E. Seffer

675 total citations
16 papers, 575 citations indexed

About

E. Seffer is a scholar working on Molecular Biology, Surgery and Physiology. According to data from OpenAlex, E. Seffer has authored 16 papers receiving a total of 575 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 11 papers in Surgery and 3 papers in Physiology. Recurrent topics in E. Seffer's work include Metabolism, Diabetes, and Cancer (11 papers), Pancreatic function and diabetes (11 papers) and Protein Kinase Regulation and GTPase Signaling (10 papers). E. Seffer is often cited by papers focused on Metabolism, Diabetes, and Cancer (11 papers), Pancreatic function and diabetes (11 papers) and Protein Kinase Regulation and GTPase Signaling (10 papers). E. Seffer collaborates with scholars based in Germany, Italy and United Kingdom. E. Seffer's co-authors include Joanne Mushack, Monika Kellerer, Hans-Ulrich Häring, Hans‐Ulrich Häring, B. Obermaier–Kusser, Luitgard Mosthaf, B Ermel, Fausto Machicao, A. Ullrich and Birgit Bossenmaier and has published in prestigious journals such as Journal of Biological Chemistry, Biochemical Journal and Biochemical and Biophysical Research Communications.

In The Last Decade

E. Seffer

16 papers receiving 551 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
E. Seffer Germany 12 400 195 159 147 63 16 575
Vildan N. Civelek United States 9 267 0.7× 216 1.1× 109 0.7× 150 1.0× 38 0.6× 9 454
Murthy S.R. Madiraju Canada 3 309 0.8× 219 1.1× 169 1.1× 157 1.1× 42 0.7× 5 533
D. Kirsch Germany 11 360 0.9× 132 0.7× 106 0.7× 145 1.0× 54 0.9× 11 461
Marina Roques France 9 357 0.9× 94 0.5× 80 0.5× 241 1.6× 63 1.0× 10 518
Hassan Jijakli Belgium 11 198 0.5× 219 1.1× 189 1.2× 85 0.6× 53 0.8× 42 482
Joanne Mushack Germany 15 537 1.3× 222 1.1× 117 0.7× 293 2.0× 101 1.6× 23 868
Ann-Marie Richard United States 10 254 0.6× 169 0.9× 122 0.8× 246 1.7× 63 1.0× 11 537
Carol Lenaghan United Kingdom 7 249 0.6× 162 0.8× 220 1.4× 222 1.5× 41 0.7× 8 554
Ingo Uphues Germany 10 404 1.0× 206 1.1× 174 1.1× 170 1.2× 65 1.0× 17 617
Scott C. McCoid United States 9 440 1.1× 210 1.1× 93 0.6× 206 1.4× 63 1.0× 9 584

Countries citing papers authored by E. Seffer

Since Specialization
Citations

This map shows the geographic impact of E. Seffer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. Seffer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. Seffer more than expected).

Fields of papers citing papers by E. Seffer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. Seffer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. Seffer. The network helps show where E. Seffer may publish in the future.

