Robert W. Maitta

1.3k total citations
51 papers, 862 citations indexed

About

Robert W. Maitta is a scholar working on Hematology, Immunology and Genetics. According to data from OpenAlex, Robert W. Maitta has authored 51 papers receiving a total of 862 indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Hematology, 19 papers in Immunology and 14 papers in Genetics. Recurrent topics in Robert W. Maitta's work include Complement system in diseases (15 papers), Blood groups and transfusion (14 papers) and Platelet Disorders and Treatments (13 papers). Robert W. Maitta is often cited by papers focused on Complement system in diseases (15 papers), Blood groups and transfusion (14 papers) and Platelet Disorders and Treatments (13 papers). Robert W. Maitta collaborates with scholars based in United States. Robert W. Maitta's co-authors include Wenbin Xiao, Hong Hong, Michael R. Jacobs, Hillard M. Lazarus, Clifford V. Harding, Afshin Shameli, Hollie M. Reeves, Caryn E. Good, Howard Meyerson and Yu Pei and has published in prestigious journals such as Blood, PLoS ONE and Infection and Immunity.

In The Last Decade

Robert W. Maitta

47 papers receiving 836 citations

Peers

Robert W. Maitta

HEMAT

NEURO

IMMUN

EPIDE

BIOCH

Robin Verhaar Netherlands

Juan I. Jorquera Spain

Deborah L. Donahue United States

Lewis Glasser United States

Francesca Minonzio Italy

Olga Ciepiela Poland

G. O. Broun United States

Sarah J. Higgins United States

Lakshmi Kannan United States

Simon J. Cleary United Kingdom

Robin Verhaar Netherlands

Robert W. Maitta

89 ×0.4

HEMAT

82 ×0.4

NEURO

36 ×0.2

IMMUN

63 ×0.5

EPIDE

158 ×1.2

BIOCH

Citations per year, relative to Robert W. Maitta Robert W. Maitta (= 1×) peers Robin Verhaar

Countries citing papers authored by Robert W. Maitta

Since Specialization

Citations

This map shows the geographic impact of Robert W. Maitta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert W. Maitta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert W. Maitta more than expected).

Fields of papers citing papers by Robert W. Maitta

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert W. Maitta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert W. Maitta. The network helps show where Robert W. Maitta may publish in the future.

Co-authorship network of co-authors of Robert W. Maitta

This figure shows the co-authorship network connecting the top 25 collaborators of Robert W. Maitta. A scholar is included among the top collaborators of Robert W. Maitta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert W. Maitta. Robert W. Maitta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Maitta, Robert W., et al.. (2025). Thrombocytopenia in Sepsis. Life. 15(2). 274–274. 5 indexed citations

Snyder, M & Robert W. Maitta. (2025). Anti-ADAMTS13 Autoantibodies in Immune-Mediated Thrombotic Thrombocytopenic Purpura. Antibodies. 14(1). 24–24. 2 indexed citations

Maitta, Robert W., et al.. (2025). Alpha synuclein and inflammaging. Heliyon. 11(2). e41981–e41981. 1 indexed citations

Maitta, Robert W., et al.. (2024). Pathological Mechanisms and Novel Testing Methods in Thrombotic Thrombocytopenic Purpura. Biomedicines. 12(3). 621–621. 1 indexed citations

Maitta, Robert W.. (2023). Apheresis collection of mononuclear cells for chimeric‐antigen receptor therapies. European Journal Of Haematology. 112(1). 36–40. 2 indexed citations

Meyerson, Howard, et al.. (2021). Initial assessment of α-synuclein structure in platelets. Journal of Thrombosis and Thrombolysis. 53(4). 950–953. 6 indexed citations

Kundrapu, Sirisha, Hollie M. Reeves, & Robert W. Maitta. (2020). Absolute Immature Platelet Counts Suggest Platelet Production Suppression during Complicated Relapsing Thrombotic Thrombocytopenic Purpura. Acta Haematologica. 144(4). 465–469. 5 indexed citations

Reeves, Hollie M. & Robert W. Maitta. (2020). Immature Platelet Dynamics in Immune-Mediated Thrombocytopenic States. Frontiers in Medicine. 7. 597734–597734. 14 indexed citations

Pei, Yu & Robert W. Maitta. (2019). Alpha synuclein in hematopoiesis and immunity. Heliyon. 5(10). e02590–e02590. 44 indexed citations

10.

Zhu, Menglei, et al.. (2019). Non-O blood group thrombotic thrombocytopenic purpura patients take longer to recover as measured by number of therapeutic plasma exchanges needed for platelet recovery. Thrombosis Research. 185. 78–84. 6 indexed citations

11.

Chen, Wei, et al.. (2018). Absolute immature platelet counts in the setting of suspected heparin-induced thrombocytopenia may predict anti-PF4-heparin immunoassay testing results. Transfusion and Apheresis Science. 57(4). 507–511. 10 indexed citations

12.

Shameli, Afshin, Wenbin Xiao, Yan Zheng, et al.. (2015). A critical role for alpha-synuclein in development and function of T lymphocytes. Immunobiology. 221(2). 333–340. 73 indexed citations

13.

Bittencourt, Cassiana E., et al.. (2015). Re-Examination of 30-Day Survival and Relapse Rates in Patients with Thrombotic Thrombocytopenic Purpura-Hemolytic Uremic Syndrome. PLoS ONE. 10(5). e0127744–e0127744. 10 indexed citations

14.

Hong, Hong, Wenbin Xiao, Lisa M. Stempak, Linda M. Sandhaus, & Robert W. Maitta. (2014). Absolute immature platelet count dynamics in diagnosing and monitoring the clinical course of thrombotic thrombocytopenic purpura. Transfusion. 55(4). 756–765. 21 indexed citations

15.

Xiao, Wenbin, Afshin Shameli, Clifford V. Harding, Howard Meyerson, & Robert W. Maitta. (2014). Late stages of hematopoiesis and B cell lymphopoiesis are regulated by α-synuclein, a key player in Parkinson's disease. Immunobiology. 219(11). 836–844. 65 indexed citations

16.

Stempak, Lisa M., et al.. (2013). Immature platelet fraction can help adjust therapy in refractory thrombotic microangiopathic hemolytic anemia cases. Transfusion and Apheresis Science. 49(3). 644–646. 16 indexed citations

17.

Maitta, Robert W., et al.. (2012). Emergency ABO‐incompatible liver transplant secondary to fulminant hepatic failure: Outcome, role of TPE and review of the literature. Journal of Clinical Apheresis. 27(6). 320–329. 11 indexed citations

18.

Zhang, Hailing, Robert W. Maitta, Pritish K. Bhattacharyya, et al.. (2011). γ-Synuclein Is a Promising New Marker for Staining Reactive Follicular Dendritic Cells, Follicular Dendritic Cell Sarcoma, Kaposi Sarcoma, and Benign and Malignant Vascular Tumors. The American Journal of Surgical Pathology. 35(12). 1857–1865. 13 indexed citations

19.

Maitta, Robert W., Linda A. Cannizzaro, & K.H. Ramesh. (2009). Association of MLL amplification with poor outcome in acute myeloid leukemia. Cancer Genetics and Cytogenetics. 192(1). 40–43. 12 indexed citations

20.

Pezzano, Mark, et al.. (1996). Positive Selection by Thymic Nurse Cells Requires IL-1β and Is Associated with an Increased Bcl-2 Expression. Cellular Immunology. 169(2). 174–184. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact