Ronald Gallagher

725 total citations
11 papers, 553 citations indexed

About

Ronald Gallagher is a scholar working on Molecular Biology, Surgery and Immunology. According to data from OpenAlex, Ronald Gallagher has authored 11 papers receiving a total of 553 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 2 papers in Surgery and 2 papers in Immunology. Recurrent topics in Ronald Gallagher's work include Pluripotent Stem Cells Research (2 papers), CRISPR and Genetic Engineering (2 papers) and Wnt/β-catenin signaling in development and cancer (2 papers). Ronald Gallagher is often cited by papers focused on Pluripotent Stem Cells Research (2 papers), CRISPR and Genetic Engineering (2 papers) and Wnt/β-catenin signaling in development and cancer (2 papers). Ronald Gallagher collaborates with scholars based in United Kingdom, United States and Netherlands. Ronald Gallagher's co-authors include Kay Samuel, James A. Ross, A Pryde, John Terrace, John P. Iredale, Judy Fletcher, Davina Wojtacha, Philip N. Newsome, David C. Hay and Stuart J. Forbes and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Oncogene and Journal of Virology.

In The Last Decade

Ronald Gallagher

11 papers receiving 542 citations

Peers

Ronald Gallagher

SURGE

HEPAT

PHYSI

Mitsuru Inamura Japan

Choongseong Han South Korea

Keiko Shimizu‐Saito Japan

Uyunbilig Borjigin China

Norihisa Ichinohe Japan

Suomi M.G. Fouraschen Netherlands

Antonio Lo Nigro Italy

Anuradha Menthena United States

T. Kuhara Japan

Adam Khalil United States

Mitsuru Inamura Japan

Ronald Gallagher

492 ×1.2

265 ×1.4

SURGE

267 ×1.6

HEPAT

141 ×1.3

50 ×0.9

PHYSI

Citations per year, relative to Ronald Gallagher Ronald Gallagher (= 1×) peers Mitsuru Inamura

Countries citing papers authored by Ronald Gallagher

Since Specialization

Citations

This map shows the geographic impact of Ronald Gallagher's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ronald Gallagher with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ronald Gallagher more than expected).

Fields of papers citing papers by Ronald Gallagher

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ronald Gallagher. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ronald Gallagher. The network helps show where Ronald Gallagher may publish in the future.

Co-authorship network of co-authors of Ronald Gallagher

This figure shows the co-authorship network connecting the top 25 collaborators of Ronald Gallagher. A scholar is included among the top collaborators of Ronald Gallagher based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ronald Gallagher. Ronald Gallagher is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

Liu, Jing, Joanna Brzeszczyńska, Kay Samuel, et al.. (2015). Efficient episomal reprogramming of blood mononuclear cells and differentiation to hepatocytes with functional drug metabolism. Experimental Cell Research. 338(2). 203–213. 24 indexed citations

Samuel, Kay, et al.. (2014). Use of IL3 and chromatin‐modifying reagents valproic acid and 5‐aza‐2′‐deoxycytidine to affect mobilized peripheral blood CD34⁺ cell fate decisions. Vox Sanguinis. 107(1). 83–89. 3 indexed citations

Tura-Ceide, Olga, G R Barclay, Kay Samuel, et al.. (2012). Late Outgrowth Endothelial Cells Resemble Mature Endothelial Cells and Are Not Derived from Bone Marrow. Stem Cells. 31(2). 338–348. 113 indexed citations

Gallagher, Ronald, et al.. (2010). Analysis of Wnt pathway genes duringex vivoexpansion and neutrophil differentiation of umbilical cord-blood derived CD34⁺cells. Vox Sanguinis. 98(3p1). e290–e294. 4 indexed citations

MacDonald, Shirley L., I. Downing, Anne Atkinson, et al.. (2010). Dendritic cells previously exposed to mannan-binding lectin enhance cytokine production in allogeneic mononuclear cell cultures. Human Immunology. 71(11). 1077–1083. 11 indexed citations

Hay, David C., Judy Fletcher, Catherine Payne, et al.. (2008). Highly efficient differentiation of hESCs to functional hepatic endoderm requires ActivinA and Wnt3a signaling. Proceedings of the National Academy of Sciences. 105(34). 12301–12306. 324 indexed citations

Gallagher, Ronald, Trevor Hay, Valérie S. Méniel, et al.. (2002). Inactivation of Apc perturbs mammary development, but only directly results in acanthoma in the context of Tcf-1 deficiency. Oncogene. 21(42). 6446–6457. 31 indexed citations

Fulton, Ruth, Ronald Gallagher, D H Crouch, & James C. Neil. (1996). Apparent uncoupling of oncogenicity from fibroblast transformation and apoptosis in a mutant myc gene transduced by feline leukemia virus. Journal of Virology. 70(2). 1154–1162. 5 indexed citations

Gallagher, Ronald, James C. Neil, & Ruth Fulton. (1995). Cloning and sequence of the feline max, and max 9 transcripts. DNA sequence. 5(5). 269–271. 2 indexed citations

10.

Gallagher, Ronald, et al.. (1993). In Vivo and In Vitro Studies of Telomeres and Telomerase. Cold Spring Harbor Symposia on Quantitative Biology. 58(0). 707–718. 29 indexed citations

11.

Gallagher, Ronald, et al.. (1991). Recovery of Intertidal Biotic Communities at Sullom Voe Following the Esso Bernicia Oil Spill of 1978. International Oil Spill Conference Proceedings. 1991(1). 461–465. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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