S. Makri

679 total citations
8 papers, 156 citations indexed

About

S. Makri is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, S. Makri has authored 8 papers receiving a total of 156 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 2 papers in Cellular and Molecular Neuroscience and 2 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in S. Makri's work include Muscle Physiology and Disorders (6 papers), Signaling Pathways in Disease (2 papers) and Cardiomyopathy and Myosin Studies (2 papers). S. Makri is often cited by papers focused on Muscle Physiology and Disorders (6 papers), Signaling Pathways in Disease (2 papers) and Cardiomyopathy and Myosin Studies (2 papers). S. Makri collaborates with scholars based in France, Algeria and Italy. S. Makri's co-authors include Pascale Guicheney, Pascale Richard, Susana Quijano‐Roy, B. Estournet, Chahnez Triki, Hammadi Ayadi, Svetlana Maugenre, Tarik Hamadouche, Jean Muller and Valérie Allamand and has published in prestigious journals such as Neuromuscular Disorders, Brain and Development and Neuropediatrics.

In The Last Decade

S. Makri

8 papers receiving 156 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
S. Makri France	5	140	43	27	23	19	8	156
C. Bouchet-Séraphin France	6	194 1.4×	47 1.1×	29 1.1×	26 1.1×	34 1.8×	11	211
C. Cini United Kingdom	5	103 0.7×	38 0.9×	40 1.5×	10 0.4×	21 1.1×	5	148
K. Bushby United Kingdom	4	130 0.9×	17 0.4×	30 1.1×	23 1.0×	20 1.1×	12	142
Cristiane Araújo Martins Moreno Brazil	9	73 0.5×	23 0.5×	38 1.4×	14 0.6×	43 2.3×	28	143
Jonathan Volpatti Canada	7	139 1.0×	35 0.8×	43 1.6×	18 0.8×	90 4.7×	9	190
Silvia Baratta Italy	7	111 0.8×	111 2.6×	7 0.3×	10 0.4×	22 1.2×	11	246
Mai Thao Bui France	5	59 0.4×	23 0.5×	10 0.4×	8 0.3×	47 2.5×	10	108
Paweł Trzaskoma Poland	9	111 0.8×	17 0.4×	11 0.4×	11 0.5×	34 1.8×	13	183
Plavi Mittal United States	4	109 0.8×	31 0.7×	20 0.7×	21 0.9×	14 0.7×	7	126
Katherine R. Chao United States	8	120 0.9×	16 0.4×	43 1.6×	45 2.0×	14 0.7×	16	177

Countries citing papers authored by S. Makri

Since Specialization

Citations

This map shows the geographic impact of S. Makri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. Makri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. Makri more than expected).

Fields of papers citing papers by S. Makri

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S. Makri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. Makri. The network helps show where S. Makri may publish in the future.

Co-authorship network of co-authors of S. Makri

This figure shows the co-authorship network connecting the top 25 collaborators of S. Makri. A scholar is included among the top collaborators of S. Makri based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S. Makri. S. Makri is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Hamadouche, Tarik, Jean Muller, Nathalie Drouot, et al.. (2015). Molecular and clinical study of a cohort of 110 Algerian patients with autosomal recessive ataxia. BMC Medical Genetics. 16(1). 36–36. 23 indexed citations

2.

Benhassine, Traki, Sonia Nouioua, S. Makri, et al.. (2013). Intragenic deletion patterns of dystrophin gene in Duchenne and Becker muscular dystrophy patients from Algeria. Genes & Genomics. 36(1). 17–24. 1 indexed citations

3.

Makri, S., et al.. (2009). G.P.14.12 Neuromyotonia in childhood: Report of a case. Neuromuscular Disorders. 19(8-9). 647–647. 1 indexed citations

4.

Makri, S., Nigel F. Clarke, Pascale Richard, et al.. (2008). Germinal mosaicism for LMNA mimics autosomal recessive congenital muscular dystrophy. Neuromuscular Disorders. 19(1). 26–28. 15 indexed citations

5.

Allamand, Valérie, Corine Gartioux, Emmanuelle Lacène, et al.. (2007). C.O.3 Endoplasmic reticulum retention of COL6 chains in Ullrich congenital muscular dystrophy. Neuromuscular Disorders. 17(9-10). 833–833. 1 indexed citations

6.

Quijano‐Roy, Susana, Itxaso Martì, S. Makri, et al.. (2005). Brain MRI abnormalities in muscular dystrophy due to FKRP mutations. Brain and Development. 28(4). 232–242. 26 indexed citations

7.

Triki, Chahnez, Susana Quijano‐Roy, Pascale Richard, et al.. (2004). New FKRP mutations causing congenital muscular dystrophy associated with mental retardation and central nervous system abnormalities. Identification of a founder mutation in Tunisian families. Neurogenetics. 5(1). 27–34. 65 indexed citations

8.

Demir, Ercan, Ana Ferreiro, Patrizia Sabatelli, et al.. (2004). Collagen VI Status and Clinical Severity in Ullrich Congenital Muscular Dystrophy: Phenotype Analysis of 11 Families Linked to theCOL6Loci. Neuropediatrics. 35(2). 103–112. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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