Amit Bera

475 total citations
15 papers, 409 citations indexed

About

Amit Bera is a scholar working on Molecular Biology, Nephrology and Cancer Research. According to data from OpenAlex, Amit Bera has authored 15 papers receiving a total of 409 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 5 papers in Nephrology and 4 papers in Cancer Research. Recurrent topics in Amit Bera's work include PI3K/AKT/mTOR signaling in cancer (7 papers), RNA modifications and cancer (4 papers) and Renal Diseases and Glomerulopathies (4 papers). Amit Bera is often cited by papers focused on PI3K/AKT/mTOR signaling in cancer (7 papers), RNA modifications and cancer (4 papers) and Renal Diseases and Glomerulopathies (4 papers). Amit Bera collaborates with scholars based in United States and Canada. Amit Bera's co-authors include Balakuntalam S. Kasinath, Nandini Ghosh‐Choudhury, Goutam Ghosh Choudhury, Falguni Das, Nirmalya Dey, Hanna E. Abboud, Meenalakshmi M. Mariappan, Stephen M. Lewis, Soumya Maity and Sanjay Pal and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and The FASEB Journal.

In The Last Decade

Amit Bera

15 papers receiving 404 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Amit Bera United States 13 311 145 99 35 34 15 409
Lalita Uttarwar Canada 7 211 0.7× 134 0.9× 64 0.6× 16 0.5× 24 0.7× 7 351
Marta Fierro‐Fernández Spain 10 268 0.9× 118 0.8× 36 0.4× 60 1.7× 41 1.2× 13 397
Joseph Washburn United States 8 331 1.1× 155 1.1× 55 0.6× 42 1.2× 16 0.5× 11 478
Eric Schordan France 12 294 0.9× 119 0.8× 108 1.1× 86 2.5× 24 0.7× 19 482
Lina K. Sciesielski Germany 9 230 0.7× 177 1.2× 27 0.3× 72 2.1× 37 1.1× 14 366
Shweta Pandya United States 4 244 0.8× 176 1.2× 72 0.7× 14 0.4× 9 0.3× 5 348
Tania Tsatralis Australia 10 191 0.6× 79 0.5× 25 0.3× 34 1.0× 14 0.4× 11 335
Yiting Jia China 10 216 0.7× 109 0.8× 25 0.3× 64 1.8× 25 0.7× 14 435
Anja Bondke Germany 6 263 0.8× 85 0.6× 38 0.4× 78 2.2× 42 1.2× 9 388

Countries citing papers authored by Amit Bera

Since Specialization
Citations

This map shows the geographic impact of Amit Bera's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amit Bera with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amit Bera more than expected).

Fields of papers citing papers by Amit Bera

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amit Bera. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amit Bera. The network helps show where Amit Bera may publish in the future.

