Thomas J. Esparza

2.7k total citations
34 papers, 2.0k citations indexed

About

Thomas J. Esparza is a scholar working on Physiology, Molecular Biology and Neurology. According to data from OpenAlex, Thomas J. Esparza has authored 34 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 12 papers in Molecular Biology and 8 papers in Neurology. Recurrent topics in Thomas J. Esparza's work include Alzheimer's disease research and treatments (15 papers), Traumatic Brain Injury and Neurovascular Disturbances (7 papers) and Neuroscience and Neuropharmacology Research (4 papers). Thomas J. Esparza is often cited by papers focused on Alzheimer's disease research and treatments (15 papers), Traumatic Brain Injury and Neurovascular Disturbances (7 papers) and Neuroscience and Neuropharmacology Research (4 papers). Thomas J. Esparza collaborates with scholars based in United States, Italy and Australia. Thomas J. Esparza's co-authors include David L. Brody, David M. Holtzman, Randall J. Bateman, Nigel J. Cairns, Nino Stocchetti, Sandra Magnoni, John R. Cirrito, Norelle C. Wildburger, Gregory J. Zipfel and Michael Spinner and has published in prestigious journals such as Science, Nature Communications and Neuron.

In The Last Decade

Thomas J. Esparza

33 papers receiving 1.9k citations

Peers

Countries citing papers authored by Thomas J. Esparza

Since Specialization

Citations

This map shows the geographic impact of Thomas J. Esparza's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas J. Esparza with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas J. Esparza more than expected).

Fields of papers citing papers by Thomas J. Esparza

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas J. Esparza. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas J. Esparza. The network helps show where Thomas J. Esparza may publish in the future.

