Stewart H. Lecker
- Molecular Biology top 0.2%
- Physiology top 0.2%
- Cell Biology top 0.2%
- Epidemiology top 1%
- Rehabilitation top 0.1%
- Co-authors
- Alfred L. GoldbergWilliam E. MitchR. Thomas JagoeMarco SandriMarcelo D. GomesStefano SchiaffinoAmi NavonKenneth Walsh
- Topics
- Muscle Physiology and Disorders (25 papers)Ubiquitin and proteasome pathways (15 papers)Genetic Neurodegenerative Diseases (9 papers)
- Cited by
- RehabilitationAgingCell Biology
- Journals
- CellProceedings of the National Academy of SciencesJournal of the American Chemical Society
- Partner nations
- United StatesCanadaItaly
In The Last Decade
Stewart H. Lecker
61 papers receiving 13.2k citations
Hit Papers
Peers
Comparison fields: 5 of 149
- Molecular Biology 10.5k
- Physiology 4.2k
- Cell Biology 2.9k
- Epidemiology 1.8k
- Rehabilitation 1.7k
Countries citing papers authored by Stewart H. Lecker
This map shows the geographic impact of Stewart H. Lecker's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stewart H. Lecker with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stewart H. Lecker more than expected).
Fields of papers citing papers by Stewart H. Lecker
This network shows the impact of papers produced by Stewart H. Lecker. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stewart H. Lecker. The network helps show where Stewart H. Lecker may publish in the future.
Co-authorship network of co-authors of Stewart H. Lecker
This figure shows the co-authorship network connecting the top 25 collaborators of Stewart H. Lecker. A scholar is included among the top collaborators of Stewart H. Lecker based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stewart H. Lecker. Stewart H. Lecker is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 2 | |
| 3 | 0 | |
| 4 | 3 | |
| 5 | 20 | |
| 6 | 62 | |
| 7 | 1 | |
| 8 | 22 | |
| 9 | 66 | |
| 10 | 102 | |
| 11 | FoxO3 Coordinately Activates Protein Degradation by the Autophagic/Lysosomal and Proteasomal Pathways in Atrophying Muscle Cellsbreakdown → | 1206 |
| 12 | 24 | |
| 13 | Protein Degradation by the Ubiquitin–Proteasome Pathway in Normal and Disease Statesbreakdown → | 959 |
| 14 | Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophybreakdown → | 2354 |
| 15 | 274 | |
| 16 | 94 | |
| 17 | Muscle Protein Breakdown and the Critical Role of the Ubiquitin-Proteasome Pathway in Normal and Disease Statesbreakdown → | 592 |
| 18 | 5 | |
| 19 | 477 | |
| 20 | 170 |
About Stewart H. Lecker
Stewart H. Lecker is a scholar working on Nephrology, Rehabilitation and Cell Biology, having authored 63 papers that have together received 13.4k indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (25 papers), Ubiquitin and proteasome pathways (15 papers) and Genetic Neurodegenerative Diseases (9 papers). The work is most often cited by research in Rehabilitation (1.7k citations), Aging (415 citations) and Cell Biology (2.9k citations). Stewart H. Lecker has collaborated with scholars based in United States, Canada and Italy. Frequent co-authors include Alfred L. Goldberg, William E. Mitch, R. Thomas Jagoe, Marco Sandri, Marcelo D. Gomes, Stefano Schiaffino, Ami Navon, Kenneth Walsh, Carsten Skurk and Anne Picard. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Journal of the American Chemical Society.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.