T. Shirotani

683 total citations
14 papers, 488 citations indexed

About

T. Shirotani is a scholar working on Molecular Biology, Surgery and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, T. Shirotani has authored 14 papers receiving a total of 488 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 10 papers in Surgery and 4 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in T. Shirotani's work include Pancreatic function and diabetes (10 papers), Metabolism, Diabetes, and Cancer (8 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). T. Shirotani is often cited by papers focused on Pancreatic function and diabetes (10 papers), Metabolism, Diabetes, and Cancer (8 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). T. Shirotani collaborates with scholars based in Japan and United States. T. Shirotani's co-authors include Eiichi Araki, Hiroki Kishikawa, Kazuaki Yoshizato, Masayoshi Shichiri, Hiroyuki Motoshima, Nobuhiro Miyamura, Seiji Ura, Tetsuya Taguchi, M. Uehara and Kaku Tsuruzoe and has published in prestigious journals such as Diabetes, Biochemical and Biophysical Research Communications and Diabetologia.

In The Last Decade

T. Shirotani

14 papers receiving 475 citations

Peers

T. Shirotani
T. Shirotani
Citations per year, relative to T. Shirotani T. Shirotani (= 1×) peers Kazuaki Yoshizato

Countries citing papers authored by T. Shirotani

Since Specialization
Citations

This map shows the geographic impact of T. Shirotani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by T. Shirotani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites T. Shirotani more than expected).

Fields of papers citing papers by T. Shirotani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by T. Shirotani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by T. Shirotani. The network helps show where T. Shirotani may publish in the future.

Co-authorship network of co-authors of T. Shirotani

This figure shows the co-authorship network connecting the top 25 collaborators of T. Shirotani. A scholar is included among the top collaborators of T. Shirotani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with T. Shirotani. T. Shirotani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Toyonaga, Tomomi, Kaku Tsuruzoe, T. Shirotani, et al.. (2002). Heterozygous knockout of the IRS-1 gene in mice enhances obesity-linked insulin resistance: a possible model for the development of type 2 diabetes. Journal of Endocrinology. 174(2). 309–319. 44 indexed citations
2.
Furukawa, Noboru, T. Shirotani, Kazuhiko Nakamaru, et al.. (2002). Regulation of the Insulin Gene Transcription by Glucose.. Endocrine Journal. 49(2). 121–130. 4 indexed citations
3.
Yoshizato, Kazuaki, T. Shirotani, Noboru Furukawa, et al.. (2001). Identification of a cis-Acting Element and a Novel trans-Acting Factor of the Human Insulin Receptor Gene in HepG2 and Rat Liver Cells. Biochemical and Biophysical Research Communications. 280(2). 428–434. 15 indexed citations
4.
Taguchi, Tetsuya, Hideki Kishikawa, Hiroyuki Motoshima, et al.. (2000). Involvement of bradykinin in acute exercise-induced increase of glucose uptake and GLUT-4 translocation in skeletal muscle: Studies in normal and diabetic humans and rats. Metabolism. 49(7). 920–930. 51 indexed citations
5.
Motoshima, Hiroyuki, Eiichi Araki, Tetsuya Taguchi, et al.. (2000). Bradykinin enhances insulin receptor tyrosine kinase in 32D cells reconstituted with bradykinin and insulin signaling pathways. Diabetes Research and Clinical Practice. 48(3). 155–170. 17 indexed citations
6.
Shirotani, T., Eiichi Araki, K. Matsumoto, et al.. (1999). Insulin inhibits glucagon secretion by the activation of PI3-kinase in In-R1-G9 cells. Diabetes Research and Clinical Practice. 44(2). 83–92. 47 indexed citations
9.
Tsuruzoe, Kaku, Eiichi Araki, T. Shirotani, et al.. (1998). Creation and characterization of a mitochondrial DNA-depleted pancreatic beta-cell line: impaired insulin secretion induced by glucose, leucine, and sulfonylureas.. Diabetes. 47(4). 621–631. 63 indexed citations
11.
Ura, Seiji, Eiichi Araki, Hiroki Kishikawa, et al.. (1996). Molecular scanning of the insulin receptor substrate-1 (IRS-1) gene in Japanese patients with NIDDM: identification of five novel polymorphisms. Diabetologia. 39(5). 600–608. 46 indexed citations
12.
Kisanuki, Koichi, Hiroki Kishikawa, Eiichi Araki, et al.. (1995). Expression of insulin receptor on clonal pancreatic alpha cells and its possible role for insulin-stimulated negative regulation of glucagon secretion. Diabetologia. 38(4). 422–429. 42 indexed citations
13.
Shirotani, T., Hideki Kishikawa, Nakayasu Wake, et al.. (1992). Thyroxine-Binding Globulin Variant(TBG-Kumamoto): Identification of a Point Mutation and Genotype Analysis of Its Family.. Endocrinologia Japonica. 39(6). 577–584. 5 indexed citations
14.
Hamaguchi, Tomoya, Hideo Fukushima, M. Uehara, et al.. (1991). Abnormal glucagon response to arginine and its normalization in obese hyperinsulinaemic patients with glucose intolerance: importance of insulin action on pancreatic Alpha cells. Diabetologia. 34(11). 801–806. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026