Fabrice Lavial

1.6k total citations
21 papers, 1.0k citations indexed

About

Fabrice Lavial is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Fabrice Lavial has authored 21 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 9 papers in Genetics and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Fabrice Lavial's work include Pluripotent Stem Cells Research (13 papers), CRISPR and Genetic Engineering (12 papers) and Animal Genetics and Reproduction (9 papers). Fabrice Lavial is often cited by papers focused on Pluripotent Stem Cells Research (13 papers), CRISPR and Genetic Engineering (12 papers) and Animal Genetics and Reproduction (9 papers). Fabrice Lavial collaborates with scholars based in France, United Kingdom and Spain. Fabrice Lavial's co-authors include Bertrand Pain, Jacques Samarut, Hervé Acloque, Nathalie Allioli, Philippe Durand, Marie‐Hélène Perrard, Frédéric Chalmel, Véronique Azuara, Alain Puisieux and Anne‐Pierre Morel and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Genes & Development.

In The Last Decade

Fabrice Lavial

21 papers receiving 991 citations

Peers

Fabrice Lavial
Amy N. Shore United States
Deborah Hughes United Kingdom
Kibibi Ganz United States
William S. Einhorn United States
Clare Wise United Kingdom
Jikui Guan Sweden
Wendy Roeb United States
Samar P. Shah United States
Amy N. Shore United States
Fabrice Lavial
Citations per year, relative to Fabrice Lavial Fabrice Lavial (= 1×) peers Amy N. Shore

Countries citing papers authored by Fabrice Lavial

Since Specialization
Citations

This map shows the geographic impact of Fabrice Lavial's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fabrice Lavial with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fabrice Lavial more than expected).

Fields of papers citing papers by Fabrice Lavial

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fabrice Lavial. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fabrice Lavial. The network helps show where Fabrice Lavial may publish in the future.

Co-authorship network of co-authors of Fabrice Lavial

This figure shows the co-authorship network connecting the top 25 collaborators of Fabrice Lavial. A scholar is included among the top collaborators of Fabrice Lavial based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fabrice Lavial. Fabrice Lavial is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lavial, Fabrice, et al.. (2023). Cellular plasticity in reprogramming, rejuvenation and tumorigenesis: a pioneer TF perspective. Trends in Cell Biology. 34(3). 255–267. 20 indexed citations
2.
Durand, Sébastien, Juliana Blin, Sylvie Martel, et al.. (2023). RSL24D1 sustains steady-state ribosome biogenesis and pluripotency translational programs in embryonic stem cells. Nature Communications. 14(1). 356–356. 11 indexed citations
3.
Lavial, Fabrice, et al.. (2023). Molecular versatility during pluripotency progression. Nature Communications. 14(1). 68–68. 11 indexed citations
4.
Puisieux, Alain, Roxane M. Pommier, Anne‐Pierre Morel, & Fabrice Lavial. (2018). Cellular Pliancy and the Multistep Process of Tumorigenesis. Cancer Cell. 33(2). 164–172. 69 indexed citations
5.
Mehlen, Patrick & Fabrice Lavial. (2016). Le facteur nétrine-1 régule la reprogrammation cellulaire vers l’état pluripotent. médecine/sciences. 32(3). 241–244. 1 indexed citations
6.
Ozmadenci, Duygu, Olivier Féraud, Suzy Markossian, et al.. (2015). Netrin-1 regulates somatic cell reprogramming and pluripotency maintenance. Nature Communications. 6(1). 7398–7398. 34 indexed citations
7.
Gibert, Benjamin, Céline Delloye‐Bourgeois, Charles‐Henry Gattolliat, et al.. (2014). Regulation by miR181 Family of the Dependence Receptor CDON Tumor Suppressive Activity in Neuroblastoma. JNCI Journal of the National Cancer Institute. 106(11). 25 indexed citations
8.
Ichim, Gabriel, Marie‐May Coissieux, Marie-Pierre Lambert, et al.. (2013). Dependence receptor TrkC is a putative colon cancer tumor suppressor. Proceedings of the National Academy of Sciences. 110(8). 3017–3022. 50 indexed citations
9.
Percharde, Michelle, Fabrice Lavial, Jia-Hui Ng, et al.. (2012). Ncoa3 functions as an essential Esrrb coactivator to sustain embryonic stem cell self-renewal and reprogramming. Genes & Development. 26(20). 2286–2298. 73 indexed citations
10.
Lavial, Fabrice, Sylvain Bessonnard, Yusuke Ohnishi, et al.. (2012). Bmi1 facilitates primitive endoderm formation by stabilizing Gata6 during early mouse development. Genes & Development. 26(13). 1445–1458. 20 indexed citations
11.
Acloque, Hervé, Fabrice Lavial, & Bertrand Pain. (2012). Astacin‐like metallo‐endopeptidase is dynamically expressed in embryonic stem cells and embryonic epithelium during morphogenesis. Developmental Dynamics. 241(3). 574–582. 10 indexed citations
12.
Voisin, Sophie, et al.. (2012). Pluripotent genes in avian stem cells. Development Growth & Differentiation. 55(1). 41–51. 17 indexed citations
13.
Theunissen, Thorold W., Yael Costa, Aliaksandra Radzisheuskaya, et al.. (2011). Reprogramming capacity of Nanog is functionally conserved in vertebrates and resides in a unique homeodomain. Journal of Cell Science. 124(22). e1–e1. 2 indexed citations
14.
Theunissen, Thorold W., Yael Costa, Aliaksandra Radzisheuskaya, et al.. (2011). Reprogramming capacity of Nanog is functionally conserved in vertebrates and resides in a unique homeodomain. Development. 138(22). 4853–4865. 62 indexed citations
15.
Alder, Olivia, Fabrice Lavial, Emily Brookes, et al.. (2010). Ring1B and Suv39h1 delineate distinct chromatin states at bivalent genes during early mouse lineage commitment. Development. 137(15). 2483–2492. 92 indexed citations
16.
Lavial, Fabrice & Bertrand Pain. (2010). Chicken embryonic stem cells as a non‐mammalian embryonic stem cell model. Development Growth & Differentiation. 52(1). 101–114. 32 indexed citations
17.
Allioli, Nathalie, Fabrice Lavial, Frédéric Chalmel, et al.. (2010). Role of miR-34c microRNA in the late steps of spermatogenesis. RNA. 16(4). 720–731. 242 indexed citations
18.
Lavial, Fabrice, et al.. (2009). Ectopic expression of Cvh (Chicken Vasa homologue) mediates the reprogramming of chicken embryonic stem cells to a germ cell fate. Developmental Biology. 330(1). 73–82. 58 indexed citations
19.
Lavial, Fabrice, Hervé Acloque, Guillaume Montillet, et al.. (2008). Molecular control of pluripotency and germ line competency in chicken embryonic stem cells. Cell Research. 18(S1). S106–S106. 1 indexed citations
20.
Lavial, Fabrice, Hervé Acloque, Federica Bertocchini, et al.. (2007). The Oct4 homologue PouV and Nanog regulate pluripotency in chicken embryonic stem cells. Development. 134(19). 3549–3563. 173 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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