Taro Ishiguro

1.6k total citations
33 papers, 967 citations indexed

About

Taro Ishiguro is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Taro Ishiguro has authored 33 papers receiving a total of 967 indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 14 papers in Cellular and Molecular Neuroscience and 8 papers in Neurology. Recurrent topics in Taro Ishiguro's work include Genetic Neurodegenerative Diseases (12 papers), Mitochondrial Function and Pathology (5 papers) and Ion channel regulation and function (5 papers). Taro Ishiguro is often cited by papers focused on Genetic Neurodegenerative Diseases (12 papers), Mitochondrial Function and Pathology (5 papers) and Ion channel regulation and function (5 papers). Taro Ishiguro collaborates with scholars based in Japan, United States and Australia. Taro Ishiguro's co-authors include Chikahiko Eguchi, Kinya Ishikawa, Kaoru Yamada, T Sugimoto, Hidehiro Mizusawa, Atsuo Goto, Masao Ishii, Masanori Yoshioka, Tetsuichiro Muto and T. Tsuruo and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Neuron and PLoS ONE.

In The Last Decade

Taro Ishiguro

32 papers receiving 955 citations

Peers

Taro Ishiguro
Jyoti Disa United States
Isamu Aiba United States
Chen-Hsiung Yeh United States
Anne Vaslin Switzerland
Taro Ishiguro
Citations per year, relative to Taro Ishiguro Taro Ishiguro (= 1×) peers Isabelle Limon

Countries citing papers authored by Taro Ishiguro

Since Specialization
Citations

This map shows the geographic impact of Taro Ishiguro's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Taro Ishiguro with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Taro Ishiguro more than expected).

Fields of papers citing papers by Taro Ishiguro

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Taro Ishiguro. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Taro Ishiguro. The network helps show where Taro Ishiguro may publish in the future.

