Shanshan Xiong

2.7k citations
51 papers · 1.8k indexed · 1 hit paper · h-index 20

Shanshan Xiong

47 papers receiving 1.8k citations

Hit Papers

Repopulated microglia are solely derived from the prolife...3952018202620202023100200300

Peers

Shanshan Xiong
Comparison fields: 5 of 121
  • Neurology 535
  • Developmental Neuroscience 131
  • Biological Psychiatry 64
  • Immunology 477
  • Hepatology 138
Replace Yang Xiang with:
Yang Xiang China
Anna E. Beaudin United States
Cristina M. Ramírez Spain
Han Wang China
Jining Lü United States
Lan Zhao China
Manuel Varas‐Godoy Chile
Luca Massimino Italy
Jian Ye United States
Hiroshi Takahashi Japan
Shanshan Xiong relative to Yang Xiang China Yang Xiang's profile →
Citations per field
00.5×4.7×
Yang Xiang · 1×
Citations per year

Countries citing papers authored by Shanshan Xiong

Since Specialization
Citations

This map shows the geographic impact of Shanshan Xiong's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shanshan Xiong with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shanshan Xiong more than expected).

Fields of papers citing papers by Shanshan Xiong

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shanshan Xiong. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shanshan Xiong. The network helps show where Shanshan Xiong may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Shanshan Xiong, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Shanshan Xiong Line = papers co-authored together Shanshan Xiong links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20250
2 20247
3 20247
4 20240
5 20230
6 20238
7 202210
8 2021151
9 202118
10 20206
11
Repopulated microglia are solely derived from the proliferation of residual microglia after acute depletionbreakdown →
2018395
12 201824
13 201852
14 201849
15 201851
16 201874
17 201737
18 20161
19 20061
20 200550

About Shanshan Xiong

Shanshan Xiong is a scholar working on Neurology, Hepatology and Developmental Neuroscience, having authored 51 papers that have together received 1.8k indexed citations. Recurring topics across this work include Inflammatory Bowel Disease (14 papers), Neuroinflammation and Neurodegeneration Mechanisms (6 papers), Immune cells in cancer (6 papers), Microscopic Colitis (5 papers), Hepatitis B Virus Studies (5 papers), Hepatitis C virus research (5 papers), Gut microbiota and health (4 papers) and IL-33, ST2, and ILC Pathways (4 papers). The work is most often cited by research in Neurology (535 citations), Developmental Neuroscience (131 citations) and Biological Psychiatry (64 citations). Shanshan Xiong has collaborated with scholars based in China, United States and Hong Kong. Frequent co-authors include Ti‐Fei Yuan, Yanxia Rao, Bo Peng, Thaddeus S. Stappenbeck, Yubin Huang, Zhen Xu, Tianzhun Wu, Lei Zhao, Yu-Xiang Liang and Kwok‐Fai So. Their work appears in journals such as Nature Neuroscience, Cell, Cell Reports, Therapeutic Advances in Gastroenterology and Clinical Therapeutics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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