Seiichi Oyadomari

15.4k total citations · 6 hit papers
90 papers, 12.6k citations indexed

About

Seiichi Oyadomari is a scholar working on Cell Biology, Molecular Biology and Surgery. According to data from OpenAlex, Seiichi Oyadomari has authored 90 papers receiving a total of 12.6k indexed citations (citations by other indexed papers that have themselves been cited), including 55 papers in Cell Biology, 39 papers in Molecular Biology and 26 papers in Surgery. Recurrent topics in Seiichi Oyadomari's work include Endoplasmic Reticulum Stress and Disease (51 papers), Pancreatic function and diabetes (26 papers) and Autophagy in Disease and Therapy (14 papers). Seiichi Oyadomari is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (51 papers), Pancreatic function and diabetes (26 papers) and Autophagy in Disease and Therapy (14 papers). Seiichi Oyadomari collaborates with scholars based in Japan, United States and Sweden. Seiichi Oyadomari's co-authors include Masataka Mori, Tomomi Gotoh, Eiichi Araki, David Ron, Heather P. Harding, Yuhong Zhang, Shizuo Akira, Kohsuke Takeda, Isabel Novoa and Akio Koizumi and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Seiichi Oyadomari

89 papers receiving 12.4k citations

Hit Papers

Roles of CHOP/GADD153 in ... 2001 2026 2009 2017 2003 2013 2004 2002 2002 500 1000 1.5k 2.0k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Roles of CHOP/GADD153 in endoplasmic reticulum stress
    2003 2.4k citations Seiichi Oyadomari, Masataka Mori profile →
  2. Obesity-induced gut microbial metabolite promotes liver cancer through senescence secretome
    2013 1.7k citations Seiichi Oyadomari et al. profile →
  3. CHOP induces death by promoting protein synthesis and oxidation in the stressed endoplasmic reticulum
    2004 1.6k citations Seiichi Oyadomari et al. profile →
  4. Targeted disruption of the Chop gene delays endoplasmic reticulum stress–mediated diabetes
    2002 760 citations Seiichi Oyadomari, Akio Koizumi et al. Journal of Clinical Investigation profile →
  5. Targeted disruption of the Chop gene delays endoplasmic reticulum stress–mediated diabetes
    2002 682 citations Seiichi Oyadomari, Akio Koizumi et al. Journal of Clinical Investigation profile →
  6. Nitric oxide-induced apoptosis in pancreatic β cells is mediated by the endoplasmic reticulum stress pathway
    2001 509 citations Seiichi Oyadomari, Kohsuke Takeda et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Seiichi Oyadomari Japan 38 7.0k 5.9k 4.2k 2.8k 1.7k 90 12.6k
Fumihiko Urano United States 50 9.3k 1.3× 7.3k 1.2× 5.1k 1.2× 3.5k 1.3× 1.5k 0.9× 122 15.6k
Ann–Hwee Lee United States 36 8.1k 1.2× 5.6k 0.9× 5.3k 1.3× 2.9k 1.1× 2.0k 1.2× 50 14.4k
Neal N. Iwakoshi United States 31 5.4k 0.8× 3.6k 0.6× 3.4k 0.8× 2.3k 0.8× 1.3k 0.8× 47 10.3k
Tomomi Gotoh Japan 46 3.9k 0.6× 4.1k 0.7× 2.2k 0.5× 1.8k 0.7× 1.9k 1.1× 95 9.5k
Joungmok Kim South Korea 27 4.2k 0.6× 8.7k 1.5× 6.7k 1.6× 1.2k 0.4× 1.6k 0.9× 50 15.2k
Kazunori Imaizumi Japan 47 4.6k 0.7× 4.8k 0.8× 3.0k 0.7× 988 0.4× 1.4k 0.8× 135 9.4k
Cem Z. Görgün United States 22 3.5k 0.5× 5.2k 0.9× 5.3k 1.3× 2.4k 0.9× 4.3k 2.6× 34 12.7k
Kazutoshi Mori Japan 64 17.7k 2.5× 12.3k 2.1× 8.4k 2.0× 3.4k 1.2× 1.6k 0.9× 151 23.3k
Feroz R. Papa United States 30 5.1k 0.7× 4.2k 0.7× 2.8k 0.7× 1.3k 0.5× 527 0.3× 49 8.0k
Satoshi Waguri Japan 47 4.0k 0.6× 8.8k 1.5× 8.5k 2.0× 870 0.3× 2.3k 1.4× 134 16.7k

Countries citing papers authored by Seiichi Oyadomari

Since Specialization
Citations

This map shows the geographic impact of Seiichi Oyadomari's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Seiichi Oyadomari with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Seiichi Oyadomari more than expected).

Fields of papers citing papers by Seiichi Oyadomari

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Seiichi Oyadomari. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Seiichi Oyadomari. The network helps show where Seiichi Oyadomari may publish in the future.

