Shan Wei

47 papers receiving 768 citations

Peers

Shan Wei
Comparison fields: 5 of 105
  • Biological Psychiatry 16
  • Genetics 64
  • Neurology 47
  • Infectious Diseases 89
  • Cardiology and Cardiovascular Medicine 102
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Tong Lei China
Mingqiang Shen China
Shuang Wu China
Hongna Yang China
Lü Zhou China
Helen J. Dranse Canada
Stefania Greco Italy
Elisabetta Liverani United States
Madhu S. Malo United States
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Citations per year

Countries citing papers authored by Shan Wei

Since Specialization
Citations

This map shows the geographic impact of Shan Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shan Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shan Wei more than expected).

Fields of papers citing papers by Shan Wei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shan Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shan Wei. The network helps show where Shan Wei may publish in the future.

Co-authors

The 25 scholars most cited alongside Shan Wei, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Shan Wei Line = papers co-authored together Shan Wei links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 49 papers — load more, or switch the sort, to bring in the rest.

#Work
1 202083
2 199975
3 201745
4 201844
5 201139
6 201538
7 202237
8
A modest reduction in c-myc expression has minimal effects on cell growth and apoptosis but dramatically reduces susceptibility to Ras and Raf transformation.
200134
9 199925
10 201525
11 200322
12 201821
13 201721
14 201819
15 202018
16 202416
17 202216
18 201815
19 201714
20 201613

About Shan Wei

Shan Wei is a scholar working on Physiology, Immunology, Psychiatry and Mental health, Molecular Biology and Cardiology and Cardiovascular Medicine, having authored 49 papers that have together received 773 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (12 papers), Dementia and Cognitive Impairment Research (8 papers), Advanced Glycation End Products research (4 papers), Virus-based gene therapy research (3 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers), Cardiovascular Health and Disease Prevention (3 papers), Blood Pressure and Hypertension Studies (3 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (3 papers). The work is most often cited by research in Biological Psychiatry (16 citations), Genetics (64 citations), Neurology (47 citations), Infectious Diseases (89 citations) and Cardiology and Cardiovascular Medicine (102 citations). Shan Wei has collaborated with scholars based in China, United States and Hong Kong. Frequent co-authors include John E. Sanderson, Louis T.C. Chow, Yuansen Hu, Qiumin Qu, Liangjun Dang, Yangyong Lv, Ling Gao, He Huang, Binsheng Wang and Suhang Shang. Their work appears in journals such as Journal of Alzheimer s Disease, Frontiers in Aging Neuroscience, BMC Public Health, Lipids in Health and Disease and Journal of the Neurological Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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