Thomas J. Gardella

9.5k total citations
129 papers, 6.7k citations indexed

About

Thomas J. Gardella is a scholar working on Molecular Biology, Oncology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Thomas J. Gardella has authored 129 papers receiving a total of 6.7k indexed citations (citations by other indexed papers that have themselves been cited), including 112 papers in Molecular Biology, 72 papers in Oncology and 16 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Thomas J. Gardella's work include Bone health and treatments (67 papers), Protein Kinase Regulation and GTPase Signaling (50 papers) and Receptor Mechanisms and Signaling (36 papers). Thomas J. Gardella is often cited by papers focused on Bone health and treatments (67 papers), Protein Kinase Regulation and GTPase Signaling (50 papers) and Receptor Mechanisms and Signaling (36 papers). Thomas J. Gardella collaborates with scholars based in United States, Japan and United Kingdom. Thomas J. Gardella's co-authors include Harald Jüppner, Jean‐Pierre Vilardaga, John T. Potts, Thomas Dean, Miriam M. Susskind, Michael D. Luck, Henry M. Kronenberg, Robert Gensure, Sébastien Ferrandon and Michael Mannstadt and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Biological Chemistry.

In The Last Decade

Thomas J. Gardella

126 papers receiving 6.6k citations

Peers

Thomas J. Gardella
Jolene J. Windle United States
Anthony J. Mason United States
Ken–ichi Yamamura Japan
Ned R. Siegel United States
Rodrigo Bravo United States
Paul K. Goldsmith United States
Hiroshi Ohnishi Japan
Kei Tashiro Japan
Tim M. Townes United States
Ned Mantei Switzerland
Jolene J. Windle United States
Thomas J. Gardella
Citations per year, relative to Thomas J. Gardella Thomas J. Gardella (= 1×) peers Jolene J. Windle

Countries citing papers authored by Thomas J. Gardella

Since Specialization
Citations

This map shows the geographic impact of Thomas J. Gardella's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas J. Gardella with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas J. Gardella more than expected).

Fields of papers citing papers by Thomas J. Gardella

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas J. Gardella. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas J. Gardella. The network helps show where Thomas J. Gardella may publish in the future.