Co-authorship network of co-authors of Taro Ishiguro

This figure shows the co-authorship network connecting the top 25 collaborators of Taro Ishiguro. A scholar is included among the top collaborators of Taro Ishiguro based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Taro Ishiguro. Taro Ishiguro is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ishiguro, Taro, et al.. (2025). Predictive Genetic Testing and Genetic Counseling for Hereditary Neuromuscular Diseases in Japan: A Case Series of 40 Clients. Neurology and Clinical Neuroscience. 13(5). 360–365. 1 indexed citations
2.
Ishiguro, Taro, Yoshitaka Nagai, & Kinya Ishikawa. (2021). Insight Into Spinocerebellar Ataxia Type 31 (SCA31) From Drosophila Model. Frontiers in Neuroscience. 15. 648133–648133. 9 indexed citations
3.
Ishiguro, Taro, et al.. (2020). Dupilumab for Atopic Dermatitis, a Possible Risk Factor of Juvenile Ischemic Stroke: A Case Report. Journal of Stroke and Cerebrovascular Diseases. 29(6). 104763–104763. 5 indexed citations
4.
Ishiguro, Taro, Nozomu Sato, Morio Ueyama, et al.. (2017). Regulatory Role of RNA Chaperone TDP-43 for RNA Misfolding and Repeat-Associated Translation in SCA31. Neuron. 94(1). 108–124.e7. 91 indexed citations
5.
Takahashi, Makoto, Taro Ishiguro, Nozomu Sato, et al.. (2013). Cytoplasmic Location of α1A Voltage-Gated Calcium Channel C-Terminal Fragment (Cav2.1-CTF) Aggregate Is Sufficient to Cause Cell Death. PLoS ONE. 8(3). e50121–e50121. 11 indexed citations
6.
Toru, Shuta, et al.. (2012). Paradoxical cerebral embolism with patent foramen ovale and deep venous thrombosis caused by a massive myoma uteri. Clinical Neurology and Neurosurgery. 115(6). 760–761. 5 indexed citations
7.
Takahashi, Makoto, Kinya Ishikawa, Nozomu Sato, et al.. (2012). Reduced brain‐derived neurotrophic factor (BDNF) mRNA expression and presence of BDNF‐immunoreactive granules in the spinocerebellar ataxia type 6 (SCA6) cerebellum. Neuropathology. 32(6). 595–603. 26 indexed citations
8.
Ishiguro, Taro, et al.. (2010). Gene trapping identifies chloride channel 4 as a novel inducer of colon cancer cell migration, invasion and metastases. British Journal of Cancer. 102(4). 774–782. 18 indexed citations
9.
Ishiguro, Taro, Kinya Ishikawa, Makoto Takahashi, et al.. (2009). The carboxy-terminal fragment of α1A calcium channel preferentially aggregates in the cytoplasm of human spinocerebellar ataxia type 6 Purkinje cells. Acta Neuropathologica. 119(4). 447–464. 45 indexed citations
10.
Watase, Kei, Curtis F. Barrett, Taisuke Miyazaki, et al.. (2008). Spinocerebellar ataxia type 6 knockin mice develop a progressive neuronal dysfunction with age-dependent accumulation of mutant Ca V 2.1 channels. Proceedings of the National Academy of Sciences. 105(33). 11987–11992. 122 indexed citations
11.
Ishikawa, Kinya, M Sakamoto, Taiji Tsunemi, et al.. (2008). Direct and accurate measurement of CAG repeat configuration in the ataxin-1 (ATXN-1) gene by “dual-fluorescence labeled PCR-restriction fragment length analysis”. Journal of Human Genetics. 53(4). 287–295. 4 indexed citations
12.
Ishikawa, Kinya, et al.. (2008). Analyses of copy number and mRNA expression level of the alpha-synuclein gene in multiple system atrophy.. PubMed. 55(1). 145–53. 40 indexed citations
13.
Amino, Takeshi, Kinya Ishikawa, Shuta Toru, et al.. (2007). Redefining the disease locus of 16q22.1-linked autosomal dominant cerebellar ataxia. Journal of Human Genetics. 52(8). 643–649. 20 indexed citations
14.
Yamamoto, Masayoshi, Taro Ishiguro, Kazue Tazaki, Kazuhisa Komura, & Kaoru UENO. (1996). 237Np in Hemp-Palm Leaves of Bontenchiku for Fishing Gear Used by the Fifth Fukuryu-Maru. Health Physics. 70(5). 744–748. 21 indexed citations
15.
Tomioka, Kiyoshi, Taro Ishiguro, Hiroshi Mizuguchi, et al.. (1991). Stereoselective reactions. XVII. Absolute structure-cytotoxic activity relationships of steganacin congeners and analogs. Journal of Medicinal Chemistry. 34(1). 54–57. 38 indexed citations
16.
Tanaka, Hirofumi, Taro Ishiguro, Chikahiko Eguchi, Kayoko Saito, & E Ozawa. (1991). Expression of a dystrophin-related protein associated with the skeletal muscle cell membrane. Histochemistry and Cell Biology. 96(1). 1–5. 71 indexed citations
17.
Goto, Atsuo, Taro Ishiguro, Kaoru Yamada, et al.. (1990). Isolation of a urinary digitalis-like factor indistinguishable from digoxin. Biochemical and Biophysical Research Communications. 173(3). 1093–1101. 123 indexed citations
18.
Goto, Atsuo, Kaoru Yamada, Masao Ishii, et al.. (1989). Existence of a polar digitalis-like factor in mammalian hypothalamus. Biochemical and Biophysical Research Communications. 161(3). 953–958. 15 indexed citations
19.
Goto, Atsuo, Kaoru Yamada, Masao Ishii, et al.. (1988). Presence of digitalis-like factor in mammalian plasma. Biochemical and Biophysical Research Communications. 152(1). 322–327. 30 indexed citations
20.
Goto, Atsuo, Kaoru Yamada, Masao Ishii, et al.. (1988). Purification and characterization of human urine-derived digitalis-like factor. Biochemical and Biophysical Research Communications. 154(3). 847–853. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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