Sandrine Bétuing
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 5%
- Cardiology and Cardiovascular Medicine top 5%
- Physiology top 10%
- Surgery
- Co-authors
- Jocelyne CabocheEmmanuel RozeChristophe GraveleauE. Dale AbelDongfang ZhangRobert C. CookseyHeinrich TaegtmeyerMartin E. Young
- Topics
- Genetic Neurodegenerative Diseases (14 papers)Mitochondrial Function and Pathology (8 papers)Receptor Mechanisms and Signaling (6 papers)
- Partner nations
- FranceUnited StatesCanada
In The Last Decade
Sandrine Bétuing
27 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 94
- Molecular Biology 936
- Cellular and Molecular Neuroscience 576
- Cardiology and Cardiovascular Medicine 371
- Physiology 316
- Surgery 250
Countries citing papers authored by Sandrine Bétuing
This map shows the geographic impact of Sandrine Bétuing's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sandrine Bétuing with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sandrine Bétuing more than expected).
Fields of papers citing papers by Sandrine Bétuing
This network shows the impact of papers produced by Sandrine Bétuing. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sandrine Bétuing. The network helps show where Sandrine Bétuing may publish in the future.
Co-authorship network of co-authors of Sandrine Bétuing
This figure shows the co-authorship network connecting the top 25 collaborators of Sandrine Bétuing. A scholar is included among the top collaborators of Sandrine Bétuing based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sandrine Bétuing. Sandrine Bétuing is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 5 | |
| 3 | 42 | |
| 4 | 12 | |
| 5 | 17 | |
| 6 | 23 | |
| 7 | 147 | |
| 8 | 55 | |
| 9 | 210 | |
| 10 | 40 | |
| 11 | 116 | |
| 12 | 51 | |
| 13 | 5 | |
| 14 | 46 | |
| 15 | 278 | |
| 16 | 29 | |
| 17 | 314 | |
| 18 | 21 | |
| 19 | 23 | |
| 20 | 2 |
About Sandrine Bétuing
Sandrine Bétuing is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Immunology and Allergy, having authored 28 papers that have together received 1.7k indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (14 papers), Mitochondrial Function and Pathology (8 papers) and Receptor Mechanisms and Signaling (6 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (576 citations), Aging (31 citations) and Cardiology and Cardiovascular Medicine (371 citations). Sandrine Bétuing has collaborated with scholars based in France, United States and Canada. Frequent co-authors include Jocelyne Caboche, Emmanuel Roze, Christophe Graveleau, E. Dale Abel, Dongfang Zhang, Robert C. Cooksey, Heinrich Taegtmeyer, Martin E. Young, Darrell D. Belke and Donald A. McClain. Their work appears in journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.