Stephen M. Lewis

2.2k total citations
44 papers, 1.7k citations indexed

About

Stephen M. Lewis is a scholar working on Molecular Biology, Cancer Research and Cell Biology. According to data from OpenAlex, Stephen M. Lewis has authored 44 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Molecular Biology, 14 papers in Cancer Research and 10 papers in Cell Biology. Recurrent topics in Stephen M. Lewis's work include Extracellular vesicles in disease (13 papers), RNA modifications and cancer (12 papers) and RNA and protein synthesis mechanisms (11 papers). Stephen M. Lewis is often cited by papers focused on Extracellular vesicles in disease (13 papers), RNA modifications and cancer (12 papers) and RNA and protein synthesis mechanisms (11 papers). Stephen M. Lewis collaborates with scholars based in Canada, United States and Germany. Stephen M. Lewis's co-authors include Martin Holčı́k, Stéphan Vagner, Rodney J. Ouellette, Sophie Bonnal, Myriam Gorospe, Anirban Ghosh, Tyson E. Graber, Stephen Baird, Simi Chacko and Urszula Liwak and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and SHILAP Revista de lepidopterología.

In The Last Decade

Stephen M. Lewis

44 papers receiving 1.7k citations

Peers

Stephen M. Lewis
Rainer Will Germany
Stephen M. Lewis
Citations per year, relative to Stephen M. Lewis Stephen M. Lewis (= 1×) peers Rainer Will

Countries citing papers authored by Stephen M. Lewis

Since Specialization
Citations

This map shows the geographic impact of Stephen M. Lewis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen M. Lewis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen M. Lewis more than expected).

Fields of papers citing papers by Stephen M. Lewis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stephen M. Lewis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen M. Lewis. The network helps show where Stephen M. Lewis may publish in the future.

Co-authorship network of co-authors of Stephen M. Lewis

This figure shows the co-authorship network connecting the top 25 collaborators of Stephen M. Lewis. A scholar is included among the top collaborators of Stephen M. Lewis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stephen M. Lewis. Stephen M. Lewis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Peña‐Castillo, Lourdes, et al.. (2023). Extracellular vesicle small RNA cargo discriminates non-cancer donors from pediatric B-lymphoblastic leukemia patients. Frontiers in Oncology. 13. 1272883–1272883. 1 indexed citations
2.
Wajnberg, Gabriel, Éric P. Allain, Jeremy Roy, et al.. (2023). Application of annotation-agnostic RNA sequencing data analysis tools for biomarker discovery in liquid biopsy. SHILAP Revista de lepidopterología. 3. 1127661–1127661. 5 indexed citations
3.
Roy, Jeremy, Catherine A. Taylor, Rodney J. Ouellette, & Stephen M. Lewis. (2022). Peptide-Affinity Isolation of Extracellular Vesicles and Cell-Free DNA From Human Plasma. Methods in molecular biology. 2508. 341–352. 3 indexed citations
4.
Bullerwell, Charles E., Pierre Deprez, Andrew P. Joy, et al.. (2021). EBF1 drives hallmark B cell gene expression by enabling the interaction of PAX5 with the MLL H3K4 methyltransferase complex. Scientific Reports. 11(1). 1537–1537. 15 indexed citations
5.
Bera, Amit, et al.. (2020). Cellular stress orchestrates the localization of hnRNP H to stress granules. Experimental Cell Research. 394(1). 112111–112111. 14 indexed citations
6.
Smith, Nicole C., Gabriel Wajnberg, Simi Chacko, et al.. (2020). Characterization of miRNAs in Extracellular Vesicles Released From Atlantic Salmon Monocyte-Like and Macrophage-Like Cells. Frontiers in Immunology. 11. 587931–587931. 17 indexed citations
7.
Joy, Andrew P., D. Craig Ayre, Ian C. Chute, et al.. (2018). Proteome profiling of extracellular vesicles captured with the affinity peptide Vn96: comparison of Laemmli and TRIzol© protein‐extraction methods. Journal of Extracellular Vesicles. 7(1). 25 indexed citations
8.
Ayre, D. Craig, Ian C. Chute, Andrew P. Joy, et al.. (2017). CD24 induces changes to the surface receptors of B cell microvesicles with variable effects on their RNA and protein cargo. Scientific Reports. 7(1). 8642–8642. 18 indexed citations
9.
Salsman, Jayme, Daniel Gaston, Kimberly R. Kukurba, et al.. (2017). PML nuclear bodies contribute to the basal expression of the mTOR inhibitor DDIT4. Scientific Reports. 7(1). 45038–45038. 17 indexed citations
10.
Desnoyers, Guillaume, et al.. (2015). Decreased eIF3e Expression Can Mediate Epithelial-to-Mesenchymal Transition through Activation of the TGFβ Signaling Pathway. Molecular Cancer Research. 13(10). 1421–1430. 16 indexed citations
11.
Grabher, Clemens, Ian C. Chute, D. Léger, et al.. (2015). Epigenetic therapy restores normal hematopoiesis in a zebrafish model of NUP98–HOXA9-induced myeloid disease. Leukemia. 29(10). 2086–2097. 30 indexed citations
12.
Ghosh, Anirban, Michelle Davey, Ian C. Chute, et al.. (2014). Rapid Isolation of Extracellular Vesicles from Cell Culture and Biological Fluids Using a Synthetic Peptide with Specific Affinity for Heat Shock Proteins. PLoS ONE. 9(10). e110443–e110443. 158 indexed citations
13.
Lewis, Stephen M., et al.. (2012). Decreased eIF3e/Int6 expression causes epithelial-to-mesenchymal transition in breast epithelial cells. Oncogene. 32(31). 3598–3605. 27 indexed citations
14.
Poon, Pak P., et al.. (2010). The Yeast Arf GTPase-activating Protein Age1 Is Regulated by Phospholipase D for Post-Golgi Vesicular Transport. Journal of Biological Chemistry. 286(7). 5187–5196. 8 indexed citations
15.
Laflamme, Mark, et al.. (2009). Multiple isoforms of PAX5 are expressed in both lymphomas and normal B‐cells. British Journal of Haematology. 147(3). 328–338. 17 indexed citations
16.
Lewis, Stephen M., et al.. (2007). Subcellular Relocalization of a Trans-acting Factor Regulates XIAP IRES-dependent Translation. Molecular Biology of the Cell. 18(4). 1302–1311. 94 indexed citations
17.
Baird, Stephen, Stephen M. Lewis, Marcel Turcotte, & Martin Holčı́k. (2007). A search for structurally similar cellular internal ribosome entry sites. Nucleic Acids Research. 35(14). 4664–4677. 62 indexed citations
18.
Cloutier, M., et al.. (2006). Internal Ribosome Entry Site-mediated Translation of Apaf-1, but Not XIAP, Is Regulated during UV-induced Cell Death. Journal of Biological Chemistry. 281(22). 15155–15163. 34 indexed citations
19.
Holčı́k, Martin, Tyson E. Graber, Stephen M. Lewis, et al.. (2005). Spurious splicing within the XIAP 5′ UTR occurs in the Rluc/Fluc but not the βgal/CAT bicistronic reporter system. RNA. 11(11). 1605–1609. 57 indexed citations
20.
Lewis, Stephen M., Pak P. Poon, Richard A. Singer, Gerald C. Johnston, & Anne Spang. (2004). The ArfGAP Glo3 Is Required for the Generation of COPI Vesicles. Molecular Biology of the Cell. 15(9). 4064–4072. 61 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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