William G. Bernard

1.1k total citations
15 papers, 619 citations indexed

About

William G. Bernard is a scholar working on Molecular Biology, Surgery and Pulmonary and Respiratory Medicine. According to data from OpenAlex, William G. Bernard has authored 15 papers receiving a total of 619 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 5 papers in Surgery and 2 papers in Pulmonary and Respiratory Medicine. Recurrent topics in William G. Bernard's work include Congenital heart defects research (7 papers), Tissue Engineering and Regenerative Medicine (4 papers) and Pluripotent Stem Cells Research (3 papers). William G. Bernard is often cited by papers focused on Congenital heart defects research (7 papers), Tissue Engineering and Regenerative Medicine (4 papers) and Pluripotent Stem Cells Research (3 papers). William G. Bernard collaborates with scholars based in United Kingdom, United States and Australia. William G. Bernard's co-authors include Sanjay Sinha, Felipe Serrano, Alessandra Granata, Laure Gambardella, Dharini Iyer, Priya Sastry, Alessandro Bertero, Johannes Bargehr, Fotios Sampaziotis and Martin R. Bennett and has published in prestigious journals such as Journal of Biological Chemistry, Nature Genetics and Nature Biotechnology.

In The Last Decade

William G. Bernard

15 papers receiving 614 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
William G. Bernard United Kingdom 12 432 220 91 87 81 15 619
Dharini Iyer United States 10 363 0.8× 186 0.8× 64 0.7× 56 0.6× 56 0.7× 13 539
Ethan Radzinsky United States 5 598 1.4× 267 1.2× 30 0.3× 42 0.5× 43 0.5× 7 700
Laureen Jacquet United Kingdom 9 363 0.8× 97 0.4× 40 0.4× 46 0.5× 103 1.3× 13 527
Sonia Stefanovic France 16 617 1.4× 142 0.6× 112 1.2× 61 0.7× 34 0.4× 26 725
Catherine Paulissen Belgium 8 868 2.0× 251 1.1× 77 0.8× 45 0.5× 28 0.3× 9 944
Esra Çağavi Türkiye 10 428 1.0× 127 0.6× 31 0.3× 26 0.3× 80 1.0× 21 666
Shah R. Ali United States 6 448 1.0× 243 1.1× 29 0.3× 57 0.7× 30 0.4× 9 685
Sandra Menke Germany 7 600 1.4× 380 1.7× 23 0.3× 35 0.4× 117 1.4× 7 725
Pyry I. Toivanen Finland 11 304 0.7× 90 0.4× 57 0.6× 31 0.4× 28 0.3× 17 477
Chikai Zhou Sweden 13 547 1.3× 98 0.4× 70 0.8× 31 0.4× 37 0.5× 18 742

Countries citing papers authored by William G. Bernard

Since Specialization
Citations

This map shows the geographic impact of William G. Bernard's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William G. Bernard with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William G. Bernard more than expected).

Fields of papers citing papers by William G. Bernard

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William G. Bernard. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William G. Bernard. The network helps show where William G. Bernard may publish in the future.

Co-authorship network of co-authors of William G. Bernard

This figure shows the co-authorship network connecting the top 25 collaborators of William G. Bernard. A scholar is included among the top collaborators of William G. Bernard based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William G. Bernard. William G. Bernard is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Ong, Lay Ping, Johannes Bargehr, Vincent Knight-Schrijver, et al.. (2023). Epicardially secreted fibronectin drives cardiomyocyte maturation in 3D-engineered heart tissues. Stem Cell Reports. 18(4). 936–951. 13 indexed citations
2.
Macrae, Robyn, Maria Colzani, Thomas Williams, et al.. (2022). Inducible apelin receptor knockdown reduces differentiation efficiency and contractility of hESC-derived cardiomyocytes. Cardiovascular Research. 119(2). 587–598. 8 indexed citations
3.
Gambardella, Laure, Victoria Moignard, William G. Bernard, et al.. (2020). BNC1: A master regulator of human epicardial heterogeneity and function. Journal of Molecular and Cellular Cardiology. 140. 30–31. 1 indexed citations
4.
Bargehr, Johannes, Lay Ping Ong, Maria Colzani, et al.. (2019). Epicardial cells derived from human embryonic stem cells augment cardiomyocyte-driven heart regeneration. Nature Biotechnology. 37(8). 895–906. 142 indexed citations
5.
Gambardella, Laure, Victoria Moignard, Simon Andrews, et al.. (2019). BNC1 regulates cell heterogeneity in human pluripotent stem cell-derived epicardium. Development. 146(24). 26 indexed citations
6.
Serrano, Felipe, William G. Bernard, Alessandra Granata, et al.. (2018). A Novel Human Pluripotent Stem Cell-Derived Neural Crest Model of Treacher Collins Syndrome Shows Defects in Cell Death and Migration. Stem Cells and Development. 28(2). 81–100. 28 indexed citations
7.
Yiangou, Loukia, Rodrigo A. Grandy, Carola Maria Morell, et al.. (2018). Method to Synchronize Cell Cycle of Human Pluripotent Stem Cells without Affecting Their Fundamental Characteristics. Stem Cell Reports. 12(1). 165–179. 35 indexed citations
8.
Bargehr, Johannes, Peter Hofsteen, Shiv Bhandari, et al.. (2017). 5728Human embryonic stem cell derived epicardial cells advance cardiomyocyte-based heart regeneration. European Heart Journal. 38(suppl_1). 2 indexed citations
9.
Granata, Alessandra, et al.. (2016). An iPSC-derived vascular model of Marfan syndrome identifies key mediators of smooth muscle cell death. Nature Genetics. 49(1). 97–109. 144 indexed citations
10.
Bargehr, Johannes, Christine Cheung, William G. Bernard, et al.. (2016). Embryological Origin of Human Smooth Muscle Cells Influences Their Ability to Support Endothelial Network Formation. Stem Cells Translational Medicine. 5(7). 946–959. 23 indexed citations
11.
Bertero, Alessandro, Matthias Pawlowski, Daniel Ortmann, et al.. (2016). Optimized inducible shRNA and CRISPR/Cas9 platforms for in vitro studies of human development using hPSCs. Development. 143(23). 4405–4418. 57 indexed citations
12.
Iyer, Dharini, Laure Gambardella, William G. Bernard, et al.. (2016). Robust derivation of epicardium and its differentiated smooth muscle cell progeny from human pluripotent stem cells. Development. 143(5). 904–904. 46 indexed citations
13.
Granata, Alessandra, William G. Bernard, Ning Zhao, et al.. (2014). Temporal and Embryonic Lineage-Dependent Regulation of Human Vascular SMC Development by NOTCH3. Stem Cells and Development. 24(7). 846–856. 13 indexed citations
14.
Pantazaka, Evangelia, Emily Taylor, William G. Bernard, & Colin W. Taylor. (2013). Ca2+ signals evoked by histamine H1 receptors are attenuated by activation of prostaglandin EP2 and EP4 receptors in human aortic smooth muscle cells. British Journal of Pharmacology. 169(7). 1624–1634. 16 indexed citations
15.
Tulloch, L.B., J. W. Howie, Krzysztof Wypijewski, et al.. (2011). The Inhibitory Effect of Phospholemman on the Sodium Pump Requires Its Palmitoylation. Journal of Biological Chemistry. 286(41). 36020–36031. 65 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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