Jean Bastin, et al.. (2018). Next generation sequencing of RNA reveals novel targets of resveratrol with possible implications for Canavan disease. Molecular Genetics and Metabolism. 126(1). 64–76. 14 indexed citations

11.

Doktor, Thomas Koed, et al.. (2017). DFI-seq identification of environment-specific gene expression in uropathogenic Escherichia coli. BMC Microbiology. 17(1). 99–99. 5 indexed citations

12.

Doktor, Thomas Koed, Yimin Hua, Henriette S. Andersen, et al.. (2016). RNA-sequencing of a mouse-model of spinal muscular atrophy reveals tissue-wide changes in splicing of U12-dependent introns. Nucleic Acids Research. 45(1). 395–416. 82 indexed citations

13.

Bruun, Gitte Hoffmann, Thomas Koed Doktor, Akio Masuda, et al.. (2016). Global identification of hnRNP A1 binding sites for SSO-based splicing modulation. BMC Biology. 14(1). 54–54. 56 indexed citations

14.

Doktor, Thomas Koed, et al.. (2014). Absence of an Intron Splicing Silencer in Porcine Smn1 Intron 7 Confers Immunity to the Exon Skipping Mutation in Human SMN2. PLoS ONE. 9(6). e98841–e98841. 5 indexed citations

15.

Bruun, Gitte Hoffmann, Thomas Koed Doktor, & Brage Storstein Andresen. (2013). A synonymous polymorphic variation in ACADM exon 11 affects splicing efficiency and may affect fatty acid oxidation. Molecular Genetics and Metabolism. 110(1-2). 122–128. 20 indexed citations

16.

Andersen, Henriette S., et al.. (2010). The PAH c.30C>G synonymous variation (p.G10G) creates a common exonic splicing silencer. Molecular Genetics and Metabolism. 100. 1 indexed citations

17.

Zavadáková, Petra, et al.. (2010). The deep intronic c.903+469T>C mutation in the MTRR gene creates an SF2/ASF binding exonic splicing enhancer, which leads to pseudoexon activation and causes the cblE type of homocystinuria. Human Mutation. 31(4). 437–444. 45 indexed citations

18.

Doktor, Thomas Koed, Lisbeth Dahl Schroeder, Anne Vested, et al.. (2010). SMN2 exon 7 splicing is inhibited by binding of hnRNP A1 to a common ESS motif that spans the 3′ splice site. Human Mutation. 32(2). 220–230. 36 indexed citations

19.

Dobrowolski, Steven F., Henriette S. Andersen, Thomas Koed Doktor, & Brage Storstein Andresen. (2010). The phenylalanine hydroxylase c.30C>G synonymous variation (p.G10G) creates a common exonic splicing silencer. Molecular Genetics and Metabolism. 100(4). 316–323. 18 indexed citations

20.

Nielsen, Karsten, Suzette Sørensen, Luca Cartegni, et al.. (2007). Seemingly Neutral Polymorphic Variants May Confer Immunity to Splicing-Inactivating Mutations: A Synonymous SNP in Exon 5 of MCAD Protects from Deleterious Mutations in a Flanking Exonic Splicing Enhancer. The American Journal of Human Genetics. 80(3). 416–432. 117 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact