Sergio Ruiz

5.9k total citations
49 papers, 2.9k citations indexed

About

Sergio Ruiz is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Sergio Ruiz has authored 49 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 42 papers in Molecular Biology, 16 papers in Oncology and 5 papers in Cell Biology. Recurrent topics in Sergio Ruiz's work include CRISPR and Genetic Engineering (18 papers), Pluripotent Stem Cells Research (15 papers) and Cancer-related Molecular Pathways (12 papers). Sergio Ruiz is often cited by papers focused on CRISPR and Genetic Engineering (18 papers), Pluripotent Stem Cells Research (15 papers) and Cancer-related Molecular Pathways (12 papers). Sergio Ruiz collaborates with scholars based in Spain, United States and Sweden. Sergio Ruiz's co-authors include Jesús M. Paramio, Carmen Segrelles, Athanasia D. Panopoulos, Juan Carlos Izpisúa Belmonte, Mirentxu Santos, José L. Jorcano, Kun Zhang, Dinh Diep, Margaret Lutz and W. Travis Berggren and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Sergio Ruiz

47 papers receiving 2.9k citations

Peers

Sergio Ruiz
Jamila Laoukili Netherlands
Shoutian Zhu United States
Arnout Schepers Netherlands
Sergio Ruiz
Citations per year, relative to Sergio Ruiz Sergio Ruiz (= 1×) peers Akira Kurisaki

Countries citing papers authored by Sergio Ruiz

Since Specialization
Citations

This map shows the geographic impact of Sergio Ruiz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sergio Ruiz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sergio Ruiz more than expected).

Fields of papers citing papers by Sergio Ruiz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sergio Ruiz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sergio Ruiz. The network helps show where Sergio Ruiz may publish in the future.

