Sarah A. Busch

3.2k total citations · 1 hit paper
15 papers, 2.5k citations indexed

About

Sarah A. Busch is a scholar working on Cellular and Molecular Neuroscience, Pathology and Forensic Medicine and Surgery. According to data from OpenAlex, Sarah A. Busch has authored 15 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Cellular and Molecular Neuroscience, 7 papers in Pathology and Forensic Medicine and 6 papers in Surgery. Recurrent topics in Sarah A. Busch's work include Nerve injury and regeneration (10 papers), Spinal Cord Injury Research (7 papers) and Neurogenesis and neuroplasticity mechanisms (6 papers). Sarah A. Busch is often cited by papers focused on Nerve injury and regeneration (10 papers), Spinal Cord Injury Research (7 papers) and Neurogenesis and neuroplasticity mechanisms (6 papers). Sarah A. Busch collaborates with scholars based in United States, United Kingdom and Australia. Sarah A. Busch's co-authors include Jerry Silver, Kevin P. Horn, Fernando X. Cuascut, Yingjie Shen, Alicia L. Hawthorne, Daniel J. Silver, Zhigang He, Alan P. Tenney, John G. Flanagan and Kai Liu and has published in prestigious journals such as Nature, Science and Journal of Neuroscience.

In The Last Decade

Sarah A. Busch

15 papers receiving 2.5k citations

Hit Papers

PTPσ Is a Receptor for Ch... 2009 2026 2014 2020 2009 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sarah A. Busch United States 13 1.6k 882 835 706 454 15 2.5k
Kevin P. Horn United States 17 1.3k 0.8× 794 0.9× 630 0.8× 588 0.8× 365 0.8× 28 2.3k
Jared H. Miller United States 4 2.0k 1.3× 860 1.0× 1.2k 1.5× 740 1.0× 386 0.9× 5 2.9k
Gavin Bennett United Kingdom 15 1.6k 1.0× 706 0.8× 658 0.8× 888 1.3× 543 1.2× 36 2.6k
Amanda Tran United States 12 1.2k 0.7× 934 1.1× 545 0.7× 680 1.0× 363 0.8× 16 2.2k
Catherine Doller United States 10 1.6k 1.0× 676 0.8× 840 1.0× 568 0.8× 330 0.7× 10 2.3k
Reena J. Popat United Kingdom 9 1.4k 0.9× 687 0.8× 649 0.8× 580 0.8× 515 1.1× 14 2.2k
Andrea Tedeschi United States 18 1.5k 1.0× 630 0.7× 882 1.1× 965 1.4× 254 0.6× 31 2.5k
Andrés Hurtado United States 21 1.2k 0.8× 684 0.8× 567 0.7× 510 0.7× 209 0.5× 29 2.2k
Robert J. McKeon United States 18 1.5k 0.9× 536 0.6× 636 0.8× 636 0.9× 524 1.2× 21 2.3k
Jae K. Lee United States 28 2.0k 1.2× 1.3k 1.5× 1.2k 1.4× 1.2k 1.7× 174 0.4× 62 3.7k

Countries citing papers authored by Sarah A. Busch

Since Specialization
Citations

This map shows the geographic impact of Sarah A. Busch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah A. Busch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah A. Busch more than expected).

Fields of papers citing papers by Sarah A. Busch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah A. Busch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah A. Busch. The network helps show where Sarah A. Busch may publish in the future.

