Taro Ishiguro
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 5%
- Neurology top 10%
- Cardiology and Cardiovascular Medicine
- Pulmonary and Respiratory Medicine
- Co-authors
- Chikahiko EguchiKinya IshikawaKaoru YamadaT SugimotoHidehiro MizusawaAtsuo GotoMasao IshiiMasanori Yoshioka
- Topics
- Genetic Neurodegenerative Diseases (12 papers)Mitochondrial Function and Pathology (5 papers)Ion channel regulation and function (5 papers)
- Partner nations
- JapanUnited StatesAustralia
In The Last Decade
Taro Ishiguro
32 papers receiving 955 citations
Peers
Comparison fields: 5 of 84
- Molecular Biology 704
- Cellular and Molecular Neuroscience 370
- Neurology 135
- Cardiology and Cardiovascular Medicine 95
- Pulmonary and Respiratory Medicine 87
Countries citing papers authored by Taro Ishiguro
This map shows the geographic impact of Taro Ishiguro's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Taro Ishiguro with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Taro Ishiguro more than expected).
Fields of papers citing papers by Taro Ishiguro
This network shows the impact of papers produced by Taro Ishiguro. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Taro Ishiguro. The network helps show where Taro Ishiguro may publish in the future.
Co-authorship network of co-authors of Taro Ishiguro
This figure shows the co-authorship network connecting the top 25 collaborators of Taro Ishiguro. A scholar is included among the top collaborators of Taro Ishiguro based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Taro Ishiguro. Taro Ishiguro is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 9 | |
| 3 | 5 | |
| 4 | 91 | |
| 5 | 11 | |
| 6 | 5 | |
| 7 | 26 | |
| 8 | 18 | |
| 9 | 45 | |
| 10 | 122 | |
| 11 | 4 | |
| 12 | Analyses of copy number and mRNA expression level of the alpha-synuclein gene in multiple system atrophy. | 40 |
| 13 | 20 | |
| 14 | 21 | |
| 15 | 38 | |
| 16 | 71 | |
| 17 | 123 | |
| 18 | 15 | |
| 19 | 30 | |
| 20 | 30 |
About Taro Ishiguro
Taro Ishiguro is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology, having authored 33 papers that have together received 967 indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (12 papers), Mitochondrial Function and Pathology (5 papers) and Ion channel regulation and function (5 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (370 citations), Molecular Biology (704 citations) and Neurology (135 citations). Taro Ishiguro has collaborated with scholars based in Japan, United States and Australia. Frequent co-authors include Chikahiko Eguchi, Kinya Ishikawa, Kaoru Yamada, T Sugimoto, Hidehiro Mizusawa, Atsuo Goto, Masao Ishii, Masanori Yoshioka, Tetsuichiro Muto and T. Tsuruo. Their work appears in journals such as Proceedings of the National Academy of Sciences, Neuron and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.