Co-authorship network of co-authors of Sergio Ruiz

This figure shows the co-authorship network connecting the top 25 collaborators of Sergio Ruiz. A scholar is included among the top collaborators of Sergio Ruiz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sergio Ruiz. Sergio Ruiz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Protasoni, Margherita, Camille Stephan‐Otto Attolini, Nicolás Herranz, et al.. (2024). Cyclophilin D plays a critical role in the survival of senescent cells. The EMBO Journal. 43(23). 5972–6000. 12 indexed citations
2.
Berenguer, Juan, Alejandro Álvaro‐Meca, Sergio Ruiz, et al.. (2023). Three Years of the Coronavirus Disease 2019 Pandemic in a European Region: A Population-Based Longitudinal Assessment in Madrid Between 2020 and 2022. Open Forum Infectious Diseases. 11(1). ofad635–ofad635.
3.
Guldner, Ian H., Min-Zu Wu, Yuriko Hishida, et al.. (2020). Cell surface GRP78 promotes stemness in normal and neoplastic cells. Scientific Reports. 10(1). 3474–3474. 39 indexed citations
4.
Ruiz, Sergio, Cristina Mayor‐Ruiz, Vanesa Lafarga, et al.. (2016). A Genome-wide CRISPR Screen Identifies CDC25A as a Determinant of Sensitivity to ATR Inhibitors. Molecular Cell. 62(2). 307–313. 152 indexed citations
5.
Ruiz, Sergio, Andrés J. López‐Contreras, Mathieu Gabut, et al.. (2015). Limiting replication stress during somatic cell reprogramming reduces genomic instability in induced pluripotent stem cells. Nature Communications. 6(1). 8036–8036. 74 indexed citations
6.
Menacho-Márquez, Mauricio, Ramón Garcı́a-Escudero, Virginia Ojeda, et al.. (2013). The Rho Exchange Factors Vav2 and Vav3 Favor Skin Tumor Initiation and Promotion by Engaging Extracellular Signaling Loops. PLoS Biology. 11(7). e1001615–e1001615. 51 indexed citations
7.
Ruiz, Sergio, Athanasia D. Panopoulos, Núria Montserrat, et al.. (2012). Generation of a Drug-inducible Reporter System to Study Cell Reprogramming in Human Cells. Journal of Biological Chemistry. 287(48). 40767–40778. 14 indexed citations
8.
Ruiz, Sergio, Athanasia D. Panopoulos, Karl‐Dimiter Bissig, et al.. (2010). A High Proliferation Rate Is Required for Cell Reprogramming and Maintenance of Human Embryonic Stem Cell Identity. Current Biology. 21(1). 45–52. 243 indexed citations
9.
Ruiz, Sergio, et al.. (2010). High-Efficient Generation of Induced Pluripotent Stem Cells from Human Astrocytes. PLoS ONE. 5(12). e15526–e15526. 53 indexed citations
10.
Ruiz, Sergio, Eugenio Santos, & Xosé R. Bustelo. (2009). The Use of Knockout Mice Reveals a Synergistic Role of the Vav1 and Rasgrf2 Gene Deficiencies in Lymphomagenesis and Metastasis. PLoS ONE. 4(12). e8229–e8229. 21 indexed citations
11.
Martínez‐Cruz, Ana Belén, Mirentxu Santos, M. Fernanda Lara, et al.. (2008). Spontaneous Squamous Cell Carcinoma Induced by the Somatic Inactivation of Retinoblastoma and Trp53 Tumor Suppressors. Cancer Research. 68(3). 683–692. 54 indexed citations
12.
Santos, Mirentxu, Sergio Ruiz, M. Fernanda Lara, et al.. (2008). Susceptibility of pRb‐deficient epidermis to chemical skin carcinogenesis is dependent on the p107 allele dosage. Molecular Carcinogenesis. 47(11). 815–821. 14 indexed citations
13.
Segrelles, Carmen, Jerry Lu, Mirentxu Santos, et al.. (2007). Deregulated Activity of Akt in Epithelial Basal Cells Induces Spontaneous Tumors and Heightened Sensitivity to Skin Carcinogenesis. Cancer Research. 67(22). 10879–10888. 78 indexed citations
14.
Ruiz, Sergio, Eugenio Santos, & Xosé R. Bustelo. (2007). RasGRF2, a Guanosine Nucleotide Exchange Factor for Ras GTPases, Participates in T-Cell Signaling Responses. Molecular and Cellular Biology. 27(23). 8127–8142. 46 indexed citations
15.
Ruiz, Sergio, Antonio Castro-Castro, & Xosé R. Bustelo. (2007). CD147 Inhibits the Nuclear Factor of Activated T-cells by Impairing Vav1 and Rac1 Downstream Signaling. Journal of Biological Chemistry. 283(9). 5554–5566. 36 indexed citations
16.
Ruiz, Sergio, Mirentxu Santos, & Jesús M. Paramio. (2006). Is the Loss of pRb Essential for the Mouse Skin. Cell Cycle. 5(6). 625–630. 19 indexed citations
17.
Segrelles, Carmen, M.A. Moral, M. Fernanda Lara, et al.. (2005). Molecular determinants of Akt-induced keratinocyte transformation. Oncogene. 25(8). 1174–1185. 74 indexed citations
18.
Ruiz, Sergio, Carmen Segrelles, Mirentxu Santos, M. Fernanda Lara, & Jesús M. Paramio. (2004). Functional link between retinoblastoma family of proteins and the Wnt signaling pathway in mouse epidermis. Developmental Dynamics. 230(3). 410–418. 18 indexed citations
19.
Segrelles, Carmen, et al.. (2002). Expression, localization, and activity of glycogen synthase kinase 3β during mouse skin tumorigenesis. Molecular Carcinogenesis. 35(4). 180–185. 58 indexed citations
20.
Segrelles, Carmen, Sergio Ruiz, Paloma Pérez, et al.. (2002). Functional roles of Akt signaling in mouse skin tumorigenesis. Oncogene. 21(1). 53–64. 157 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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