Michel Kœnig

9.9k total citations · 3 hit papers
70 papers, 5.4k citations indexed

About

Michel Kœnig is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Genetics. According to data from OpenAlex, Michel Kœnig has authored 70 papers receiving a total of 5.4k indexed citations (citations by other indexed papers that have themselves been cited), including 62 papers in Cellular and Molecular Neuroscience, 56 papers in Molecular Biology and 11 papers in Genetics. Recurrent topics in Michel Kœnig's work include Genetic Neurodegenerative Diseases (57 papers), Mitochondrial Function and Pathology (44 papers) and DNA Repair Mechanisms (19 papers). Michel Kœnig is often cited by papers focused on Genetic Neurodegenerative Diseases (57 papers), Mitochondrial Function and Pathology (44 papers) and DNA Repair Mechanisms (19 papers). Michel Kœnig collaborates with scholars based in France, United States and Germany. Michel Kœnig's co-authors include Jean‐Louis Mandel, Mireille Cossée, Pierre Rustin, Alexandra Dürr, Victoria Campuzano, Christine Tranchant, Mathieu Anheim, Alexis Brice, Hélène Puccio and Arnold Münnich and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Circulation.

In The Last Decade

Michel Kœnig

69 papers receiving 5.3k citations

Hit Papers

Aconitase and mitochondrial iron–sulphur protein deficien... 1996 2026 2006 2016 1997 1996 2001 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Aconitase and mitochondrial iron–sulphur protein deficiency in Friedreich ataxia
    1997 786 citations Agnès Rötig, Pascale de Lonlay et al. Nature Genetics profile →
  2. Clinical and Genetic Abnormalities in Patients with Friedreich's Ataxia
    1996 769 citations Alexandra Dürr, Mireille Cossée et al. profile →
  3. Mouse models for Friedreich ataxia exhibit cardiomyopathy, sensory nerve defect and Fe-S enzyme deficiency followed by intramitochondrial iron deposits
    2001 588 citations Hélène Puccio, Mireille Cossée et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michel Kœnig France 34 4.4k 3.6k 1.0k 806 534 70 5.4k
Hélène Puccio France 39 3.9k 0.9× 2.4k 0.6× 525 0.5× 779 1.0× 268 0.5× 62 4.7k
Cinzia Gellera Italy 41 3.7k 0.8× 2.3k 0.6× 1.7k 1.7× 260 0.3× 479 0.9× 155 5.5k
Franco Taroni Italy 44 3.2k 0.7× 2.3k 0.6× 1.3k 1.3× 551 0.7× 367 0.7× 156 5.2k
Samir Belal Tunisia 23 1.1k 0.2× 1.0k 0.3× 416 0.4× 299 0.4× 131 0.2× 61 1.9k
Manabu Doyu Japan 42 4.1k 0.9× 3.4k 0.9× 2.5k 2.5× 576 0.7× 249 0.5× 107 6.8k
Pieter A. Bolhuis Netherlands 31 1.8k 0.4× 2.2k 0.6× 909 0.9× 489 0.6× 293 0.5× 63 4.0k
Mark W. Bêcher United States 23 2.3k 0.5× 1.9k 0.5× 1.9k 1.9× 630 0.8× 190 0.4× 44 4.7k
Masayuki Yokochi Japan 11 1.9k 0.4× 1.9k 0.5× 3.0k 3.0× 576 0.7× 325 0.6× 19 4.7k
Marijke Joosse Netherlands 14 1.4k 0.3× 1.1k 0.3× 1.8k 1.8× 347 0.4× 283 0.5× 16 3.9k
Géraldine Liot France 21 2.2k 0.5× 1.2k 0.3× 466 0.5× 443 0.5× 129 0.2× 27 3.2k

Countries citing papers authored by Michel Kœnig

Since Specialization
Citations

This map shows the geographic impact of Michel Kœnig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michel Kœnig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michel Kœnig more than expected).

Fields of papers citing papers by Michel Kœnig

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michel Kœnig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michel Kœnig. The network helps show where Michel Kœnig may publish in the future.

