Tadahilo Oshida

562 total citations
7 papers, 477 citations indexed

About

Tadahilo Oshida is a scholar working on Immunology, Dermatology and Molecular Biology. According to data from OpenAlex, Tadahilo Oshida has authored 7 papers receiving a total of 477 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 4 papers in Dermatology and 1 paper in Molecular Biology. Recurrent topics in Tadahilo Oshida's work include Immune Cell Function and Interaction (5 papers), Dermatology and Skin Diseases (4 papers) and T-cell and B-cell Immunology (3 papers). Tadahilo Oshida is often cited by papers focused on Immune Cell Function and Interaction (5 papers), Dermatology and Skin Diseases (4 papers) and T-cell and B-cell Immunology (3 papers). Tadahilo Oshida collaborates with scholars based in Japan, United States and United Kingdom. Tadahilo Oshida's co-authors include Hirohisa Saito, Yuji Sugita, Hiromasa Inoue, Nicholas A. Cacalano, Shinjiro Hamano, Satoru Fukuyama, Katsuhiko Hayashi, Koichiro Matsumoto, Anne O’Garra and Kyoko Inagaki‐Ohara and has published in prestigious journals such as Nature Medicine, Biochemical and Biophysical Research Communications and FEBS Letters.

In The Last Decade

Tadahilo Oshida

7 papers receiving 467 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tadahilo Oshida Japan 6 280 198 124 101 57 7 477
Rasmus Boye Kjellerup Denmark 10 465 1.7× 143 0.7× 134 1.1× 106 1.0× 222 3.9× 13 604
Wanhu Tang United States 12 356 1.3× 81 0.4× 134 1.1× 57 0.6× 85 1.5× 21 541
Julia Lopatnikova Russia 12 237 0.8× 108 0.5× 137 1.1× 30 0.3× 22 0.4× 57 419
G Tonel Switzerland 7 583 2.1× 92 0.5× 87 0.7× 122 1.2× 287 5.0× 9 654
Gaelle Poyet France 5 489 1.7× 180 0.9× 61 0.5× 50 0.5× 232 4.1× 6 700
Michael J. Pattison United Kingdom 10 259 0.9× 163 0.8× 189 1.5× 30 0.3× 9 0.2× 12 563
Julia Tseng United States 7 230 0.8× 149 0.8× 230 1.9× 74 0.7× 13 0.2× 7 506
Patricia L. Orchansky Canada 12 314 1.1× 121 0.6× 108 0.9× 35 0.3× 8 0.1× 14 425
Y Basaki Japan 8 156 0.6× 102 0.5× 169 1.4× 155 1.5× 39 0.7× 12 512
Markward Ständer Germany 9 345 1.2× 81 0.4× 49 0.4× 116 1.1× 157 2.8× 9 425

Countries citing papers authored by Tadahilo Oshida

Since Specialization
Citations

This map shows the geographic impact of Tadahilo Oshida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tadahilo Oshida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tadahilo Oshida more than expected).

Fields of papers citing papers by Tadahilo Oshida

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tadahilo Oshida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tadahilo Oshida. The network helps show where Tadahilo Oshida may publish in the future.

Co-authorship network of co-authors of Tadahilo Oshida

This figure shows the co-authorship network connecting the top 25 collaborators of Tadahilo Oshida. A scholar is included among the top collaborators of Tadahilo Oshida based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tadahilo Oshida. Tadahilo Oshida is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Yoshida, Ning Lu, Yuji Sugita, Tohru Arai, et al.. (2005). Mite-antigen Stimulates MAL Expression in Peripheral Blood T Cells of Mite-sensitive Subjects. Allergology International. 54(2). 273–282. 1 indexed citations
2.
Matsumoto, Yoshiko, Yukiho Imai, Ning Lu Yoshida, et al.. (2004). Upregulation of the transcript level of GTPase activating protein KIAA0603 in T cells from patients with atopic dermatitis. FEBS Letters. 572(1-3). 135–140. 19 indexed citations
3.
Oshida, Tadahilo, Ning Lu Yoshida, Noriko Yuyama, et al.. (2003). Analysis of Highly Expressed Genes in Monocytes from Atopic Dermatitis Patients. International Archives of Allergy and Immunology. 132(2). 156–167. 17 indexed citations
4.
Inoue, Hiromasa, Katsuhiko Hayashi, Satoru Fukuyama, et al.. (2003). SOCS-3 regulates onset and maintenance of TH2-mediated allergic responses. Nature Medicine. 9(8). 1047–1054. 300 indexed citations
5.
Imai, Yukiho, Ryoichi Hashida, Yuji Sugita, et al.. (2002). Cloning and characterization of the highly expressed ETEA gene from blood cells of atopic dermatitis patients. Biochemical and Biophysical Research Communications. 297(5). 1282–1290. 19 indexed citations
6.
Yoshida, Ning Lu, Toshiyuki Miyashita, U Mami, et al.. (2002). Analysis of gene expression patterns during glucocorticoid-induced apoptosis using oligonucleotide arrays. Biochemical and Biophysical Research Communications. 293(4). 1254–1261. 65 indexed citations
7.
Matsumoto, Yoshiko, Tadahilo Oshida, Yukiho Imai, et al.. (2002). Identification of Highly Expressed Genes in Peripheral Blood T Cells from Patients with Atopic Dermatitis. International Archives of Allergy and Immunology. 129(4). 327–340. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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