Co-authorship network of co-authors of Seiichi Oyadomari

This figure shows the co-authorship network connecting the top 25 collaborators of Seiichi Oyadomari. A scholar is included among the top collaborators of Seiichi Oyadomari based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Seiichi Oyadomari. Seiichi Oyadomari is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fujimura, Masatake, et al.. (2024). Methylmercury-induced brain neuronal death in CHOP-knockout mice. The Journal of Toxicological Sciences. 49(2). 55–60. 2 indexed citations
2.
Miyake, Masato, Yoshimasa Hamada, Kazue Tsugawa, et al.. (2022). The multifaceted role of ATF4 in regulating glucose-stimulated insulin secretion. Biochemical and Biophysical Research Communications. 611. 165–171. 4 indexed citations
3.
Nguyen, Dinh Thi, Thuong Manh Le, Tsuyoshi Hattori, et al.. (2021). The ATF6β-calreticulin axis promotes neuronal survival under endoplasmic reticulum stress and excitotoxicity. Scientific Reports. 11(1). 13 indexed citations
4.
Saito, Yu, Tetsuya Ikemoto, Kazunori Tokuda, et al.. (2021). Effective three‐dimensional culture of hepatocyte‐like cells generated from human adipose‐derived mesenchymal stem cells. Journal of Hepato-Biliary-Pancreatic Sciences. 28(9). 705–715. 9 indexed citations
5.
Kitajima, Shojiro, Wendi Sun, Kian Leong Lee, et al.. (2021). A KDM6 inhibitor potently induces ATF4 and its target gene expression through HRI activation and by UTX inhibition. Scientific Reports. 11(1). 4538–4538. 6 indexed citations
6.
Li, Jinghe, Ryota Inoue, Yu Togashi, et al.. (2021). Imeglimin Ameliorates β-Cell Apoptosis by Modulating the Endoplasmic Reticulum Homeostasis Pathway. Diabetes. 71(3). 424–439. 51 indexed citations
7.
Kato, Hiroaki, Kohki Okabe, Masato Miyake, et al.. (2020). ER-resident sensor PERK is essential for mitochondrial thermogenesis in brown adipose tissue. Life Science Alliance. 3(3). e201900576–e201900576. 29 indexed citations
8.
Yasue, Akihiro, Kenichi Suzuki, Hirofumi Fujita, et al.. (2020). Fgf10-CRISPR mosaic mutants demonstrate the gene dose-related loss of the accessory lobe and decrease in the number of alveolar type 2 epithelial cells in mouse lung. PLoS ONE. 15(10). e0240333–e0240333. 2 indexed citations
9.
Kitakaze, Keisuke, Shusuke Taniuchi, Yoshimasa Hamada, et al.. (2019). Cell-based HTS identifies a chemical chaperone for preventing ER protein aggregation and proteotoxicity. eLife. 8. 22 indexed citations
10.
Mimura, Junsei, Shusuke Taniuchi, Kunio Kosaka, et al.. (2019). Concomitant Nrf2- and ATF4-Activation by Carnosic Acid Cooperatively Induces Expression of Cytoprotective Genes. International Journal of Molecular Sciences. 20(7). 1706–1706. 30 indexed citations
11.
Sato, Daiki, Kenji Teranishi, N. Shimomura, et al.. (2018). Activation of Endoplasmic Reticulum Stress Response by Applying of Nanosecond Pulsed Electric Fields for Medical Application. 456–460. 4 indexed citations
12.
Hashimoto, Osamu, Masayuki Funaba, Kazunari Sekiyama, et al.. (2018). Activin E Controls Energy Homeostasis in Both Brown and White Adipose Tissues as a Hepatokine. Cell Reports. 25(5). 1193–1203. 51 indexed citations
13.
Miyake, Masato, Shusuke Taniuchi, Miho Oyadomari, et al.. (2018). PERK-mediated translational control is required for collagen secretion in chondrocytes. Scientific Reports. 8(1). 773–773. 18 indexed citations
14.
15.
Inukai, Kouichi, Akihiko Kudo, Fumiyuki Nakagawa, et al.. (2014). Long-Term Low Carbohydrate Diet Leads to Deleterious Metabolic Manifestations in Diabetic Mice. PLoS ONE. 9(8). e104948–e104948. 8 indexed citations
16.
Morotomi‐Yano, Keiko, Seiichi Oyadomari, Hidenori Akiyama, & Ken‐ichi Yano. (2012). Nanosecond pulsed electric fields act as a novel cellular stress that induces translational suppression accompanied by eIF2α phosphorylation and 4E-BP1 dephosphorylation. Experimental Cell Research. 318(14). 1733–1744. 36 indexed citations
17.
Oyadomari, Seiichi, Akio Koizumi, Kiyoshi Takeda, et al.. (2002). Targeted disruption of the Chop gene delays endoplasmic reticulum stress–mediated diabetes. Journal of Clinical Investigation. 109(4). 525–532. 48 indexed citations
18.
Oyadomari, Seiichi, Akio Koizumi, Kohsuke Takeda, et al.. (2002). Targeted disruption of the Chop gene delays endoplasmic reticulum stress–mediated diabetes. Journal of Clinical Investigation. 109(4). 525–532. 760 indexed citations breakdown →
19.
Kawahara, Kohichi, Tomomi Gotoh, Seiichi Oyadomari, et al.. (2001). Nitric oxide inhibits the proliferation of murine microglial MG5 cells by a mechanism involving p21 but independent of p53 and cyclic guanosine monophosphate. Neuroscience Letters. 310(2-3). 89–92. 9 indexed citations
20.
Kawahara, Kohichi, Seiichi Oyadomari, Tomomi Gotoh, et al.. (2001). Induction of CHOP and apoptosis by nitric oxide in p53‐deficient microglial cells. FEBS Letters. 506(2). 135–139. 86 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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