Ron Smits

8.7k citations
95 papers · 6.5k indexed · 2 hit papers · h-index 41

Ron Smits

95 papers receiving 6.4k citations

Hit Papers

Mutations in the APC tumour suppressor gene cause chromos...5452001202620092017200400600

Peers

Ron Smits
Comparison fields: 5 of 128
  • Pathology and Forensic Medicine 1.5k
  • Oncology 1.9k
  • Cancer Research 961
  • Molecular Biology 4.1k
  • Cell Biology 957
Replace Yoichi Furukawa with:
Yoichi Furukawa Japan
Jan Köster Netherlands
Tarik Möröy Germany
Mitsuru Emi Japan
Pierre Dubus France
Iris Albert United States
Elena Sancho Spain
W. J. Pledger United States
Eugenio Santos Spain
Vundavalli V. Murty United States
Ron Smits relative to Yoichi Furukawa Japan Yoichi Furukawa's profile →
Citations per field
00.5×1.6×
Yoichi Furukawa · 1×
Citations per year

Countries citing papers authored by Ron Smits

Since Specialization
Citations

This map shows the geographic impact of Ron Smits's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ron Smits with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ron Smits more than expected).

Fields of papers citing papers by Ron Smits

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ron Smits. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ron Smits. The network helps show where Ron Smits may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Ron Smits, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Ron Smits Line = papers co-authored together Ron Smits links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20249
2 20235
3 20216
4 202033
5 201932
6 20189
7 2016118
8 201664
9 201650
10 201623
11 201448
12 201242
13 201251
14 2009282
15 2006127
16 200558
17 200343
18 200062
19 1998116
20 19963

About Ron Smits

Ron Smits is a scholar working on Pathology and Forensic Medicine, Molecular Biology, Oncology, Hepatology and Rheumatology, having authored 95 papers that have together received 6.5k indexed citations. Recurring topics across this work include Wnt/β-catenin signaling in development and cancer (41 papers), Cancer-related gene regulation (29 papers), Genetic factors in colorectal cancer (26 papers), Soft tissue tumor case studies (8 papers), Digestive system and related health (8 papers), Cancer-related Molecular Pathways (7 papers), Cancer Cells and Metastasis (6 papers) and Microtubule and mitosis dynamics (4 papers). The work is most often cited by research in Pathology and Forensic Medicine (1.5k citations), Oncology (1.9k citations), Cancer Research (961 citations), Molecular Biology (4.1k citations) and Cell Biology (957 citations). Ron Smits has collaborated with scholars based in Netherlands, United States and China. Frequent co-authors include Riccardo Fodde, Hans Clevers, Menno F. Kielman, Cor Breukel, Cláudia Gaspar, Maikel P. Peppelenbosch, Patrick Franken, Ernst J. Kuipers, Wenhui Wang and Lia Smit. Their work appears in journals such as Gastroenterology, Genes Chromosomes and Cancer, Oncogene, Carcinogenesis and Scientific Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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