Co-authorship network of co-authors of Michel Kœnig

This figure shows the co-authorship network connecting the top 25 collaborators of Michel Kœnig. A scholar is included among the top collaborators of Michel Kœnig based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michel Kœnig. Michel Kœnig is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schneider, Anouck, Marie‐Claire Vincent, Christine Coubes, et al.. (2024). Confined placental mosaicism is a diagnostic pitfall in dystrophinopathies: a clinical report. European Journal of Human Genetics. 34(1). 161–164.
2.
Marelli, Cécilia, Mathieu Anheim, Christian Gény, et al.. (2017). Xeroderma pigmentosum complementation group F: A rare cause of cerebellar ataxia with chorea. Journal of the Neurological Sciences. 376. 198–201. 14 indexed citations
3.
Guissart, Claire, Nathalie Drouot, İbrahim Öncel, et al.. (2015). Genes for spinocerebellar ataxia with blindness and deafness (SCABD/SCAR3, MIM# 271250 and SCABD2). European Journal of Human Genetics. 24(8). 1154–1159. 15 indexed citations
4.
Salih, Mustafa A., Emeline Mundwiller, Arif O. Khan, et al.. (2013). Correction: New Findings in a Global Approach to Dissect the Whole Phenotype of PLA2G6 Gene Mutations. PLoS ONE. 8(11). 6 indexed citations
5.
Stefanatos, Rhoda, Berit Adam, H.J.M. Smeets, et al.. (2010). Variant CCG and GGC repeats within the CTG expansion dramatically modify mutational dynamics and likely contribute toward unusual symptoms in some myotonic dystrophy type 1 patients. Human Molecular Genetics. 19(8). 1399–1412. 120 indexed citations
6.
Anheim, Mathieu, Marie‐Céline Fleury, J. Franques, et al.. (2008). Clinical and Molecular Findings of Ataxia With Oculomotor Apraxia Type 2 in 4 Families. Archives of Neurology. 65(7). 958–62. 26 indexed citations
7.
Sarsero, Joseph P., Lingli Li, Timothy P. Holloway, et al.. (2004). Human BAC-mediated rescue of the Friedreich ataxia knockout mutation in transgenic mice. Mammalian Genome. 15(5). 370–382. 38 indexed citations
8.
Ristow, Michael, Hindrik Mulder, Tim J. Schulz, et al.. (2003). Frataxin deficiency in pancreatic islets causes diabetes due to loss of β cell mass. Journal of Clinical Investigation. 112(4). 527–534. 107 indexed citations
9.
Ristow, Michael, Hindrik Mulder, Tim J. Schulz, et al.. (2003). Frataxin deficiency in pancreatic islets causes diabetes due to loss of β cell mass. Journal of Clinical Investigation. 112(4). 527–534. 7 indexed citations
10.
Santos, Miguel M., Carlos J. Miranda, Joanne E. Levy, et al.. (2003). Iron metabolism in mice with partial frataxin deficiency. The Cerebellum. 2(2). 146–153. 5 indexed citations
11.
Bomont, Pascale, Christine Ioos, Cengiz Yalçınkaya, et al.. (2003). Identification of seven novel mutations in theGAN gene. Human Mutation. 21(4). 446–446. 42 indexed citations
12.
Barbot, Clara, Nobutada Tachi, Pascale Bomont, et al.. (2002). Autosomal recessive ataxias: a new gene - aprataxin - responsible for ataxia-ocular apraxia 1, and a new locus on 9q34. European Journal of Human Genetics. 10. 272–273. 3 indexed citations
13.
Tazir, Mériem, Jean-Michel Vallat, Pascale Bomont, et al.. (2002). Genetic heterogeneity in giant axonal neuropathy: an Algerian family not linked to chromosome 16q24.1. Neuromuscular Disorders. 12(9). 849–852. 21 indexed citations
14.
Benomar, Ali, Farid Meggouh, Ahmed Bouhouche, et al.. (2002). Clinical comparison between AVED patients with 744 del A mutation and Friedreich ataxia with GAA expansion in 15 Moroccan families. Journal of the Neurological Sciences. 198(1-2). 25–29. 40 indexed citations
15.
Barbot, Clara, Nobutada Tachi, Naoki Kozuka, et al.. (2001). Homozygosity Mapping of Portuguese and Japanese Forms of Ataxia-Oculomotor Apraxia to 9p13, and Evidence for Genetic Heterogeneity. The American Journal of Human Genetics. 68(2). 501–508. 58 indexed citations
16.
Delatycki, Martin B., Melanie A. Knight, Michel Kœnig, et al.. (1999). G130V, a common FRDA point mutation, appears to have arisen from a common founder. Human Genetics. 105(4). 343–346. 33 indexed citations
17.
Martinello, Flávia, et al.. (1998). Supplemental therapy in isolated vitamin E deficiency improves the peripheral neuropathy and prevents the progression of ataxia. Journal of the Neurological Sciences. 156(2). 177–179. 39 indexed citations
18.
Rötig, Agnès, Pascale de Lonlay, Dominique Chrétien, et al.. (1997). Aconitase and mitochondrial iron–sulphur protein deficiency in Friedreich ataxia. Nature Genetics. 17(2). 215–217. 786 indexed citations breakdown →
19.
Moltó, María Dolores, Juan J. Vílchez, Massimo Pandolfo, et al.. (1994). Mapping of Friedreich's AtaxiaLocus by Identification ofRecombination Events in PatientsHomozygous by Descent. European Journal of Human Genetics. 2(4). 291–299. 8 indexed citations
20.
Fujita, Ricardo, André Hanauer, Anne Vincent, Jean‐Louis Mandel, & Michel Kœnig. (1991). Physical mapping of two loci (D9S5 and D9S15) tightly linked to Friedreich ataxia locus (FRDA) and identification of nearby CpG islands by pulse-field gel electrophoresis. Genomics. 10(4). 915–920. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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