Sugako Oka

1.9k total citations
26 papers, 1.6k citations indexed

About

Sugako Oka is a scholar working on Molecular Biology, Oncology and Physiology. According to data from OpenAlex, Sugako Oka has authored 26 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 4 papers in Oncology and 4 papers in Physiology. Recurrent topics in Sugako Oka's work include DNA Repair Mechanisms (14 papers), Mitochondrial Function and Pathology (10 papers) and Alzheimer's disease research and treatments (4 papers). Sugako Oka is often cited by papers focused on DNA Repair Mechanisms (14 papers), Mitochondrial Function and Pathology (10 papers) and Alzheimer's disease research and treatments (4 papers). Sugako Oka collaborates with scholars based in Japan, United States and Greece. Sugako Oka's co-authors include Yusaku Nakabeppu, Kunihiko Sakumi, Daisuke Tsuchimoto, Julio Leon, Mizuki Ohno, Zijing Sheng, Nona Abolhassani, Masato Furuichi, Toru Iwaki and Frank M. LaFerla and has published in prestigious journals such as Journal of Clinical Investigation, Nature Communications and Journal of Neuroscience.

In The Last Decade

Sugako Oka

25 papers receiving 1.5k citations

Peers

Sugako Oka

PHYSI

ONCOL

CMN

Irina N. Gaisina United States

Yejun Tan United States

Yibing Li China

Scott Maynard United States

Liang Tao China

Lori A. Sturtz United States

Zhiyong Gao United States

Timothy Coskran United States

Yukiko Kaneko Japan

Irina N. Gaisina United States

Sugako Oka

931 ×0.9

270 ×0.8

PHYSI

141 ×0.7

135 ×0.9

ONCOL

211 ×1.5

CMN

Citations per year, relative to Sugako Oka Sugako Oka (= 1×) peers Irina N. Gaisina

Countries citing papers authored by Sugako Oka

Since Specialization

Citations

This map shows the geographic impact of Sugako Oka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sugako Oka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sugako Oka more than expected).

Fields of papers citing papers by Sugako Oka

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sugako Oka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sugako Oka. The network helps show where Sugako Oka may publish in the future.

Co-authorship network of co-authors of Sugako Oka

This figure shows the co-authorship network connecting the top 25 collaborators of Sugako Oka. A scholar is included among the top collaborators of Sugako Oka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sugako Oka. Sugako Oka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ranjan, Atul, Alejandro Parrales, Shigeto Nishikawa, et al.. (2025). Suppression of stress granule formation is a vulnerability imposed by mutant p53. Nature Communications. 16(1). 2365–2365. 1 indexed citations

Oka, Sugako, et al.. (2022). Endogenous ROS production in early differentiation state suppresses endoderm differentiation via transient FOXC1 expression. Cell Death Discovery. 8(1). 150–150. 12 indexed citations

Oka, Sugako, Julio Leon, Kunihiko Sakumi, et al.. (2021). MTH1 and OGG1 maintain a low level of 8-oxoguanine in Alzheimer's brain, and prevent the progression of Alzheimer's pathogenesis. Scientific Reports. 11(1). 5819–5819. 33 indexed citations

Ulges, Alexander, Tobias Bopp, Andrea Schäfer, et al.. (2017). Role of the DNA repair glycosylase OGG1 in the activation of murine splenocytes. DNA repair. 58. 13–20. 7 indexed citations

Abolhassani, Nona, Julio Leon, Zijing Sheng, et al.. (2016). Molecular pathophysiology of impaired glucose metabolism, mitochondrial dysfunction, and oxidative DNA damage in Alzheimer's disease brain. Mechanisms of Ageing and Development. 161(Pt A). 95–104. 107 indexed citations

Oka, Sugako, Julio Leon, Kunihiko Sakumi, et al.. (2016). Human mitochondrial transcriptional factor A breaks the mitochondria-mediated vicious cycle in Alzheimer’s disease. Scientific Reports. 6(1). 37889–37889. 61 indexed citations

