Stewart Bates

4.3k citations
18 papers · 3.0k indexed · h-index 16
Topics
Cancer-related Molecular Pathways (16 papers)Cancer Research and Treatments (7 papers)Cell death mechanisms and regulation (3 papers)

In The Last Decade

Stewart Bates

18 papers receiving 2.9k citations

Peers

Stewart Bates
Comparison fields: 5 of 98
  • Oncology 2.2k
  • Molecular Biology 2.2k
  • Biotechnology 500
  • Cancer Research 438
  • Cell Biology 359
Replace Andrew C. Phillips with:
Andrew C. Phillips United States
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Elisheva Yonish-Rouach France
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Citations per field
00.5×1.5×
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Citations per year

Countries citing papers authored by Stewart Bates

Since Specialization
Citations

This map shows the geographic impact of Stewart Bates's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stewart Bates with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stewart Bates more than expected).

Fields of papers citing papers by Stewart Bates

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stewart Bates. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stewart Bates. The network helps show where Stewart Bates may publish in the future.

Co-authorship network of co-authors of Stewart Bates

This figure shows the co-authorship network connecting the top 25 collaborators of Stewart Bates. A scholar is included among the top collaborators of Stewart Bates based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stewart Bates. Stewart Bates is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
#WorkIndexed citations
1 204
2 261
3 186
4 14
5 149
6 237
7 89
8 42
9
Regulation and function of the cyclin-dependent kinase inhibitor p16CDKN2.
3
10 228
11 306
12 259
13 93
14
Evidence for different modes of action of cyclin-dependent kinase inhibitors: p15 and p16 bind to kinases, p21 and p27 bind to cyclins.
155
15 313
16
Absence of cyclin D/cdk complexes in cells lacking functional retinoblastoma protein.
162
17
CDK6 (PLSTIRE) and CDK4 (PSK-J3) are a distinct subset of the cyclin-dependent kinases that associate with cyclin D1.
229
18 23

About Stewart Bates

Stewart Bates is a scholar working on Biotechnology, Oncology and Ophthalmology, having authored 18 papers that have together received 3.0k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (16 papers), Cancer Research and Treatments (7 papers) and Cell death mechanisms and regulation (3 papers). The work is most often cited by research in Oncology (2.2k citations), Biotechnology (500 citations) and Molecular Biology (2.2k citations). Stewart Bates has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include Karen H. Vousden, Godefridus J. Peters, David Parry, Andrew C. Phillips, Kevin M. Ryan, David J. Mann, Robert L. Ludwig, Éva Bálint, Laura Bonetta and Clive Dickson. Their work appears in journals such as Genes & Development, The EMBO Journal and Molecular Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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