Co-authorship network of co-authors of E. Seffer

This figure shows the co-authorship network connecting the top 25 collaborators of E. Seffer. A scholar is included among the top collaborators of E. Seffer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E. Seffer. E. Seffer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Mosthaf, Luitgard, et al.. (2009). Insulin leads to a parallel translocation of PI-3-kinase and protein kinase C ζ. Experimental and Clinical Endocrinology & Diabetes. 104(1). 19–24. 4 indexed citations
3.
Kellerer, Monika, et al.. (1998). The Long Acting Human Insulin Analog HOE 901: Characteristics of Insulin Signalling in Comparison to Asp(B10) and Regular Insulin. Hormone and Metabolic Research. 30(3). 123–129. 81 indexed citations
4.
Mosthaf, Luitgard, Lucia Berti, Monika Kellerer, et al.. (1995). C-Terminus or Juxtamembrane Deletions in the Insulin Receptor do not Affect the Glucose-Dependent Inhibition of the Tyrosine Kinase Activity. European Journal of Biochemistry. 227(3). 787–791. 5 indexed citations
5.
Mosthaf, Luitgard, Lucia Berti, Monika Kellerer, et al.. (1995). C‐Terminus or Juxtamembrane Deletions in the Insulin Receptor do not Affect the Glucose‐Dependent Inhibition of the Tyrosine Kinase Activity. European Journal of Biochemistry. 227(3). 787–791. 6 indexed citations
6.
Berti, Lucia, Luitgard Mosthaf, Monika Kellerer, et al.. (1994). Glucose-induced translocation of protein kinase C isoforms in rat-1 fibroblasts is paralleled by inhibition of the insulin receptor tyrosine kinase.. Journal of Biological Chemistry. 269(5). 3381–3386. 104 indexed citations
7.
Kellerer, Monika, Giorgio Sesti, E. Seffer, et al.. (1993). Altered pattern of insulin receptor isotypes in skeletal muscle membranes of Type 2 (non-insulin-dependent) diabetic subjects. Diabetologia. 36(7). 628–632. 54 indexed citations
8.
Kellerer, Monika, Fausto Machicao, Lucia Berti, et al.. (1993). Inositol phospho-oligosaccharides from rat fibroblasts and adipocytes stimulate 3-O-methylglucose transport. Biochemical Journal. 295(3). 699–704. 10 indexed citations
9.
Vogt, B., B. Obermaier–Kusser, E. Seffer, et al.. (1992). Subcellular distribution of GLUT 4 in the skeletal muscle of lean type 2 (non-insulin-dependent) diabetic patients in the basal state. Diabetologia. 35(5). 456–463. 32 indexed citations
10.
Kellerer, Monika, Fausto Machicao, E. Seffer, et al.. (1991). Stimulation of phospholipase C activity by insulin is mediated by both isotypes of the human insulin receptor. Biochemical and Biophysical Research Communications. 181(2). 566–572. 13 indexed citations
11.
Vogt, B., Joanne Mushack, E. Seffer, & Hans‐Ulrich Häring. (1991). The translocation of the glucose transporter sub-types GLUT1 and GLUT4 in isolated fat cells is differently regulated by phorbol esters. Biochemical Journal. 275(3). 597–600. 34 indexed citations
12.
Kellerer, Monika, B. Obermaier–Kusser, E. Schleicher, et al.. (1991). Insulin activates GTP binding to a 40 kDa protein in fat cells. Biochemical Journal. 276(1). 103–108. 27 indexed citations
13.
Vogt, Beate, Joanne Mushack, E. Seffer, & Hans-Ulrich Häring. (1990). The phorbol ester TPA induces a translocation of the insulin sensitive glucose carrier (GLUT4) in fat cells. Biochemical and Biophysical Research Communications. 168(3). 1089–1094. 11 indexed citations
14.
Kellerer, Monika, E. Seffer, Joanne Mushack, B. Obermaier–Kusser, & Hans‐Ulrich Häring. (1990). TPA inhibits insulin stimulated PIP hydrolysis in fat cell membranes: Evidence for modulation of insulin dependent phospholipase C by proteinkinase C. Biochemical and Biophysical Research Communications. 172(2). 446–454. 15 indexed citations
15.
Machicao, Fausto, Joanne Mushack, E. Seffer, B Ermel, & Hans-Ulrich Häring. (1990). Mannose, glucosamine and inositol monophosphate inhibit the effects of insulin on lipogenesis. Further evidence for a role for inositol phosphate-oligosaccharides in insulin action.. PubMed. 266(3). 909–16. 48 indexed citations
16.
Obermaier–Kusser, B., Joanne Mushack, E. Seffer, et al.. (1989). Further evidence for a two-step model of glucose-transport regulation. Inositol phosphate-oligosaccharides regulate glucose-carrier activity. Biochemical Journal. 261(3). 699–705. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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