Stuart D. Critz

3.0k total citations · 1 hit paper
30 papers, 2.5k citations indexed

About

Stuart D. Critz is a scholar working on Pathology and Forensic Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Stuart D. Critz has authored 30 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Pathology and Forensic Medicine, 12 papers in Molecular Biology and 10 papers in Cellular and Molecular Neuroscience. Recurrent topics in Stuart D. Critz's work include Cardiac Ischemia and Reperfusion (16 papers), Neuroscience and Neuropharmacology Research (7 papers) and Cardiac electrophysiology and arrhythmias (7 papers). Stuart D. Critz is often cited by papers focused on Cardiac Ischemia and Reperfusion (16 papers), Neuroscience and Neuropharmacology Research (7 papers) and Cardiac electrophysiology and arrhythmias (7 papers). Stuart D. Critz collaborates with scholars based in United States, Canada and Poland. Stuart D. Critz's co-authors include James M. Downey, Michael V. Cohen, Xi‐Ming Yang, Atsushi Nakano, Guang S. Liu, John H. Byrne, Yankun Yue, Tilley Pain, Gerd Heusch and Christopher Baines and has published in prestigious journals such as Journal of Biological Chemistry, Circulation Research and Journal of Neurophysiology.

In The Last Decade

Stuart D. Critz

30 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Opening of Mitochondrial K_ATPChannels Triggers the Preconditioned State by Generating Free Radicals

2000 552 citations Xi‐Ming Yang, Stuart D. Critz et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Stuart D. Critz United States	25	1.5k	989	731	547	533	30	2.5k
Petr Pauček United States	18	2.6k 1.7×	1.9k 1.9×	980 1.3×	700 1.3×	976 1.8×	22	3.6k
Bruce D. Ziman United States	25	715 0.5×	1.7k 1.7×	225 0.3×	489 0.9×	183 0.3×	45	2.7k
Heinz Gögelein Germany	30	469 0.3×	1.9k 1.9×	235 0.3×	878 1.6×	70 0.1×	55	2.8k
Noel W. Davies United Kingdom	25	716 0.5×	1.9k 2.0×	155 0.2×	1.1k 2.1×	63 0.1×	36	2.8k
Bhawanjit K. Brar United States	25	373 0.2×	902 0.9×	88 0.1×	262 0.5×	141 0.3×	32	2.3k
Masafumi Tagaya Japan	20	563 0.4×	966 1.0×	108 0.1×	661 1.2×	478 0.9×	29	2.5k
Mark J. Dunne United Kingdom	40	706 0.5×	2.6k 2.6×	113 0.2×	991 1.8×	72 0.1×	119	4.7k
Alicia Mattiazzi Argentina	34	622 0.4×	2.1k 2.2×	125 0.2×	448 0.8×	25 0.0×	113	3.3k
Kathryn A. Yamada United States	41	494 0.3×	3.1k 3.2×	122 0.2×	524 1.0×	32 0.1×	68	4.7k
Tomoe Y. Nakamura Japan	29	234 0.2×	1.9k 1.9×	63 0.1×	490 0.9×	45 0.1×	56	2.5k

Countries citing papers authored by Stuart D. Critz

Since Specialization

Citations

This map shows the geographic impact of Stuart D. Critz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stuart D. Critz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stuart D. Critz more than expected).

Fields of papers citing papers by Stuart D. Critz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stuart D. Critz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stuart D. Critz. The network helps show where Stuart D. Critz may publish in the future.

Co-authorship network of co-authors of Stuart D. Critz

This figure shows the co-authorship network connecting the top 25 collaborators of Stuart D. Critz. A scholar is included among the top collaborators of Stuart D. Critz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stuart D. Critz. Stuart D. Critz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Kuno, Atsushi, Stuart D. Critz, Lin Cui, et al.. (2007). Protein kinase C protects preconditioned rabbit hearts by increasing sensitivity of adenosine A2b-dependent signaling during early reperfusion. Journal of Molecular and Cellular Cardiology. 43(3). 262–271. 103 indexed citations

Kuno, Atsushi, et al.. (2006). Nicorandil opens mitochondrial KATP channels not only directly but also through a NO-PKG-dependent pathway. Basic Research in Cardiology. 102(1). 73–79. 21 indexed citations

Critz, Stuart D., Michael V. Cohen, & James M. Downey. (2005). Mechanisms of acetylcholine- and bradykinin-induced preconditioning. Vascular Pharmacology. 42(5-6). 201–209. 32 indexed citations

Qin, Qining, Xi‐Ming Yang, Lin Cui, et al.. (2004). Exogenous NO triggers preconditioning via a cGMP- and mitoK_ATP-dependent mechanism. American Journal of Physiology-Heart and Circulatory Physiology. 287(2). H712–H718. 77 indexed citations

