Shaoping Xie

3.4k citations
20 papers · 1.4k indexed · h-index 17
Topics
Asthma and respiratory diseases (4 papers)NF-κB Signaling Pathways (3 papers)Cytokine Signaling Pathways and Interactions (3 papers)

In The Last Decade

Shaoping Xie

20 papers receiving 1.4k citations

Peers

Shaoping Xie
Comparison fields: 5 of 104
  • Molecular Biology 874
  • Physiology 282
  • Genetics 229
  • Immunology 206
  • Cancer Research 173
Replace William F. Jester with:
William F. Jester United States
Xi Shi China
Shuji Kubo Japan
Rebecca Hoffman United States
Kristina Forsman‐Semb Sweden
Olivier Kassel Germany
Ritsuko Shimizu Japan
Benoît Hennuy Belgium
Avinash Abhyankar United States
Stephen Wilson United States
Shaoping Xie relative to William F. Jester United States William F. Jester's profile →
Citations per field
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William F. Jester · 1×
Citations per year

Countries citing papers authored by Shaoping Xie

Since Specialization
Citations

This map shows the geographic impact of Shaoping Xie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shaoping Xie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shaoping Xie more than expected).

Fields of papers citing papers by Shaoping Xie

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shaoping Xie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shaoping Xie. The network helps show where Shaoping Xie may publish in the future.

Co-authorship network of co-authors of Shaoping Xie

This figure shows the co-authorship network connecting the top 25 collaborators of Shaoping Xie. A scholar is included among the top collaborators of Shaoping Xie based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shaoping Xie. Shaoping Xie is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 11
2 99
3 21
4 21
5 37
6
Expression of transforming growth factor- (TGF-) in chronic idiopathic cough
1
7 20
8 34
9 91
10 18
11 78
12 54
13 33
14 72
15 83
16 80
17 279
18 7
19 318
20 32

About Shaoping Xie

Shaoping Xie is a scholar working on Cancer Research, Developmental Neuroscience and Physiology, having authored 20 papers that have together received 1.4k indexed citations. Recurring topics across this work include Asthma and respiratory diseases (4 papers), NF-κB Signaling Pathways (3 papers) and Cytokine Signaling Pathways and Interactions (3 papers). The work is most often cited by research in Molecular Biology (874 citations), Cancer Research (173 citations) and Physiology (282 citations). Shaoping Xie has collaborated with scholars based in United Kingdom, United States and Singapore. Frequent co-authors include Kian Fan Chung, Maria B. Sukkar, Razao Issa, Taiping Chen, Yoshihide Ueda, En Li, Ute Oltmanns, Masahiro Nogami, Masaki Okano and Katsuzumi Okumura. Their work appears in journals such as Nucleic Acids Research, Journal of Biological Chemistry and The Journal of Immunology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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