Stewart Bates

4.3k total citations
18 papers, 3.0k citations indexed

About

Stewart Bates is a scholar working on Oncology, Molecular Biology and Biotechnology. According to data from OpenAlex, Stewart Bates has authored 18 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Oncology, 11 papers in Molecular Biology and 7 papers in Biotechnology. Recurrent topics in Stewart Bates's work include Cancer-related Molecular Pathways (16 papers), Cancer Research and Treatments (7 papers) and Cell death mechanisms and regulation (3 papers). Stewart Bates is often cited by papers focused on Cancer-related Molecular Pathways (16 papers), Cancer Research and Treatments (7 papers) and Cell death mechanisms and regulation (3 papers). Stewart Bates collaborates with scholars based in United States, United Kingdom and Germany. Stewart Bates's co-authors include Karen H. Vousden, Godefridus J. Peters, David Parry, Andrew C. Phillips, Kevin M. Ryan, David J. Mann, Robert L. Ludwig, Éva Bálint, Laura Bonetta and Clive Dickson and has published in prestigious journals such as Genes & Development, The EMBO Journal and Molecular Cell.

In The Last Decade

Stewart Bates

18 papers receiving 2.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Stewart Bates United States	16	2.2k	2.2k	500	438	359	18	3.0k
Carl G. Maki United States	27	1.8k 0.8×	2.3k 1.1×	400 0.8×	584 1.3×	328 0.9×	62	2.9k
Barbara A. Osborne United States	6	1.7k 0.8×	2.1k 1.0×	474 0.9×	469 1.1×	175 0.5×	8	3.0k
Andrew C. Phillips United States	18	1.7k 0.8×	2.1k 0.9×	333 0.7×	378 0.9×	270 0.8×	29	2.7k
Elisheva Yonish-Rouach France	12	1.7k 0.8×	1.9k 0.9×	502 1.0×	417 1.0×	154 0.4×	20	2.7k
Daniele Bergamaschi United Kingdom	23	1.6k 0.7×	2.1k 1.0×	496 1.0×	593 1.4×	305 0.8×	42	3.1k
Rebecca Haffner Israel	12	1.4k 0.6×	1.8k 0.8×	383 0.8×	336 0.8×	204 0.6×	15	2.3k
Aart G. Jochemsen Netherlands	26	3.0k 1.3×	3.0k 1.4×	658 1.3×	545 1.2×	365 1.0×	37	3.7k
Mark K. Saville United Kingdom	23	1.9k 0.8×	2.7k 1.2×	267 0.5×	490 1.1×	362 1.0×	33	3.2k
Anne M. Theodoras United States	8	2.1k 0.9×	2.5k 1.1×	186 0.4×	399 0.9×	622 1.7×	8	3.2k
Mark A. Subler United States	32	1.5k 0.7×	2.0k 0.9×	268 0.5×	412 0.9×	226 0.6×	65	3.0k

Countries citing papers authored by Stewart Bates

Since Specialization

Citations

This map shows the geographic impact of Stewart Bates's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stewart Bates with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stewart Bates more than expected).

Fields of papers citing papers by Stewart Bates

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stewart Bates. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stewart Bates. The network helps show where Stewart Bates may publish in the future.

Co-authorship network of co-authors of Stewart Bates

This figure shows the co-authorship network connecting the top 25 collaborators of Stewart Bates. A scholar is included among the top collaborators of Stewart Bates based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stewart Bates. Stewart Bates is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Phillips, Andrew C., Mary K. Ernst, Stewart Bates, Nancy R. Rice, & Karen H. Vousden. (1999). E2F-1 Potentiates Cell Death by Blocking Antiapoptotic Signaling Pathways. Molecular Cell. 4(5). 771–781. 204 indexed citations

Bates, Stewart & Karen H. Vousden. (1999). Mechanisms of p53-mediated apoptosis. Cellular and Molecular Life Sciences. 55(1). 28–37. 261 indexed citations

