Roni Scherzer‐Attali

545 total citations
9 papers, 465 citations indexed

About

Roni Scherzer‐Attali is a scholar working on Physiology, Pharmacology and Molecular Biology. According to data from OpenAlex, Roni Scherzer‐Attali has authored 9 papers receiving a total of 465 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Physiology, 5 papers in Pharmacology and 4 papers in Molecular Biology. Recurrent topics in Roni Scherzer‐Attali's work include Alzheimer's disease research and treatments (7 papers), Cholinesterase and Neurodegenerative Diseases (5 papers) and Computational Drug Discovery Methods (3 papers). Roni Scherzer‐Attali is often cited by papers focused on Alzheimer's disease research and treatments (7 papers), Cholinesterase and Neurodegenerative Diseases (5 papers) and Computational Drug Discovery Methods (3 papers). Roni Scherzer‐Attali collaborates with scholars based in Israel, Switzerland and United States. Roni Scherzer‐Attali's co-authors include Ehud Gazit, Daniel Segal, Anat Frydman‐Marom, Dan Frenkel, Dorit Farfara, Tali Benromano, Aviad Levin, Ronit Shaltiel‐Karyo, Robert Vassar and Deborah E. Shalev and has published in prestigious journals such as Angewandte Chemie International Edition, PLoS ONE and The Journal of Physical Chemistry B.

In The Last Decade

Roni Scherzer‐Attali

9 papers receiving 461 citations

Peers

Countries citing papers authored by Roni Scherzer‐Attali

Since Specialization

Citations

This map shows the geographic impact of Roni Scherzer‐Attali's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Roni Scherzer‐Attali with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Roni Scherzer‐Attali more than expected).

Fields of papers citing papers by Roni Scherzer‐Attali

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Roni Scherzer‐Attali. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Roni Scherzer‐Attali. The network helps show where Roni Scherzer‐Attali may publish in the future.

Co-authorship network of co-authors of Roni Scherzer‐Attali

This figure shows the co-authorship network connecting the top 25 collaborators of Roni Scherzer‐Attali. A scholar is included among the top collaborators of Roni Scherzer‐Attali based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Roni Scherzer‐Attali. Roni Scherzer‐Attali is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

#	Work	Indexed citations
1	Cl-NQTrp Alleviates Tauopathy Symptoms in a Model Organism through the Inhibition of Tau Aggregation-Engendered Toxicity 2016 ·Neurodegenerative Diseases ·Moran Frenkel‐Pinter, (unknown), Roni Scherzer‐Attali, (unknown), Idan Alyagor, (unknown), Ehud Gazit, Daniel Segal	22
2	Naphthoquinone-Tryptophan Hybrid Inhibits Aggregation of the Tau-Derived Peptide PHF6 and Reduces Neurotoxicity 2016 ·Journal of Alzheimer s Disease ·Moran Frenkel‐Pinter, (unknown), Roni Scherzer‐Attali, (unknown), Idan Alyagor, (unknown), Ehud Gazit, Daniel Segal	40
3	Monitoring and Targeting the Initial Dimerization Stage of Amyloid Self‐Assembly 2014 ·Angewandte Chemie International Edition ·Yaron Bram, Ayala Lampel, Ronit Shaltiel‐Karyo, (unknown), Roni Scherzer‐Attali, Daniel Segal, Ehud Gazit	20
4	Generic inhibition of amyloidogenic proteins by two naphthoquinone–tryptophan hybrid molecules 2012 ·Proteins Structure Function and Bioinformatics ·Roni Scherzer‐Attali, Ronit Shaltiel‐Karyo, (unknown), Daniel Segal, Ehud Gazit	46
5	Methylations of Tryptophan-Modified Naphthoquinone Affect Its Inhibitory Potential toward Aβ Aggregation 2012 ·The Journal of Physical Chemistry B ·Roni Scherzer‐Attali, Marino Convertino, Riccardo Pellarin, Ehud Gazit, Daniel Segal, Amedeo Caflisch	15
6	Naphthoquinone-tyrptophan reduces neurotoxic Aβ56 levels and improves cognition in Alzheimer's disease animal model 2012 ·Neurobiology of Disease* ·Roni Scherzer‐Attali, Dorit Farfara, Itzik Cooper, Aviad Levin, Tali Benromano, Dorit Trudler, (unknown), Ronit Shaltiel‐Karyo, Deborah E. Shalev, (unknown), Ehud Gazit, Daniel Segal, Dan Frenkel	33
7	Orally Administrated Cinnamon Extract Reduces β-Amyloid Oligomerization and Corrects Cognitive Impairment in Alzheimer's Disease Animal Models 2011 ·PLoS ONE ·Anat Frydman‐Marom, Aviad Levin, Dorit Farfara, Tali Benromano, Roni Scherzer‐Attali, (unknown), Robert Vassar, Daniel Segal, Ehud Gazit, Dan Frenkel, Michael Ovadia	158
8	Quantitative structure–activity relationship analysis of β-amyloid aggregation inhibitors 2010 ·Journal of Computer-Aided Molecular Design ·Shiri Stempler, Michal Levy‐Sakin, Anat Frydman‐Marom, Yaniv Amir, Roni Scherzer‐Attali, Ludmila Buzhansky, Ehud Gazit, Hanoch Senderowitz	5
9	Complete Phenotypic Recovery of an Alzheimer's Disease Model by a Quinone-Tryptophan Hybrid Aggregation Inhibitor 2010 ·PLoS ONE ·Roni Scherzer‐Attali, Riccardo Pellarin, Marino Convertino, Anat Frydman‐Marom, (unknown), (unknown), Michal Levy‐Sakin, Deborah E. Shalev, Amedeo Caflisch, Ehud Gazit, Daniel Segal	126

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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