Co-authorship network of co-authors of Thomas J. Esparza

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas J. Esparza. A scholar is included among the top collaborators of Thomas J. Esparza based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas J. Esparza. Thomas J. Esparza is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Monitoring T-Cell Activation in the Tumor Microenvironment by PET Imaging of the Chemokine CXCL9 2025 ·Journal of Nuclear Medicine ·Orit Jacobson, Hongwei H. Zhang, (unknown), Behnaz Ghaemi, Falguni Basuli, (unknown), (unknown), (unknown), Sundar Ganesan, Thomas J. Esparza, Freddy E. Escorcia, Peter L. Choyke, Joshua Μ. Farber	0
2	Real-time environmental surveillance of SARS-CoV-2 aerosols 2023 ·Nature Communications ·Joseph V. Puthussery, (unknown), (unknown), (unknown), Benjamin J. Sumlin, (unknown), (unknown), Nishit Shetty, (unknown), (unknown), David L. Brody, Thomas J. Esparza, Traci L. Bricker, Adrianus C. M. Boon, Carla M. Yuede, John R. Cirrito, Rajan K. Chakrabarty	30
3	Rapid Direct Detection of SARS-CoV-2 Aerosols in Exhaled Breath at the Point of Care 2023 ·ACS Sensors ·(unknown), Nishit Shetty, (unknown), Joseph V. Puthussery, Benjamin J. Sumlin, (unknown), (unknown), (unknown), David L. Brody, Thomas J. Esparza, Jane A. O’Halloran, Rachel M. Presti, Traci L. Bricker, Adrianus C. M. Boon, Carla M. Yuede, John R. Cirrito, Rajan K. Chakrabarty	15
4	Enhanced in vivo blood brain barrier transcytosis of macromolecular cargo using an engineered pH-sensitive mouse transferrin receptor binding nanobody 2023 ·Fluids and Barriers of the CNS ·Thomas J. Esparza, (unknown), (unknown), (unknown), Joong Hee Kim, David L. Brody	24
5	Selection of single domain anti-transferrin receptor antibodies for blood-brain barrier transcytosis using a neurotensin based assay and histological assessment of target engagement in a mouse model of Alzheimer’s related amyloid-beta pathology 2022 ·PLoS ONE ·(unknown), Thomas J. Esparza, David L. Brody	9
6	Sensitive detection of extremely small iron oxide nanoparticles in living mice using MP2RAGE with advanced image co-registration 2021 ·Scientific Reports ·Joong H. Kim, Stephen Dodd, Frank Q. Ye, Andrew K. Knutsen, Duong Thanh Nguyen, Haitao Wu, (unknown), Simone Mastrogiacomo, Thomas J. Esparza, Rolf E. Swenson, David L. Brody	4
7	Unbiased high-content screening reveals Aβ- and tau-independent synaptotoxic activities in human brain homogenates from Alzheimer’s patients and high-pathology controls 2021 ·PLoS ONE ·Hao Jiang, Thomas J. Esparza, Terrance T. Kummer, David L. Brody	3
8	Live Neuron High-Content Screening Reveals Synaptotoxic Activity in Alzheimer Mouse Model Homogenates 2020 ·Scientific Reports ·Hao Jiang, Thomas J. Esparza, Terrance T. Kummer, Haining Zhong, Jens Rettig, David L. Brody	8
9	High affinity nanobodies block SARS-CoV-2 spike receptor binding domain interaction with human angiotensin converting enzyme 2020 ·Scientific Reports ·Thomas J. Esparza, Negin P. Martin, George P. Anderson, Ellen R. Goldman, David L. Brody	85
10	Repetitive Concussive and Subconcussive Injury in a Human Tau Mouse Model Results in Chronic Cognitive Dysfunction and Disruption of White Matter Tracts, But Not Tau Pathology 2018 ·Journal of Neurotrauma ·Mihika Gangolli, (unknown), Thomas J. Esparza, (unknown), (unknown), David L. Brody	18
11	Diversity of Amyloid-beta Proteoforms in the Alzheimer’s Disease Brain 2017 ·Scientific Reports ·Norelle C. Wildburger, Thomas J. Esparza, Richard D. LeDuc, Ryan T. Fellers, Paul M. Thomas, Nigel J. Cairns, Neil L. Kelleher, Randall J. Bateman, David L. Brody	118
12	Age-Dependent Effects of apoE Reduction Using Antisense Oligonucleotides in a Model of β-amyloidosis 2017 ·Neuron ·Tien‐Phat V. Huynh, Fan Liao, Caroline Massad Francis, Grace O. Robinson, Javier Remolina Serrano, Hong Jiang, Joseph Roh, Mary Beth Finn, Patrick M. Sullivan, Thomas J. Esparza, Floy R. Stewart, Thomas E. Mahan, Jason D. Ulrich, Tracy Cole, David M. Holtzman	144
13	Non-canonical soluble amyloid-beta aggregates and plaque buffering: controversies and future directions for target discovery in Alzheimer’s disease 2017 ·Alzheimer s Research & Therapy ·David L. Brody, Hao Jiang, Norelle C. Wildburger, Thomas J. Esparza	58
14	Soluble Amyloid-beta Aggregates from Human Alzheimer’s Disease Brains 2016 ·Scientific Reports ·Thomas J. Esparza, Norelle C. Wildburger, Hao Jiang, Mihika Gangolli, Nigel J. Cairns, Randall J. Bateman, David L. Brody	98
15	Quantitative assessments of traumatic axonal injury in human brain: concordance of microdialysis and advanced MRI 2015 ·Brain ·Sandra Magnoni, Christine L. Mac Donald, Thomas J. Esparza, V. Conte, (unknown), (unknown), Giulio Bertani, (unknown), Antonella Costa, (unknown), Abraham Z. Snyder, Joshua S. Shimony, Fabio Triulzi, Nino Stocchetti, David L. Brody	38
16	Human Apolipoprotein E4 Worsens Acute Axonal Pathology but Not Amyloid-β Immunoreactivity After Traumatic Brain Injury in 3×TG-AD Mice 2013 ·Journal of Neuropathology & Experimental Neurology ·Rachel E. Bennett, Thomas J. Esparza, H.A. Lewis, (unknown), Christine L. Mac Donald, Patrick M. Sullivan, David L. Brody	33
17	Distinct Temporal and Anatomical Distributions of Amyloid-β and Tau Abnormalities following Controlled Cortical Impact in Transgenic Mice 2011 ·PLoS ONE ·Hien Tran, Laura Sánchez, Thomas J. Esparza, David L. Brody	71
18	Tau elevations in the brain extracellular space correlate with reduced amyloid-β levels and predict adverse clinical outcomes after severe traumatic brain injury 2011 ·Brain ·Sandra Magnoni, Thomas J. Esparza, V. Conte, Marco Carbonara, Giorgio Carrabba, David M. Holtzman, G.J. Zipfel, Nino Stocchetti, David L. Brody	135
19	Traumatic brain injury reduces soluble extracellular amyloid-β in mice: A methodologically novel combined microdialysis-controlled cortical impact study 2010 ·Neurobiology of Disease ·Katherine E. Schwetye, John R. Cirrito, Thomas J. Esparza, Christine L. Mac Donald, David M. Holtzman, David L. Brody	39
20	Activation of Type B T Cells after Protein Immunization Reveals Novel Pathways of In Vivo Presentation of Peptides 2007 ·The Journal of Immunology ·Scott B. Lovitch, Thomas J. Esparza, George G. Schweitzer, Jeremy Herzog, Emil R. Unanue	20

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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