Rute A. Tomaz

1.5k total citations
14 papers, 466 citations indexed

About

Rute A. Tomaz is a scholar working on Molecular Biology, Genetics and Biomedical Engineering. According to data from OpenAlex, Rute A. Tomaz has authored 14 papers receiving a total of 466 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 4 papers in Genetics and 3 papers in Biomedical Engineering. Recurrent topics in Rute A. Tomaz's work include Pluripotent Stem Cells Research (6 papers), CRISPR and Genetic Engineering (5 papers) and Epigenetics and DNA Methylation (4 papers). Rute A. Tomaz is often cited by papers focused on Pluripotent Stem Cells Research (6 papers), CRISPR and Genetic Engineering (5 papers) and Epigenetics and DNA Methylation (4 papers). Rute A. Tomaz collaborates with scholars based in United Kingdom, Portugal and France. Rute A. Tomaz's co-authors include Branca Cavaco, Valeriano Leite, Ludovic Vallier, Joana G. Silva, Maria João Bugalho, Véronique Azuara, Rodrigo A. Grandy, Fabrice Lavial, Anil Chandrashekran and António E. Pinto and has published in prestigious journals such as Nature Communications, Genes & Development and Development.

In The Last Decade

Rute A. Tomaz

14 papers receiving 466 citations

Peers

Rute A. Tomaz

GENET

EDM

HEPAT

SURGE

Laura J. Zitur United States

Peter Zentis Germany

I. Koornneef Netherlands

Mats Grände Sweden

Bae Jun Oh South Korea

Silvia Álvarez-Cubela United States

Saravanan Ramamoorthy United States

Tatsuo Shimada Japan

Muhamad Ashraf Rostam Australia

Mona Malek Mohammadi Germany

Laura J. Zitur United States

Rute A. Tomaz

298 ×1.0

41 ×0.3

GENET

9 ×0.1

EDM

25 ×0.3

HEPAT

118 ×1.7

SURGE

Citations per year, relative to Rute A. Tomaz Rute A. Tomaz (= 1×) peers Laura J. Zitur

Countries citing papers authored by Rute A. Tomaz

Since Specialization

Citations

This map shows the geographic impact of Rute A. Tomaz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rute A. Tomaz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rute A. Tomaz more than expected).

Fields of papers citing papers by Rute A. Tomaz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rute A. Tomaz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rute A. Tomaz. The network helps show where Rute A. Tomaz may publish in the future.

Co-authorship network of co-authors of Rute A. Tomaz

This figure shows the co-authorship network connecting the top 25 collaborators of Rute A. Tomaz. A scholar is included among the top collaborators of Rute A. Tomaz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rute A. Tomaz. Rute A. Tomaz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Tomaz, Rute A., Sandra Petrus-Reurer, Carola Maria Morell, et al.. (2022). Generation of functional hepatocytes by forward programming with nuclear receptors. eLife. 11. 15 indexed citations

Ortmann, Daniel, Stephanie Brown, Anne Czechanski, et al.. (2020). Naive Pluripotent Stem Cells Exhibit Phenotypic Variability that Is Driven by Genetic Variation. Cell stem cell. 27(3). 470–481.e6. 31 indexed citations

Bell, Emma, Edward Curry, Wout Megchelenbrink, et al.. (2020). Dynamic CpG methylation delineates subregions within super-enhancers selectively decommissioned at the exit from naive pluripotency. Nature Communications. 11(1). 1112–1112. 24 indexed citations

Rashidi, Hassan, N. Thin Luu, Salamah Mohammad Alwahsh, et al.. (2018). 3D human liver tissue from pluripotent stem cells displays stable phenotype in vitro and supports compromised liver function in vivo. Archives of Toxicology. 92(10). 3117–3129. 90 indexed citations

Yiangou, Loukia, Rodrigo A. Grandy, Carola Maria Morell, et al.. (2018). Method to Synchronize Cell Cycle of Human Pluripotent Stem Cells without Affecting Their Fundamental Characteristics. Stem Cell Reports. 12(1). 165–179. 35 indexed citations

Grandy, Rodrigo A., Rute A. Tomaz, & Ludovic Vallier. (2018). Modeling Disease with Human Inducible Pluripotent Stem Cells. Annual Review of Pathology Mechanisms of Disease. 14(1). 449–468. 16 indexed citations

Tomaz, Rute A., Jennifer Harman, Lauriane Fritsch, et al.. (2017). Jmjd2c facilitates the assembly of essential enhancer-protein complexes at the onset of embryonic stem cell differentiation. Development. 144(4). 567–579. 19 indexed citations

Silva, Joana G., Rute A. Tomaz, António E. Pinto, et al.. (2014). Identification of a novel germline FOXE1 variant in patients with familial non-medullary thyroid carcinoma (FNMTC). Endocrine. 49(1). 204–214. 51 indexed citations

Percharde, Michelle, Fabrice Lavial, Jia-Hui Ng, et al.. (2012). Ncoa3 functions as an essential Esrrb coactivator to sustain embryonic stem cell self-renewal and reprogramming. Genes & Development. 26(20). 2286–2298. 73 indexed citations

10.

Lavial, Fabrice, Sylvain Bessonnard, Yusuke Ohnishi, et al.. (2012). Bmi1 facilitates primitive endoderm formation by stabilizing Gata6 during early mouse development. Genes & Development. 26(13). 1445–1458. 20 indexed citations

11.

Tomaz, Rute A., Inês Sousa, Joana G. Silva, et al.. (2012). FOXE1 polymorphisms are associated with familial and sporadic nonmedullary thyroid cancer susceptibility. Clinical Endocrinology. 77(6). 926–933. 45 indexed citations

12.

Domingues, Rita, et al.. (2011). Identification of the first germline HRPT2 whole‐gene deletion in a patient with primary hyperparathyroidism. Clinical Endocrinology. 76(1). 33–38. 31 indexed citations

13.

Tomaz, Rute A., Branca Cavaco, & Valeriano Leite. (2010). Differential Methylation as a Cause of Allele Dropout at the Imprinted GNAS Locus. Genetic Testing and Molecular Biomarkers. 14(4). 455–460. 4 indexed citations

14.

Cavaco, Branca, Rute A. Tomaz, Fernando Fonseca, et al.. (2010). Clinical and genetic characterization of Portuguese patients with pseudohypoparathyroidism type Ib. Endocrine. 37(3). 408–414. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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