Bálint, Éva, Stewart Bates, & Karen H. Vousden. (1999). Mdm2 binds p73α without targeting degradation. Oncogene. 18(27). 3923–3929. 186 indexed citations

Bates, Stewart, Emma Hickman, & Karen H. Vousden. (1999). Reversal of p53-induced cell-cycle arrest. Molecular Carcinogenesis. 24(1). 7–14. 14 indexed citations

Bates, Stewart, Kevin M. Ryan, Andrew C. Phillips, & Karen H. Vousden. (1998). Cell cycle arrest and DNA endoreduplication following p21Waf1/Cip1 expression. Oncogene. 17(13). 1691–1703. 149 indexed citations

Phillips, Andrew C., Stewart Bates, Kevin M. Ryan, Kristian Helin, & Karen H. Vousden. (1997). Induction of DNA synthesis and apoptosis are separable functions of E2F-1.. Genes & Development. 11(14). 1853–1863. 237 indexed citations

Palmero, Ignacio, Beth B. McConnell, David Parry, et al.. (1997). Accumulation of p16INK4a in mouse fibroblasts as a function of replicative senescence and not of retinoblastoma gene status. Oncogene. 15(5). 495–503. 89 indexed citations

Hickman, Emma, Stewart Bates, & Karen H. Vousden. (1997). Perturbation of the p53 response by human papillomavirus type 16 E7. Journal of Virology. 71(5). 3710–3718. 42 indexed citations

Peters, Godefridus J., David Parry, Eiji Hara, et al.. (1997). Regulation and function of the cyclin-dependent kinase inhibitor p16CDKN2.. PubMed. 11 Suppl 3. 352–5. 3 indexed citations

10.

Ludwig, Robert L., Stewart Bates, & Karen H. Vousden. (1996). Differential Activation of Target Cellular Promoters by p53 Mutants with Impaired Apoptotic Function. Molecular and Cellular Biology. 16(9). 4952–4960. 228 indexed citations

11.

Bates, Stewart & Karen H. Vousden. (1996). p53 in signaling checkpoint arrest or apoptosis. Current Opinion in Genetics & Development. 6(1). 12–18. 306 indexed citations

12.

Rowan, Sheldon, R Ludwig, Ygal Haupt, et al.. (1996). Specific loss of apoptotic but not cell-cycle arrest function in a human tumor derived p53 mutant.. The EMBO Journal. 15(4). 827–838. 259 indexed citations

13.

Bates, Stewart & Gordon Peters. (1995). Cyclin D1 as a cellular proto-oncogene. Seminars in Cancer Biology. 6(2). 73–82. 93 indexed citations

14.

Hall, Marcia, Stewart Bates, & Godefridus J. Peters. (1995). Evidence for different modes of action of cyclin-dependent kinase inhibitors: p15 and p16 bind to kinases, p21 and p27 bind to cyclins.. PubMed. 11(8). 1581–8. 155 indexed citations

15.

Parry, David, Stewart Bates, David J. Mann, & Godefridus J. Peters. (1995). Lack of cyclin D-Cdk complexes in Rb-negative cells correlates with high levels of p16INK4/MTS1 tumour suppressor gene product.. The EMBO Journal. 14(3). 503–511. 313 indexed citations

16.

Bates, Stewart, David Parry, Laura Bonetta, et al.. (1994). Absence of cyclin D/cdk complexes in cells lacking functional retinoblastoma protein.. PubMed. 9(6). 1633–40. 162 indexed citations

17.

Bates, Stewart, Laura Bonetta, Derek C. Macallan, et al.. (1994). CDK6 (PLSTIRE) and CDK4 (PSK-J3) are a distinct subset of the cyclin-dependent kinases that associate with cyclin D1.. PubMed. 9(1). 71–9. 229 indexed citations

18.

Fisher, Richard I., Stewart Bates, Benjamín C. Bostick, Narendra Tuteja, & Volker Diehl. (1984). Neoplastic cells obtained from Hodgkin's disease function as accessory cells for mitogen-induced human T cell proliferative responses.. The Journal of Immunology. 132(5). 2672–2677. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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