Co-authorship network of co-authors of Amit Bera

This figure shows the co-authorship network connecting the top 25 collaborators of Amit Bera. A scholar is included among the top collaborators of Amit Bera based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amit Bera. Amit Bera is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Das, Falguni, Amit Bera, Nandini Ghosh‐Choudhury, et al.. (2021). High glucose-stimulated enhancer of zeste homolog-2 (EZH2) forces suppression of deptor to cause glomerular mesangial cell pathology. Cellular Signalling. 86. 110072–110072. 10 indexed citations
2.
Mathavarajah, Sabateeshan, Amit Bera, Jayme Salsman, et al.. (2021). Haploinsufficient tumor suppressor PRP4K is negatively regulated during epithelial‐to‐mesenchymal transition. The FASEB Journal. 35(11). e22001–e22001. 3 indexed citations
3.
Bera, Amit, et al.. (2020). Cellular stress orchestrates the localization of hnRNP H to stress granules. Experimental Cell Research. 394(1). 112111–112111. 14 indexed citations
4.
Bera, Amit & Stephen M. Lewis. (2020). Regulation of Epithelial-to-Mesenchymal Transition by Alternative Translation Initiation Mechanisms and Its Implications for Cancer Metastasis. International Journal of Molecular Sciences. 21(11). 4075–4075. 18 indexed citations
5.
Maity, Soumya, Amit Bera, Nandini Ghosh‐Choudhury, et al.. (2018). microRNA-181a downregulates deptor for TGFβ-induced glomerular mesangial cell hypertrophy and matrix protein expression. Experimental Cell Research. 364(1). 5–15. 16 indexed citations
6.
Bera, Amit, Falguni Das, Nandini Ghosh‐Choudhury, et al.. (2017). Reciprocal regulation of miR-214 and PTEN by high glucose regulates renal glomerular mesangial and proximal tubular epithelial cell hypertrophy and matrix expansion. American Journal of Physiology-Cell Physiology. 313(4). C430–C447. 59 indexed citations
7.
Das, Falguni, Nirmalya Dey, Amit Bera, et al.. (2016). MicroRNA-214 Reduces Insulin-like Growth Factor-1 (IGF-1) Receptor Expression and Downstream mTORC1 Signaling in Renal Carcinoma Cells. Journal of Biological Chemistry. 291(28). 14662–14676. 35 indexed citations
8.
Dey, Nirmalya, Amit Bera, Falguni Das, et al.. (2015). High glucose enhances microRNA-26a to activate mTORC1 for mesangial cell hypertrophy and matrix protein expression. Cellular Signalling. 27(7). 1276–1285. 37 indexed citations
9.
Das, Falguni, Nandini Ghosh‐Choudhury, Nirmalya Dey, et al.. (2014). High Glucose Forces a Positive Feedback Loop Connecting Akt Kinase and FoxO1 Transcription Factor to Activate mTORC1 Kinase for Mesangial Cell Hypertrophy and Matrix Protein Expression. Journal of Biological Chemistry. 289(47). 32703–32716. 36 indexed citations
10.
Das, Falguni, Amit Bera, Nandini Ghosh‐Choudhury, et al.. (2014). TGFβ-Induced Deptor Suppression Recruits mTORC1 and Not mTORC2 to Enhance Collagen I (α2) Gene Expression. PLoS ONE. 9(10). e109608–e109608. 33 indexed citations
11.
Bera, Amit, Falguni Das, Nandini Ghosh‐Choudhury, et al.. (2014). microRNA-21-induced dissociation of PDCD4 from rictor contributes to Akt-IKKβ-mTORC1 axis to regulate renal cancer cell invasion. Experimental Cell Research. 328(1). 99–117. 45 indexed citations
12.
Bera, Amit, Falguni Das, Nandini Ghosh‐Choudhury, et al.. (2014). A positive feedback loop involving Erk5 and Akt turns on mesangial cell proliferation in response to PDGF. American Journal of Physiology-Cell Physiology. 306(11). C1089–C1100. 19 indexed citations
13.
Das, Falguni, Nandini Ghosh‐Choudhury, Amit Bera, et al.. (2013). Transforming Growth Factor β Integrates Smad 3 to Mechanistic Target of Rapamycin Complexes to Arrest Deptor Abundance for Glomerular Mesangial Cell Hypertrophy. Journal of Biological Chemistry. 288(11). 7756–7768. 28 indexed citations
14.
Bera, Amit, Nandini Ghosh‐Choudhury, Nirmalya Dey, et al.. (2013). NFκB-mediated cyclin D1 expression by microRNA-21 influences renal cancer cell proliferation. Cellular Signalling. 25(12). 2575–2586. 39 indexed citations
15.
Das, Falguni, Nandini Ghosh‐Choudhury, Amit Bera, Balakuntalam S. Kasinath, & Goutam Ghosh Choudhury. (2013). TGFβ‐induced PI 3 kinase‐dependent Mnk‐1 activation is necessary for Ser‐209 phosphorylation of eIF4E and mesangial cell hypertrophy. Journal of Cellular Physiology. 228(7). 1617–1626. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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