Leon, Julio, Kunihiko Sakumi, Erika Castillo, et al.. (2016). 8-Oxoguanine accumulation in mitochondrial DNA causes mitochondrial dysfunction and impairs neuritogenesis in cultured adult mouse cortical neurons under oxidative conditions. Scientific Reports. 6(1). 22086–22086. 81 indexed citations

Kunisada, Makoto, Eiji Nakano, Ryusuke Ono, et al.. (2014). Inhibitory Effects of Dietary Spirulina platensis on UVB-Induced Skin Inflammatory Responses and Carcinogenesis. Journal of Investigative Dermatology. 134(10). 2610–2619. 50 indexed citations

Oka, Sugako, Julio Leon, Daisuke Tsuchimoto, Kunihiko Sakumi, & Yusaku Nakabeppu. (2014). MUTYH, an adenine DNA glycosylase, mediates p53 tumor suppression via PARP-dependent cell death. Oncogenesis. 3(10). e121–e121. 46 indexed citations

10.

Tsuji, Hideo, Naofumi Shiomi, Takanori Katsube, et al.. (2013). Nature of nontargeted radiation effects observed during fractionated irradiation-induced thymic lymphomagenesis in mice. Journal of Radiation Research. 54(3). 453–466. 3 indexed citations

11.

Hokama, Masaaki, Sugako Oka, Julio Leon, et al.. (2013). Altered Expression of Diabetes-Related Genes in Alzheimer's Disease Brains: The Hisayama Study. Cerebral Cortex. 24(9). 2476–2488. 258 indexed citations

12.

Murakami, Yusuke, Yasuhiro Ikeda, Noriko Yoshida, et al.. (2012). MutT Homolog-1 Attenuates Oxidative DNA Damage and Delays Photoreceptor Cell Death in Inherited Retinal Degeneration. American Journal Of Pathology. 181(4). 1378–1386. 36 indexed citations

13.

Sheng, Zijing, Sugako Oka, Daisuke Tsuchimoto, et al.. (2012). 8-Oxoguanine causes neurodegeneration during MUTYH-mediated DNA base excision repair. Journal of Clinical Investigation. 122(12). 4344–4361. 111 indexed citations

14.

Oka, Sugako & Yusaku Nakabeppu. (2011). DNA glycosylase encoded by MUTYH functions as a molecular switch for programmed cell death under oxidative stress to suppress tumorigenesis. Cancer Science. 102(4). 677–682. 66 indexed citations

15.

Nakabeppu, Yusaku, Sugako Oka, Zijing Sheng, Daisuke Tsuchimoto, & Kunihiko Sakumi. (2010). Programmed cell death triggered by nucleotide pool damage and its prevention by MutT homolog-1 (MTH1) with oxidized purine nucleoside triphosphatase. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 703(1). 51–58. 58 indexed citations

16.

Behmanesh, Mehrdad, Kunihiko Sakumi, Nona Abolhassani, et al.. (2009). ITPase-deficient mice show growth retardation and die before weaning. Cell Death and Differentiation. 16(10). 1315–1322. 60 indexed citations

17.

Oka, Sugako, Mizuki Ohno, & Yusaku Nakabeppu. (2009). Construction and Characterization of a Cell Line Deficient in Repair of Mitochondrial, but Not Nuclear, Oxidative DNA Damage. Methods in molecular biology. 554. 251–264. 4 indexed citations

18.

Oka, Sugako, Mizuki Ohno, Daisuke Tsuchimoto, et al.. (2008). Two distinct pathways of cell death triggered by oxidative damage to nuclear and mitochondrial DNAs. The EMBO Journal. 27(2). 421–432. 190 indexed citations

19.

Ichikawa, Junji, Daisuke Tsuchimoto, Sugako Oka, et al.. (2007). Oxidation of mitochondrial deoxynucleotide pools by exposure to sodium nitroprusside induces cell death. DNA repair. 7(3). 418–430. 55 indexed citations

20.

Mishima, Kenji, et al.. (1997). Measurement and correlation of solubilities of oxygen in aqueous solutions containing ribose and raffinose. Fluid Phase Equilibria. 134(1-2). 277–283. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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