Subhash D. Katewa

3.0k total citations
29 papers, 2.1k citations indexed

About

Subhash D. Katewa is a scholar working on Aging, Molecular Biology and Physiology. According to data from OpenAlex, Subhash D. Katewa has authored 29 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Aging, 13 papers in Molecular Biology and 9 papers in Physiology. Recurrent topics in Subhash D. Katewa's work include Genetics, Aging, and Longevity in Model Organisms (16 papers), Mitochondrial Function and Pathology (6 papers) and Circadian rhythm and melatonin (6 papers). Subhash D. Katewa is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (16 papers), Mitochondrial Function and Pathology (6 papers) and Circadian rhythm and melatonin (6 papers). Subhash D. Katewa collaborates with scholars based in United States, India and Australia. Subhash D. Katewa's co-authors include Pankaj Kapahi, Aric N. Rogers, J. William O. Ballard, Lutz Kockel, Emma Thomas, Di Chen, Surendra S. Katyare, Seymour Benzer, Brian M. Zid and Richard G. Melvin and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Subhash D. Katewa

29 papers receiving 2.1k citations

Peers

Countries citing papers authored by Subhash D. Katewa

Since Specialization

Citations

This map shows the geographic impact of Subhash D. Katewa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Subhash D. Katewa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Subhash D. Katewa more than expected).

Fields of papers citing papers by Subhash D. Katewa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Subhash D. Katewa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Subhash D. Katewa. The network helps show where Subhash D. Katewa may publish in the future.

Co-authorship network of co-authors of Subhash D. Katewa

This figure shows the co-authorship network connecting the top 25 collaborators of Subhash D. Katewa. A scholar is included among the top collaborators of Subhash D. Katewa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Subhash D. Katewa. Subhash D. Katewa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Dietary restriction and the transcription factor clock delay eye aging to extend lifespan in Drosophila Melanogaster 2022 ·Nature Communications ·Brian A. Hodge, (unknown), Subhash D. Katewa, Ting Lian, (unknown), (unknown), Nicole Y. Leung, (unknown), David Li‐Kroeger, Simon Melov, Birgit Schilling, Craig Montell, Pankaj Kapahi	22
2	Dietary restriction improves intestinal cellular fitness to enhance gut barrier function and lifespan in D. melanogaster 2018 ·PLoS Genetics ·Kazutaka Akagi, Kenneth A. Wilson, Subhash D. Katewa, (unknown), (unknown), Tyler Hilsabeck, (unknown), (unknown), Heinrich Jasper, Pankaj Kapahi	46
3	A genome-wide screen of bacterial mutants that enhance dauer formation in C. elegans 2016 ·Scientific Reports ·Amit Khanna, Jitendra Kumar, (unknown), (unknown), Subhash D. Katewa, Patrick Li, (unknown), (unknown), (unknown), Christopher S. Nelson, Rachel B. Brem, David W. Killilea, Sean D. Mooney, Matthew S. Gill, Pankaj Kapahi	16
4	Intestinal IRE1 Is Required for Increased Triglyceride Metabolism and Longer Lifespan under Dietary Restriction 2016 ·Cell Reports ·Nuno Miguel Luis, Lifen Wang, (unknown), Hansong Deng, Subhash D. Katewa, (unknown), Jason Karpac, Heinrich Jasper, Pankaj Kapahi	51
5	Peripheral Circadian Clocks Mediate Dietary Restriction-Dependent Changes in Lifespan and Fat Metabolism in Drosophila 2015 ·Cell Metabolism ·Subhash D. Katewa, Kazutaka Akagi, Neelanjan Bose, Kuntol Rakshit, (unknown), Xiangzhong Zheng, David Hall, Sonnet S. Davis, Christopher S. Nelson, Rachel B. Brem, Arvind Ramanathan, Amita Sehgal, Jadwiga M. Giebułtowicz, Pankaj Kapahi	130
6	Intramyocellular Fatty-Acid Metabolism Plays a Critical Role in Mediating Responses to Dietary Restriction in Drosophila melanogaster 2012 ·Cell Metabolism ·Subhash D. Katewa, Fabio Demontis, (unknown), Alan Hubbard, Matthew S. Gill, Norbert Perrimon, Simon Melov, Pankaj Kapahi	133
7	Role of TOR signaling in aging and related biological processes in Drosophila melanogaster 2010 ·Experimental Gerontology ·Subhash D. Katewa, Pankaj Kapahi	109
8	Dietary restriction and aging, 2009 2010 ·Aging Cell ·Subhash D. Katewa, Pankaj Kapahi	61
9	With TOR, Less Is More: A Key Role for the Conserved Nutrient-Sensing TOR Pathway in Aging 2010 ·Cell Metabolism ·Pankaj Kapahi, Di Chen, Aric N. Rogers, Subhash D. Katewa, (unknown), Emma Thomas, Lutz Kockel	497
10	4E-BP Extends Lifespan upon Dietary Restriction by Enhancing Mitochondrial Activity in Drosophila 2009 ·Cell ·Brian M. Zid, Aric N. Rogers, Subhash D. Katewa, (unknown), (unknown), (unknown), Seymour Benzer, Pankaj Kapahi	420
11	A Candidate Complex Approach to Study Functional Mitochondrial DNA Changes: Sequence Variation and Quaternary Structure Modeling of Drosophila simulans Cytochrome c Oxidase 2008 ·Journal of Molecular Evolution ·Richard G. Melvin, Subhash D. Katewa, J. William O. Ballard	19
12	Sympatric Drosophila simulans flies with distinct mtDNA show age related differences in mitochondrial metabolism 2007 ·Insect Biochemistry and Molecular Biology ·Subhash D. Katewa, J. William O. Ballard	12
13	Comparative Analysis of Mitochondrial Genotype and Aging 2007 ·Annals of the New York Academy of Sciences ·J. William O. Ballard, Subhash D. Katewa, Richard G. Melvin, Grace Chan	13
14	MITOCHONDRIAL DNA VARIATION IS ASSOCIATED WITH MEASURABLE DIFFERENCES IN LIFE-HISTORY TRAITS AND MITOCHONDRIAL METABOLISM IN DROSOPHILA SIMULANS 2007 ·Evolution ·J. William O. Ballard, Richard G. Melvin, Subhash D. Katewa, (unknown)	99
15	Sex differences in survival and mitochondrial bioenergetics during aging inDrosophila 2007 ·Aging Cell ·J. William O. Ballard, Richard G. Melvin, Joseph T. Miller, Subhash D. Katewa	48
16	Sympatric Drosophila simulans flies with distinct mtDNA show difference in mitochondrial respiration and electron transport 2006 ·Insect Biochemistry and Molecular Biology ·Subhash D. Katewa, J. William O. Ballard	37
17	Antimalarials Inhibit Human Erythrocyte Membrane Acetylcholinesterase 2005 ·Drug and Chemical Toxicology ·Subhash D. Katewa, Surendra S. Katyare	10
18	Effect of antimalarials treatment on rat liver lysosomal function—Anin vivo study 2005 ·Indian Journal of Clinical Biochemistry ·Samir P. Patel, Subhash D. Katewa, Surendra S. Katyare	4
19	Treatment with Antimalarials Adversely Affects the Oxidative Energy Metabolism in Rat Liver Mitochondria 2004 ·Drug and Chemical Toxicology ·Subhash D. Katewa, Surendra S. Katyare	36
20	A simplified method for inorganic phosphate determination and its application for phosphate analysis in enzyme assays 2003 ·Analytical Biochemistry ·Subhash D. Katewa, Surendra S. Katyare	74

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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