Sally J. Stoehr

642 citations
21 papers · 541 indexed · h-index 13

Sally J. Stoehr

21 papers receiving 516 citations

Peers

Sally J. Stoehr
Comparison fields: 5 of 72
  • Cellular and Molecular Neuroscience 126
  • Physiology 31
  • Molecular Biology 412
  • Cell Biology 91
  • Immunology and Allergy 33
Replace E. Tsukuda with:
E. Tsukuda Japan
Yumiko Moritani Japan
S M Lohmann Germany
R Klinger Germany
Keith R. Westcott United States
Clare M.M. Haystead United States
S. OZAKI Japan
E.S. Payne United States
Christiane Nault Canada
Layla Saidi United States
Sally J. Stoehr relative to E. Tsukuda Japan E. Tsukuda's profile →
Citations per field
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Citations per year

Countries citing papers authored by Sally J. Stoehr

Since Specialization
Citations

This map shows the geographic impact of Sally J. Stoehr's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sally J. Stoehr with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sally J. Stoehr more than expected).

Fields of papers citing papers by Sally J. Stoehr

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sally J. Stoehr. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sally J. Stoehr. The network helps show where Sally J. Stoehr may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Sally J. Stoehr, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Sally J. Stoehr Line = papers co-authored together Sally J. Stoehr links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 200956
2
Structure-activity relationship at the leucine side chain in a series of N,N-dialkyldipeptidyl-amines as N-type calcium channel blockers.
20006
3 200016
4 199914
5 19997
6 199919
7 199935
8 19977
9 199592
10 199316
11 199110
12 199110
13 199019
14 19897
15 19884
16 198718
17 198676
18 198655
19 198628
20 198538

About Sally J. Stoehr

Sally J. Stoehr is a scholar working on Physiology, Immunology and Allergy and Molecular Biology, having authored 21 papers that have together received 541 indexed citations. Recurring topics across this work include Ion channel regulation and function (8 papers), Nicotinic Acetylcholine Receptors Study (4 papers), Pain Mechanisms and Treatments (3 papers), Neuroscience and Neuropharmacology Research (3 papers), Cellular transport and secretion (3 papers), Venomous Animal Envenomation and Studies (3 papers), Receptor Mechanisms and Signaling (3 papers) and Cholinesterase and Neurodegenerative Diseases (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (126 citations), Physiology (31 citations) and Molecular Biology (412 citations). Sally J. Stoehr has collaborated with scholars based in United States. Frequent co-authors include James E. Smolen, David Dooley, Bernard W. Agranoff, Aaron C. Gerlach, Neil A. Castle, Ronald W. Holz, Laurence A. Boxer, Michael H. Gelb, Scott Naisbitt and Farideh Ghomashchi. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Immunology and Biochemical Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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