Robert W. Maitta
- Biochemistry top 5%
- Blood transfusion and management 6
- Hematology top 5%
- Blood groups and transfusion 14
- Platelet Disorders and Treatments 13
- Neurology top 5%
- Parkinson's Disease Mechanisms and Treatments 11
- Neurology top 10%
- Parkinson's Disease Mechanisms and Treatments 11
-
- Complement system in diseases 15
-
- Renal Diseases and Glomerulopathies 7
-
- Hemoglobinopathies and Related Disorders 7
-
- Alzheimer's disease research and treatments 5
- Co-authors
- Wenbin XiaoHong HongMichael R. JacobsHillard M. LazarusClifford V. HardingAfshin ShameliHollie M. ReevesCaryn E. Good
- Partner nations
- United States
In The Last Decade
Robert W. Maitta
47 papers receiving 836 citations
Peers
Comparison fields: 5 of 89
- Biochemistry 133
- Hematology 222
- Management of Technology and Innovation 99
- Neurology 207
- Neurology 95
Countries citing papers authored by Robert W. Maitta
This map shows the geographic impact of Robert W. Maitta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert W. Maitta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert W. Maitta more than expected).
Fields of papers citing papers by Robert W. Maitta
This network shows the impact of papers produced by Robert W. Maitta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert W. Maitta. The network helps show where Robert W. Maitta may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Robert W. Maitta, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 5 | |
| 2 | 2025 | 2 | |
| 3 | 2025 | 1 | |
| 4 | 2024 | 1 | |
| 5 | 2023 | 2 | |
| 6 | 2021 | 6 | |
| 7 | 2020 | 5 | |
| 8 | 2020 | 14 | |
| 9 | 2019 | 44 | |
| 10 | 2019 | 6 | |
| 11 | 2018 | 10 | |
| 12 | 2015 | 73 | |
| 13 | 2015 | 10 | |
| 14 | 2014 | 21 | |
| 15 | 2014 | 65 | |
| 16 | 2013 | 16 | |
| 17 | 2012 | 11 | |
| 18 | 2011 | 13 | |
| 19 | 2009 | 12 | |
| 20 | 1996 | 20 |
About Robert W. Maitta
Robert W. Maitta is a scholar working on Hematology, Genetics and Biochemistry, having authored 51 papers that have together received 862 indexed citations. Recurring topics across this work include Complement system in diseases (15 papers), Blood groups and transfusion (14 papers), Platelet Disorders and Treatments (13 papers), Parkinson's Disease Mechanisms and Treatments (11 papers), Renal Diseases and Glomerulopathies (7 papers), Hemoglobinopathies and Related Disorders (7 papers), Blood transfusion and management (6 papers) and Alzheimer's disease research and treatments (5 papers). The work is most often cited by research in Biochemistry (133 citations), Hematology (222 citations) and Management of Technology and Innovation (99 citations). Robert W. Maitta has collaborated with scholars based in United States. Frequent co-authors include Wenbin Xiao, Hong Hong, Michael R. Jacobs, Hillard M. Lazarus, Clifford V. Harding, Afshin Shameli, Hollie M. Reeves, Caryn E. Good, Howard Meyerson and Yu Pei. Their work appears in journals such as Transfusion, Biomedicines, Thrombosis Research, European Journal Of Haematology and Vox Sanguinis.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.