Co-authorship network of co-authors of Sarah A. Busch

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah A. Busch. A scholar is included among the top collaborators of Sarah A. Busch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah A. Busch. Sarah A. Busch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Jones, James M., Marc A. DePaul, Bradley T. Lang, et al.. (2018). Multipotent Adult Progenitor Cells, but Not Tissue Inhibitor of Matrix Metalloproteinase-3, Increase Tissue Sparing and Reduce Urological Complications following Spinal Cord Injury. Journal of Neurotrauma. 36(9). 1416–1427. 3 indexed citations
2.
DePaul, Marc A., Bradley T. Lang, Rochelle Cutrone, et al.. (2015). Intravenous multipotent adult progenitor cell treatment decreases inflammation leading to functional recovery following spinal cord injury. Scientific Reports. 5(1). 16795–16795. 54 indexed citations
3.
Filous, Angela R., Amanda Tran, C. Howell, et al.. (2014). Entrapment via Synaptic-Like Connections between NG2 Proteoglycan+ Cells and Dystrophic Axons in the Lesion Plays a Role in Regeneration Failure after Spinal Cord Injury. Journal of Neuroscience. 34(49). 16369–16384. 102 indexed citations
4.
Lang, Bradley T., Jared M. Cregg, Marc A. DePaul, et al.. (2014). Modulation of the proteoglycan receptor PTPσ promotes recovery after spinal cord injury. Nature. 518(7539). 404–408. 349 indexed citations
5.
Reading, James L., Jennie H. M. Yang, Shereen Sabbah, et al.. (2013). Clinical-Grade Multipotent Adult Progenitor Cells Durably Control Pathogenic T Cell Responses in Human Models of Transplantation and Autoimmunity. The Journal of Immunology. 190(9). 4542–4552. 56 indexed citations
6.
Reading, James L., Shereen Sabbah, Sarah A. Busch, & Timothy Tree. (2012). Mesenchymal stromal cells as a means of controlling pathological T-cell responses in allogeneic islet transplantation. Current Opinion in Organ Transplantation. 18(1). 59–64. 13 indexed citations
7.
Busch, Sarah A., Jason A. Hamilton, Kevin P. Horn, et al.. (2011). Multipotent Adult Progenitor Cells Prevent Macrophage-Mediated Axonal Dieback and Promote Regrowth after Spinal Cord Injury. Journal of Neuroscience. 31(3). 944–953. 119 indexed citations
8.
Busch, Sarah A., et al.. (2011). Mesenchymal Stromal Cells as a Therapeutic Strategy to Support Islet Transplantation in Type 1 Diabetes Mellitus. PubMed. 2(2). 43–54. 6 indexed citations
9.
Busch, Sarah A., Kevin P. Horn, Fernando X. Cuascut, et al.. (2010). Adult NG2+ Cells Are Permissive to Neurite Outgrowth and Stabilize Sensory Axons during Macrophage-Induced Axonal Dieback after Spinal Cord Injury. Journal of Neuroscience. 30(1). 255–265. 136 indexed citations
10.
Shen, Yingjie, Alan P. Tenney, Sarah A. Busch, et al.. (2009). PTPσ Is a Receptor for Chondroitin Sulfate Proteoglycan, an Inhibitor of Neural Regeneration. Science. 326(5952). 592–596. 536 indexed citations breakdown →
11.
Busch, Sarah A., Kevin P. Horn, Daniel J. Silver, & Jerry Silver. (2009). Overcoming Macrophage-Mediated Axonal Dieback Following CNS Injury. Journal of Neuroscience. 29(32). 9967–9976. 186 indexed citations
12.
Horn, Kevin P., Sarah A. Busch, Alicia L. Hawthorne, Nico van Rooijen, & Jerry Silver. (2008). Another Barrier to Regeneration in the CNS: Activated Macrophages Induce Extensive Retraction of Dystrophic Axons through Direct Physical Interactions. Journal of Neuroscience. 28(38). 9330–9341. 259 indexed citations
13.
Busch, Sarah A. & Jerry Silver. (2007). The role of extracellular matrix in CNS regeneration. Current Opinion in Neurobiology. 17(1). 120–127. 368 indexed citations
14.
Udina, Esther, Matthew J. Furey, Sarah A. Busch, et al.. (2007). Electrical stimulation of intact peripheral sensory axons in rats promotes outgrowth of their central projections. Experimental Neurology. 210(1). 238–247. 144 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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