Ruth Fulton

690 total citations
21 papers, 581 citations indexed

About

Ruth Fulton is a scholar working on Immunology, Genetics and Molecular Biology. According to data from OpenAlex, Ruth Fulton has authored 21 papers receiving a total of 581 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Immunology, 10 papers in Genetics and 7 papers in Molecular Biology. Recurrent topics in Ruth Fulton's work include Virus-based gene therapy research (9 papers), T-cell and Retrovirus Studies (9 papers) and Animal Disease Management and Epidemiology (4 papers). Ruth Fulton is often cited by papers focused on Virus-based gene therapy research (9 papers), T-cell and Retrovirus Studies (9 papers) and Animal Disease Management and Epidemiology (4 papers). Ruth Fulton collaborates with scholars based in United Kingdom, Japan and United States. Ruth Fulton's co-authors include James C. Neil, M. Stewart, David Onions, James C. Neil, Mark Plumb, John T. Knowler, G.D. Birnie, Chris Bartholomew, Lesley Shield and Merlin Crossley and has published in prestigious journals such as Nature, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Ruth Fulton

21 papers receiving 573 citations

Peers

Ruth Fulton

GENET

IMMUN

VIROL

EPIDE

Louis J. Rezanka United States

C. K. Grant United Kingdom

S A Okenquist United States

Sanggu Kim United States

Stephen P. Hauptman United States

Jesse Thompson United States

Benjamin Rovinski Canada

Douglas A. Dedera United States

Quentin Tonelli United States

Tina Lenasi United States

Louis J. Rezanka United States

Ruth Fulton

144 ×0.5

GENET

143 ×0.6

265 ×1.3

IMMUN

90 ×0.6

VIROL

87 ×0.8

EPIDE

Citations per year, relative to Ruth Fulton Ruth Fulton (= 1×) peers Louis J. Rezanka

Countries citing papers authored by Ruth Fulton

Since Specialization

Citations

This map shows the geographic impact of Ruth Fulton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ruth Fulton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ruth Fulton more than expected).

Fields of papers citing papers by Ruth Fulton

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ruth Fulton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ruth Fulton. The network helps show where Ruth Fulton may publish in the future.

Co-authorship network of co-authors of Ruth Fulton

This figure shows the co-authorship network connecting the top 25 collaborators of Ruth Fulton. A scholar is included among the top collaborators of Ruth Fulton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ruth Fulton. Ruth Fulton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Fulton, Ruth, et al.. (2005). Population estimates; backseries methodology for 1992-2000.. PubMed. 18–26. 3 indexed citations

Brouillet, Jean‐Paul, Anna Kilbey, Ruth Fulton, et al.. (2001). Evi-1 Transforming and Repressor Activities Are Mediated by CtBP Co-repressor Proteins. Journal of Biological Chemistry. 276(28). 25834–25840. 79 indexed citations

Parkinson, Eric Kenneth, et al.. (2001). Detection of functional PTEN lipid phosphatase protein and enzyme activity in squamous cell carcinomas of the head and neck, despite loss of heterozygosity at this locus. British Journal of Cancer. 84(12). 1630–1634. 28 indexed citations

Stewart, M., Christos Tsatsanis, Ruth Fulton, et al.. (1999). The fit-1 common integration locus in human and mouse is closely linked to MYB. Mammalian Genome. 10(6). 556–559. 10 indexed citations

Fulton, Ruth, Ronald Gallagher, D H Crouch, & James C. Neil. (1996). Apparent uncoupling of oncogenicity from fibroblast transformation and apoptosis in a mutant myc gene transduced by feline leukemia virus. Journal of Virology. 70(2). 1154–1162. 5 indexed citations

Gallagher, Ronald, James C. Neil, & Ruth Fulton. (1995). Cloning and sequence of the feline max, and max 9 transcripts. DNA sequence. 5(5). 269–271. 2 indexed citations

Argyle, D. J., et al.. (1995). Nucleotide and predicted peptide sequence of feline interferon—gamma (IFN-γ). DNA sequence. 5(3). 169–171. 16 indexed citations

Terry, Anne, John J. Callanan, Ruth Fulton, O. Jarrett, & James C. Neil. (1995). Molecular analysis of tumours from feline immunodeficiency virus (FIV)‐infected cats: An indirect role for FIV?. International Journal of Cancer. 61(2). 227–232. 33 indexed citations

Tsatsanis, Christos, Ruth Fulton, Kazuo Nishigaki, et al.. (1994). Genetic determinants of feline leukemia virus-induced lymphoid tumors: patterns of proviral insertion and gene rearrangement. Journal of Virology. 68(12). 8296–8303. 64 indexed citations

10.

Lobelle‐Rich, Patricia, et al.. (1993). Coincident Involvement of flvi-2, c-myc, and Novel env Genes in Natural and Experimental Lymphosarcomas Induced by Feline Leukemia Virus. Virology. 196(2). 892–895. 19 indexed citations

11.

Tsujimoto, Hajime, Ruth Fulton, Kazuo Nishigaki, et al.. (1993). A Common Proviral Integration Region, fit-1, in T-Cell Tumors Induced by Myc-Containing Feline Leukemia Viruses. Virology. 196(2). 845–848. 22 indexed citations

12.

Terry, Astrid, Ruth Fulton, M. Stewart, David Onions, & James C. Neil. (1992). Pathogenesis of feline leukemia virus T17: contrasting fates of helper, v-myc, and v-tcr proviruses in secondary tumors. Journal of Virology. 66(6). 3538–3549. 13 indexed citations

13.

Neil, James C., et al.. (1991). Feline Leukaemia Virus: Generation of Pathogenic and Oncogenic Variants. Current topics in microbiology and immunology. 171. 67–93. 75 indexed citations

14.

Plumb, Mark, Ruth Fulton, Lars H. Breimer, et al.. (1991). Nuclear factor 1 activates the feline leukemia virus long terminal repeat but is posttranscriptionally down-regulated in leukemia cell lines. Journal of Virology. 65(4). 1991–1999. 26 indexed citations

15.

Tzavaras, Theodore, M. Stewart, Allan McDougall, et al.. (1990). Molecular cloning and characterization of a defective recombinant feline leukaemia virus associated with myeloid leukaemia. Journal of General Virology. 71(2). 343–354. 35 indexed citations

16.

Fulton, Ruth, Mark Plumb, Lesley Shield, & James C. Neil. (1990). Structural diversity and nuclear protein binding sites in the long terminal repeats of feline leukemia virus. Journal of Virology. 64(4). 1675–1682. 52 indexed citations

17.

Neil, James C., Ruth Fulton, Robert McFarlane, et al.. (1988). Receptor-mediated leukaemogenesis: hypothesis revisited.. PubMed. 9. 76–9. 1 indexed citations

18.

Neil, James C., Ruth Fulton, Theodore Tzavaras, et al.. (1987). Viral Transduction of Host Genes in Naturally Occurring Feline T-Cell Leukaemias: Transduction of myc and a T-Cell Antigen Receptor β-Chain Gene. Hämatologie und Bluttransfusion. 31. 372–376. 2 indexed citations

19.

Fulton, Ruth, G.D. Birnie, & John T. Knowler. (1985). Post-transcriptional regulation of rat liver gene expression by glucocorticoids. Nucleic Acids Research. 13(18). 6467–6482. 51 indexed citations

20.

Fulton, Ruth. (1960). Small fruit diseases in Michigan.. 370. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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