Co-authorship network of co-authors of Thomas J. Gardella

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas J. Gardella. A scholar is included among the top collaborators of Thomas J. Gardella based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas J. Gardella. Thomas J. Gardella is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Reyes, Monica, Patrick Hanna, Prem Swaroop Yadav, et al.. (2025). A mouse model of Jansen’s metaphyseal chondrodysplasia for investigating disease mechanisms and candidate therapeutics. Proceedings of the National Academy of Sciences. 122(23). e2500176122–e2500176122. 2 indexed citations
2.
Che, Xiangguo, Hyun‐Ju Kim, Soo Young Lee, et al.. (2025). Dimeric R25CPTH(1–34) activates the parathyroid hormone-1 receptor in vitro and stimulates bone formation in osteoporotic female mice. eLife. 13. 1 indexed citations
3.
Calder, Alistair, Jeremy Allgrove, Moira Cheung, et al.. (2024). Eiken syndrome with parathyroid hormone resistance due to a novel parathyroid hormone receptor type 1 mutation: clinical features and functional analysis. Journal of Bone and Mineral Research. 39(11). 1596–1605. 3 indexed citations
4.
Liu, Shi, Lauren My-Linh Tran, Zhen Yu, et al.. (2024). Backbone Modification Provides a Long-Acting Inverse Agonist of Pathogenic, Constitutively Active PTH1R Variants. Journal of the American Chemical Society. 146(10). 6522–6529. 2 indexed citations
5.
Hanna, Patrick, Ashok Khatri, Séverine Brabant, et al.. (2023). Homozygous Ser-1 to Pro-1 mutation in parathyroid hormone identified in hypocalcemic patients results in secretion of a biologically inactive pro-hormone. Proceedings of the National Academy of Sciences. 120(8). e2208047120–e2208047120. 3 indexed citations
6.
Dean, Thomas, Ross W. Cheloha, Jean‐Pierre Vilardaga, et al.. (2023). Altered Signaling and Desensitization Responses in PTH1R Mutants Associated with Eiken Syndrome. Communications Biology. 6(1). 599–599. 8 indexed citations
7.
Peña, Karina A., Alex D. White, Frédéric Jean‐Alphonse, et al.. (2022). Biased GPCR signaling by the native parathyroid hormone–related protein 1 to 141 relative to its N-terminal fragment 1 to 36. Journal of Biological Chemistry. 298(9). 102332–102332. 10 indexed citations
8.
Clark, Lisa J., James Krieger, Alex D. White, et al.. (2020). Allosteric interactions in the parathyroid hormone GPCR–arrestin complex formation. Nature Chemical Biology. 16(10). 1096–1104. 39 indexed citations
9.
Cheloha, Ross W., et al.. (2020). Improved GPCR ligands from nanobody tethering. Nature Communications. 11(1). 2087–2087. 45 indexed citations
10.
Biebermann, Heike, Gunnar Kleinau, Dirk Schnabel, et al.. (2018). A New Multisystem Disorder Caused by the Gαs Mutation p.F376V. The Journal of Clinical Endocrinology & Metabolism. 104(4). 1079–1089. 10 indexed citations
11.
Hattersley, Gary, Thomas Dean, Braden Corbin, Hila Bahar, & Thomas J. Gardella. (2015). Binding Selectivity of Abaloparatide for PTH-Type-1-Receptor Conformations and Effects on Downstream Signaling. Endocrinology. 157(1). 141–149. 214 indexed citations
12.
Gardella, Thomas J. & Jean‐Pierre Vilardaga. (2015). International Union of Basic and Clinical Pharmacology. XCIII. The Parathyroid Hormone Receptors—Family B G Protein–Coupled Receptors. Pharmacological Reviews. 67(2). 310–337. 81 indexed citations
13.
Cheloha, Ross W., Akira Maeda, Thomas Dean, Thomas J. Gardella, & Samuel H. Gellman. (2014). Backbone modification of a polypeptide drug alters duration of action in vivo. Nature Biotechnology. 32(7). 653–655. 101 indexed citations
14.
Vilardaga, Jean‐Pierre, Thomas J. Gardella, Vanessa L. Wehbi, & Timothy N. Feinstein. (2012). Non-canonical signaling of the PTH receptor. Trends in Pharmacological Sciences. 33(8). 423–431. 40 indexed citations
15.
Okazaki, Makoto, Sébastien Ferrandon, Jean‐Pierre Vilardaga, et al.. (2008). Prolonged signaling at the parathyroid hormone receptor by peptide ligands targeted to a specific receptor conformation. Proceedings of the National Academy of Sciences. 105(43). 16525–16530. 118 indexed citations
16.
Carter, Percy H. & Thomas J. Gardella. (2001). Zinc(II)-mediated enhancement of the agonist activity of histidine-substituted parathyroid hormone(1–14) analogues. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1538(2-3). 290–304. 4 indexed citations
17.
Shimizu, Masaru, John T. Potts, & Thomas J. Gardella. (2000). Minimization of Parathyroid Hormone. Journal of Biological Chemistry. 275(29). 21836–21843. 78 indexed citations
18.
Bergwitz, Clemens, et al.. (1997). Residues in the Membrane-spanning and Extracellular Loop Regions of the Parathyroid Hormone (PTH)-2 Receptor Determine Signaling Selectivity for PTH and PTH-related Peptide. Journal of Biological Chemistry. 272(46). 28861–28868. 70 indexed citations
19.
20.
Gardella, Thomas J., et al.. (1996). Transmembrane Residues of the Parathyroid Hormone (PTH)/PTH-related Peptide Receptor That Specifically Affect Binding and Signaling by Agonist Ligands. Journal of Biological Chemistry. 271(22). 12820–12825. 64 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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