Oldenburg, Olaf, Qining Qin, Thomas Krieg, et al.. (2004). Bradykinin induces mitochondrial ROS generation via NO, cGMP, PKG, and mitoK_ATP channel opening and leads to cardioprotection. American Journal of Physiology-Heart and Circulatory Physiology. 286(1). H468–H476. 218 indexed citations

Pace, Betty S., José Sangerman, Solomon F. Ofori‐Acquah, et al.. (2003). p38 MAP kinase activation mediates γ-globin gene induction in erythroid progenitors. Experimental Hematology. 31(11). 1089–1096. 76 indexed citations

Krenz, Maike, Olaf Oldenburg, Michael V. Cohen, et al.. (2002). Opening of ATP-sensitive potassium channels causes generation of free radicals in vascular smooth muscle cells. Basic Research in Cardiology. 97(5). 365–373. 115 indexed citations

Foley, Heather, Solomon F. Ofori‐Acquah, Akihiko Yoshimura, et al.. (2002). Stat3β Inhibits γ-Globin Gene Expression in Erythroid Cells. Journal of Biological Chemistry. 277(18). 16211–16219. 26 indexed citations

Nakano, Atsushi, Michael V. Cohen, Stuart D. Critz, & James M. Downey. (2000). SB 203580, an inhibitor of p38 MAPK, abolishes infarct-limiting effect of ischemic preconditioning in isolated rabbit hearts. Basic Research in Cardiology. 95(6). 466–471. 50 indexed citations

10.

Mocanu, Mihaela, Gary F. Baxter, Yuankun Yue, Stuart D. Critz, & Derek M. Yellon. (2000). The p38 MAPK inhibitor, SB203580, abrogates ischaemic preconditioning in rat heart but timing of administration is critical. Basic Research in Cardiology. 95(6). 472–478. 91 indexed citations

11.

Zimmer, Warren E., Ying Zhao, Aleksander F. Sikorski, et al.. (2000). The domain of brain β-spectrin responsible for synaptic vesicle association is essential for synaptic transmission. Brain Research. 881(1). 18–27. 21 indexed citations

12.

Sikorski, Aleksander F., José Sangerman, Steven R. Goodman, & Stuart D. Critz. (2000). Spectrin (βSpIIΣ1) is an essential component of synaptic transmission. Brain Research. 852(1). 161–166. 51 indexed citations

13.

Kim, Sung Ouk, Christopher Baines, Stuart D. Critz, et al.. (1999). Ischemia induced activation of heat shock protein 27 kinases and casein kinase 2 in the preconditioned rabbit heart. Biochemistry and Cell Biology. 77(6). 559–567. 42 indexed citations

14.

Baines, Christopher, Guang S. Liu, Mustafa Birincioğlu, et al.. (1999). Ischemic preconditioning depends on interaction between mitochondrial K_ATPchannels and actin cytoskeleton. American Journal of Physiology-Heart and Circulatory Physiology. 276(4). H1361–H1368. 142 indexed citations

15.

Critz, Stuart D., et al.. (1997). Pinacidil but not Nicorandil Opens ATP-sensitive K+Channels and Protects Against Simulated Ischemia in Rabbit Myocytes. Journal of Molecular and Cellular Cardiology. 29(4). 1123–1130. 25 indexed citations

16.

Critz, Stuart D., Barbara A. Wible, Héctor S. López, & A M Brown. (1993). Stable Expression and Regulation of a Rat Brain K⁺ Channel. Journal of Neurochemistry. 60(3). 1175–1178. 58 indexed citations

17.

Morganroth, Joel, Arthur Brown, Stuart D. Critz, et al.. (1993). Variability of the QTc interval: Impact on defining drug effect and low-frequency cardiac event. The American Journal of Cardiology. 72(6). B26–B31. 65 indexed citations

18.

Critz, Stuart D. & Robert E. Marc. (1992). Glutamate antagonists that block hyperpolarizing bipolar cells increase the release of dopamine from turtle retina. Visual Neuroscience. 9(3-4). 271–278. 17 indexed citations

19.

Critz, Stuart D., Douglas A. Baxter, & John H. Byrne. (1991). Modulatory effects of serotonin, FMRFamide, and myomodulin on the duration of action potentials, excitability, and membrane currents in tail sensory neurons of Aplysia. Journal of Neurophysiology. 66(6). 1912–1926. 67 indexed citations

20.

Ichinose, Mitsuyuki, Shogo Endo, Stuart D. Critz, Shirish Shenolikar, & John H. Byrne. (1990). Microcystin-LR, a potent protein phosphatase inhibitor, prolongs the serotonin- and cAMP-induced currents in sensory neurons ofAplysia californica. Brain Research. 533(1). 137–140. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact